CCR3 ELISA kit
- Known as:
- CCR3 Enzyme-linked immunosorbent assay test reagent
- Catalog number:
- DL-CCR3-Hu
- Product Quantity:
- 96T
- Category:
- Elisa Kits
- Supplier:
- WDSTD
- Gene target:
- CCR3 ELISA kit
Related genes to: CCR3 ELISA kit
- Gene:
- CCR3 NIH gene
- Name:
- C-C motif chemokine receptor 3
- Previous symbol:
- CMKBR3
- Synonyms:
- CC-CKR-3, CKR3, CD193
- Chromosome:
- 3p21.31
- Locus Type:
- gene with protein product
- Date approved:
- 1995-05-30
- Date modifiied:
- 2016-10-05
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- [This corrects the article DOI: 10.1002/ccr3.73334.]. - Source: PubMed
Publication date: 2026/08/31
- [This corrects the article DOI: 10.1002/ccr3.73142.]. - Source: PubMed
Publication date: 2026/08/30
- [This corrects the article DOI: 10.1002/ccr3.72309.]. - Source: PubMed
Publication date: 2026/08/30
- Nucleated erythroid cells (NECs) are emerging as important immunoregulators at the feto-maternal interface, yet their chemokine profiles and functional dynamics across pregnancy remain poorly understood. Using a murine allogeneic pregnancy model (CBA × C57Bl/6), we isolated placental and splenic TER-119 NECs at mid- (E12.5) and late (E19.5) gestation. Chemokine production (13-plex), chemokine receptor expression (qPCR), immunosuppressive molecules (PD-L1, TGF-β, and ROS), T-cell proliferation (CFSE), and immune cell migration (Transwell) were assessed. CD45 placental NECs were the main producers of PD-L1, TGF-β, and ROS, with maximal expression at E19.5, suggesting their potential contribution to the immunosuppressive functions observed in the total TER-119 population. Chemokine production showed a striking shift in CCL17 and CXCL9 from the spleen to the placenta as pregnancy advanced (E12.5 → E19.5). Splenic NECs displayed dominant expressions of CCR3 and CXCR4. Unexpectedly, CCL2 and CCL4 blockade enhanced immune cell migration toward placental NECs at E19.5. Placental nucleated erythroid cells potently suppressed T-cell proliferation at E12.5, and this suppressive capacity remained stable until full term. Placental NECs undergo dynamic chemokine reprogramming while maintaining stable T-cell suppression. The paradoxical enhancement of migration after CCL2/CCL4 blockade suggests a complex chemokine network warranting further investigation. These findings provide new insights into the immunobiology of pregnancy and may have implications for understanding pregnancy complications. - Source: PubMed
Publication date: 2026/08/08
Shevchenko Julia ANazarov Kirill VGizbrekht Alina ASavostyanova Tatyana AZakhareva Alena PSennikov Sergey V - Allergic rhinitis (AR) is a prevalent chronic upper airway inflammatory disorder. Olfactory dysfunction (OD) in AR patients represents a frequent and burdensome complication that significantly compromises quality of life. While the pathogenesis of AR-associated OD remains incompletely characterized, emerging evidence points to olfactory bulb (OB) microglial neuroinflammation as a crucial contributor. C-C motif chemokine ligand 7 (CCL7) is consistently upregulated in AR nasal mucosa and has been documented to drive microglial inflammation. However, its expression and function in OB microglia remain undefined. This study aimed to investigate the specific role of CCL7 in OB microglia and its potential contribution to AR-associated OD, with the goal of providing new mechanistic insights and potential intervention targets. - Source: PubMed
Publication date: 2026/08/10
Yang TingShen HuiZhao TingtingMou YakuiHu YueSong XiaoyuWang YaoChen YujiaoWang HanruiRen ChaoSong Xicheng