Rat IL-6 ELISA kit
- Known as:
- Rat Interleukin-6 Enzyme-linked immunosorbent assay test reagent
- Catalog number:
- LF-EK50651
- Product Quantity:
- 1
- Category:
- Elisa Kits
- Supplier:
- Abfron
- Gene target:
- Rat IL-6 ELISA kit
Related genes to: Rat IL-6 ELISA kit
- Gene:
- CEBPB NIH gene
- Name:
- CCAAT enhancer binding protein beta
- Previous symbol:
- TCF5
- Synonyms:
- LAP, CRP2, NFIL6, IL6DBP, C/EBP-beta
- Chromosome:
- 20q13.13
- Locus Type:
- gene with protein product
- Date approved:
- 1991-02-27
- Date modifiied:
- 2018-02-23
- Gene:
- CEBPD NIH gene
- Name:
- CCAAT enhancer binding protein delta
- Previous symbol:
- -
- Synonyms:
- CRP3, CELF, C/EBP-delta, NF-IL6-beta
- Chromosome:
- 8q11.21
- Locus Type:
- gene with protein product
- Date approved:
- 1992-06-24
- Date modifiied:
- 2018-02-23
- Gene:
- ENTPD6 NIH gene
- Name:
- ectonucleoside triphosphate diphosphohydrolase 6
- Previous symbol:
- CD39L2, IL6ST2
- Synonyms:
- NTPDase-6, dJ738P15.3
- Chromosome:
- 20p11.21
- Locus Type:
- gene with protein product
- Date approved:
- 1998-03-20
- Date modifiied:
- 2019-02-28
- Gene:
- IL6 NIH gene
- Name:
- interleukin 6
- Previous symbol:
- IFNB2
- Synonyms:
- IL-6, BSF2, HGF, HSF
- Chromosome:
- 7p15.3
- Locus Type:
- gene with protein product
- Date approved:
- 1986-01-01
- Date modifiied:
- 2017-07-12
- Gene:
- IL6RP1 NIH gene
- Name:
- interleukin 6 receptor pseudogene 1
- Previous symbol:
- IL6RL1
- Synonyms:
- -
- Chromosome:
- 9q22.2
- Locus Type:
- pseudogene
- Date approved:
- 1991-08-18
- Date modifiied:
- 2014-11-19
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- 1. This study evaluated the effects of monochromatic lights on lipid metabolism, inflammation and antioxidant capacity in broilers. Eighty newly hatched broilers were assigned to four treatment groups ( = 20/group) and exposed to white (400-700 nm), red (660 nm), green (560 nm), or blue (480 nm) LED light for 35 d.2. Abdominal adipose tissue histology, tissue weight, plasma hormones, blood biochemistry, lipid metabolism gene/protein expression, antioxidant activity and inflammatory levels were assessed.3. Green and blue light exposure increased abdominal adipose tissue weight, adipocyte size and relative area of lipid droplets ( < 0.05). These groups exhibited elevated high-density lipoprotein cholesterol (HDL-C), adiponectin (adipoQ), very low-density lipoprotein (VLDL) and Triglyceride (TG) levels ( < 0.05), concurrent with reduced free fatty acid (FFA) concentrations in plasma and adipose tissue.4. Green and blue light increased fat synthesis gene expression (and; < 0.05) while downregulating fatty acid (FA) uptake genes (; < 0.001), FA β-oxidation genes (and; < 0.05) and lipid export genes (; < 0.05) in the liver. Additionally, blue light significantly inhibited lipolysis-related genes (; < 0.001) to a greater extent than it affected fat synthesis compared with white light in abdominal adipose tissue.5. Both green and blue lights upregulated anti-inflammatory factors (IL-4, IL-10; < 0.05), elevated glutathione (GSH), total glutathione (T-GSH) and oxidised glutathione/glutathione (GSSG/GSH) ratios ( < 0.05) and reduced malondialdehyde (MDA) levels ( < 0.001) and pro-inflammatory markers (IL-6, IL-1β, TNF-α) ( < 0.05). Notably, green light treatment had the highest total antioxidant capacity (T-AOC) in the liver and abdominal adipose tissues.6. The results suggested exposure to monochromatic blue and green light leads to lipid accumulation, decreased inflammation and improved antioxidant capacity and green light may offer superior protection against oxidative stress. - Source: PubMed
Publication date: 2026/08/24
Xie NWang LHu GCao GWang DDu CWang ZChen YCao J - Aberrant new bone formation is a hallmark of ankylosing spondylitis (AS), yet the cellular mechanisms remain unclear. Bone marrow-derived mesenchymal stem cells (BMSCs) are crucial for skeletal homeostasis and may be pathologically reprogrammed under chronic inflammation. We aimed to characterize the cellular heterogeneity and differentiation states of BMSCs in AS, identify BMSC subpopulations associated with structural damage, and explore their molecular features and potential therapeutic targets. - Source: PubMed
Publication date: 2026/08/24
