Human Stat3-Regulated cDNA Plate Array Gene Expression
- Known as:
- Human Stat3-Regulated complementary Desoxyribonucleic acid Plate Array Gene Expression
- Catalog number:
- AP-0151
- Product Quantity:
- 4
- Category:
- -
- Supplier:
- Signosis 2011
- Gene target:
- Human Stat3-Regulated cDNA Plate Array Gene Expression
Related genes to: Human Stat3-Regulated cDNA Plate Array Gene Expression
- Gene:
- FOXD3 NIH gene
- Name:
- forkhead box D3
- Previous symbol:
- -
- Synonyms:
- Genesis, HFH2
- Chromosome:
- 1p31.3
- Locus Type:
- gene with protein product
- Date approved:
- 1999-12-22
- Date modifiied:
- 2015-08-25
Related products to: Human Stat3-Regulated cDNA Plate Array Gene Expression
Related articles to: Human Stat3-Regulated cDNA Plate Array Gene Expression
- Carcass traits are crucial indicators of meat production efficiency. However, the molecular regulatory mechanisms associated with these traits remain unclear. - Source: PubMed
Publication date: 2024/05/11
Zhao DiLiu RanranTan XiaodongKang HuiminWang JieMa ZhengZhao HaiquanXiang HaiZhang ZhengfenLi HuaZhao Guiping - A lot of people are dying from pancreatic cancer (PC) annually. The early detection of this cancer is particularly challenging due to the fact that symptoms tend to appear in advanced stages. It has been suggested that oxidative stress may play a role in the development of PC. Several genes regulate this process, including long noncoding RNAs (lncRNAs). There is no comprehensive study on the expression pattern of lncRNAs related to oxidative stress in PC patients. In the present case-control study, we quantified levels of oxidative stress-associated lncRNAs in PC patients versus healthy controls. - Source: PubMed
Publication date: 2024/04/25
Baradaran-Bagherian SetayeshMehrab Mohseni MahdiehSharifi RoyaAmirinejad RoyaShirvani-Farsani Zeinab - Hyperglycemia in pregnancy can increase the risk of congenital disorders, but little is known about craniofacial skeleton malformation and its corresponding medication. Our study first used meta-analysis to review the previous findings. Second, baicalin, an antioxidant, was chosen to counteract high glucose-induced craniofacial skeleton malformation. Its effectiveness was then tested by exposing chicken embryos to a combination of high glucose (HG, 50 mM) and 6 μM baicalin. Third, whole-mount immunofluorescence staining and hybridization revealed that baicalin administration could reverse HG-inhibited neural crest cells (NCC) delamination and migration through upregulating the expression of Pax7 and Foxd3, and mitigate the disordered epithelial-mesenchymal transition (EMT) process by regulating corresponding adhesion molecules and transcription factors (i.e., E-cadherin, N-cadherin, Cadherin 6B, Slug and Msx1). Finally, through bioinformatic analysis and cellular thermal shift assay, we identified the AKR1B1 gene as a potential target. In summary, these findings suggest that baicalin could be used as a therapeutic agent for high glucose-induced craniofacial skeleton malformation. - Source: PubMed
Publication date: 2024/03/07
Lu Jia-QiLuo Zhi-YanSun ChengyangWang Si-MiaoSun DixiangHuang Ruo-JingYang XuesongDing YongWang Guang - To elucidate the pro-tumorigenic role of IncRNA FOXD3-AS1 in glioblastoma. - Source: PubMed
Huang ConggangShao TingDuan FaliangLi RuixueLuo MingHuang QiaochunWang YuanLuo Zhihua - To explore the role of substrate stiffness and the mechanism beneath corneal endothelial cells' (CECs') stemness maintenance and differentiation. - Source: PubMed
Liu ShutingChen HuaXie HuataoLiu XinZhang Mingchang