CBR3 ELISA kit
- Known as:
- CBR3 Enzyme-linked immunosorbent assay test reagent
- Catalog number:
- DL-CBR3-Hu
- Product Quantity:
- 96T
- Category:
- Elisa Kits
- Supplier:
- WDSTD
- Gene target:
- CBR3 ELISA kit
Related genes to: CBR3 ELISA kit
- Gene:
- CBR3 NIH gene
- Name:
- carbonyl reductase 3
- Previous symbol:
- -
- Synonyms:
- SDR21C2
- Chromosome:
- 21q22.12
- Locus Type:
- gene with protein product
- Date approved:
- 1998-01-16
- Date modifiied:
- 2016-10-05
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- This study aimed to characterize pharmacogenomic variation in genes involved in the metabolism and transport of drugs used in Berlin-Frankfurt-Münster-based therapy in Amazonian Indigenous individuals and to compare allele frequencies with major continental populations. - Source: PubMed
Publication date: 2026/09/07
Leitão Luciana Pereira ColaresMonte NatashaMarcellino Juliana Carla RodriguesRibeiro-Dos-Santos André MaurícioWanderley Alayde Vieirade Souza Sandro Joséde Santana Souza Jorge EstefanoRibeiro-Dos-Santos ÂndreaSantos SidneyGuerreiro João FariasDos Santos Ney Pereira Carneiro - This study aimed to clarify the incidence of anthracycline-induced cardiotoxicity (ACT) and identify its clinical and candidate genetic risk factors in Chinese early-stage breast cancer patients, so as to provide evidence for clinical risk assessment and individualized cardiac protection. - Source: PubMed
Publication date: 2026/08/04
Xu ShanYang HaoHu Nan - BackgroundIndividuals with Down syndrome (DS), caused by triplication of chromosome 21 (Hsa21), face a significantly increased risk of early-onset Alzheimer's disease (AD) and epilepsy. However, the specific impact of Hsa21 genes on these risks is not yet fully understood.ObjectiveTo investigate how triplication of mouse chromosome 16 (Mmu16), homologous to Hsa21, affects amyloid-β (Aβ) accumulation in the brain and epileptic seizures in AD-DS model mice.MethodsTo generate AD-DS model mice, we crossed a mouse model of aspects of AD-an APPswe/PS1 mouse exhibiting brain Aβ accumulation and sudden death associated with epileptic seizures with DS mouse models carrying an extra copy of partial segments of mouse chromosome 16. We used three DS model lines: Ts1Cje, harboring a triplicated region encoding ∼70 Hsa21-homologous genes (-); Ts1Rhr with triplication of the - region; and a newly developed Ts1Kei mouse carrying an extra copy of the - region. Aβ accumulation was assessed by immunohistochemistry and enzyme-linked immunosorbent assay.ResultsCompared with APPswe/PS1 mice, Aβ deposition and insoluble Aβ levels in the brain decreased in APPswe/PS1-Ts1Cje mice but not in APPswe/PS1-Ts1Rhr mice. The high mortality in APPswe/PS1 mice was suppressed by either Ts1Cje- or Ts1Rhr-triplicated region. Despite a tendency for decreased Aβ accumulation in APPswe/PS1-Ts1Kei mice, the study could not be finished due to the extremely high mortality.ConclusionsThe trisomic region in Ts1Kei mice is suggested to harbor genes associated with decreased Aβ accumulation. Alternatively, the trisomic region in Ts1Rhr mice contains genes suppressing sudden death in APPswe/PS1 mice. - Source: PubMed
Publication date: 2026/07/23
Ishihara KeiichiYasui HarukaHarada KokiAmano KenjiSaito MichikoKaneda KinoKatsuda MizukiKawashita EriHata TomokaAnnoura ReinaNishimura KaneyasuTerasaki RyoSago HaruhikoShimohama ShunAkiba SatoshiYamakawa KazuhiroTakata Kazuyuki - Severe pneumonia (SP) is a life-threatening pulmonary infection characterized by high morbidity, frequent complications, and elevated mortality, with rising global incidence in recent years. This study investigated the prognostic value and molecular mechanisms of lncRNA CBR3-AS1 (CBR3-AS1) in patients with SP. - Source: PubMed
Liu ChaoLi MeieYang JunfaLong Pan - Doxorubicin (Dox) is a cornerstone in the treatment of pediatric acute lymphoblastic leukemia (ALL), but its use is limited by dose-dependent cardiotoxicity. Oxidative stress, arising from mitochondrial dysfunction, enzymatic generation of reactive oxygen species, and cardiotoxic metabolites, has been implicated as a central mechanism, with interindividual variability partly influenced by genetic factors. This study evaluated oxidative DNA damage 8-hydroxy-2'-deoxyguanosine (8-OHdG) as an integrative marker of redox-related pathways in Dox-induced cardiotoxicity. In a prospective case-control study, 93 pediatric patients with ALL treated with Dox and 63 controls were included. Cardiotoxicity was assessed by serial echocardiography, and 8-OHdG levels were measured by ELISA. Genotyping of rs3743527, rs1883112, and rs1056892 was performed, and multivariable analyses were conducted. Dox-treated patients showed higher 8-OHdG levels than controls, and patients with cardiotoxicity ( = 11) had higher levels than those without. A higher frequency and severity of cardiotoxicity was observed in female patients, although this finding should be interpreted cautiously. Although allele frequencies did not reach statistical significance, distinct distribution patterns were observed between groups. These findings suggest that 8-OHdG may function as an integrative marker of redox dysfunction associated with Dox-induced cardiotoxicity. - Source: PubMed
Publication date: 2026/06/01
Gándara-Mireles Jesús AlonsoReyes Espinoza Elio AarónLoera-Castañeda VerónicaCórdova Hurtado Lourdes PatriciaGonzález Font Antonio EmilioGrijalva Ávila Julio CesarVillanueva Fierro IgnacioLares-Asseff IsmaelMuñoz Cynthia MoraVelasco Villa GabrielaPayán Gándara HugoPatrón-Romero LeslieAlmanza-Reyes Horacio