Luo XiqingLi JinweiQi JunLin ChurongWu JingxiaoJin OuGu Jieruo - Endothelial cells (EC) dysfunction and barrier disruption are central drivers of inflammatory lung injury and adverse outcomes, yet targeted therapies remain lacking. We tested the hypothesis that a mesenchymal stem cell (MSC)-derived secretome can directly preserve EC function under inflammatory stress. We evaluated AlloEx (Vitro Biopharma), a Wharton's Jelly MSC-derived secretome enriched in bioactive RNAs, proteins, lipids, and extracellular vesicles with pro-reparative and immunomodulatory activity. - Source: PubMed
Publication date: 2026/08/24
Pokharel Marissa DOsborn Baron KSaviola Anthony JMaroney Sean PCaldwell-McGee AlyssaRichter Jillian RGetz ToddCohen Mitchell JCardenas Jessica C - Mammary tumor is one of the most common neoplasms in females and remains a major therapeutic challenge due to the high cost and adverse effects of conventional chemotherapy. Therefore, herbal products such as curcumin, baicalein and their combination are used which have anti-inflammatory effects and various other pharmacological effects. The present study evaluated the effects of curcumin and baicalein against mammary tumor induced by 7,12-dimethylbenz[a]anthracene (DMBA) in Wistar rats. Thirty-eight virgin female rats were divided into five groups: Group I (SHAM), Group II (DMBA @ 80 mg/kg BW orally single dose), Group III (DMBA @ 80 mg/kg BW orally + curcumin @ 100 mg/kg BW orally), Group IV (DMBA @ 80 mg/kg BW orally + baicalein @10 mg/kg BW i.p.), and Group V (DMBA @80 mg/kg + curcumin @100 mg/kg BW + baicalein @10 mg/kg BW). Curcumin and baicalein treatment demonstrated significant efficacy. Only small palpable growths were observed in treated groups and mild early neoplastic alterations were detected histologically, with the lowest incidence in the combination group. Curcumin and baicalein supplementation significantly ameliorated the DMBA induced changes in the body weight, hematological parameters (Hb, PCV, TEC, TLC, DLC), biochemical parameters (AST, ALT, ALKP, Total protein, glucose, BUN, Creatinine), oxidative stress parameters (NO, LPO, SOD, and Catalase), and pro-inflammatory cytokines (IL-I, IL-6, TNF-α). Moreover, immunohistochemistry and RT-PCR of epithelial-mesenchymal transition markers (e-cadherin, vimentin, α-smooth muscle actin) confirmed the ameliorative effect of curcumin and baicalein, most pronounced in the combination group. These findings suggest that curcumin and baicalein may exert synergistic beneficial effects, supporting their potential as chemopreventive agents. - Source: PubMed
Syal PriyankaSingh Nittin DevDevi Leishangthem GeetaBanga Harmanjit Singh - BACKGROUNDElucidating immune signals through well-defined cohorts of pediatric acute pancreatitis (AP) and chronic pancreatitis (CP) patients is critical. This study aimed to evaluate plasma chemokine and cytokine levels in pediatric participants with CP, compared with AP and healthy controls (HCs), to identify unique biomarkers of CP.METHODSIndividuals were identified from a prospectively collected pediatric cohort (n = 146). Immunoproteins (n = 247) were measured using the NULISAseq platform on samples from individuals with CP (n = 71), AP (n = 55), and HCs (n = 20).RESULTSThe measured analytes showed separation among the 3 groups (R2 = 0.13, P < 0.001). In the CP group, TRANCE, TWEAK, FLT-1, HGF, and TRAIL were increased when compared with HC and AP patients (FDR-corrected P <0.05). A multivariable logistic regression model including all 5 proteins provided an AUC of 0.94 (0.93-0.96) for differentiating samples from CP versus AP or HC samples. In the AP group, CRP, IL-6, CD3E, ENRAGE, and MIF were elevated compared with HCs, while FGF-2, TAFA-5, IL-33, TRANCE, and CXCL12 were downregulated in the same acute time period (FDR-corrected P < 0.05). In the 21 patients with AP for whom follow-up samples were obtained, there was a notable decrease in sequential expression of IL-6 and CRP over 12 months and increased expression of CCL25, TAFA-5, and TRANCE proteins. Additionally, TRANCE was expressed on CP pancreatic tissue.CONCLUSIONSTRANCE was increased in pediatric patients with CP and decreased in those with AP during a flare. Future studies are needed to investigate the role of TRANCE and other analytes in the pathogenesis of CP. - Source: PubMed
Publication date: 2026/08/24
Farrell Peter RLee BomiAhmed FaizanMoreno-Fernandez Maria EDixit AjayGurria Juan PabloOllberding Nicholas JDuan QingGarlapally VineetChristian PhoebeHusain Sohail ZAbu-El-Haija Maisam