ACTN3 ELISA kit
- Known as:
- ACTN3 Enzyme-linked immunosorbent assay test reagent
- Catalog number:
- DL-ACTN3-Hu
- Product Quantity:
- 96T
- Category:
- Elisa Kits
- Supplier:
- WDSTD
- Gene target:
- ACTN3 ELISA kit
Related genes to: ACTN3 ELISA kit
- Gene:
- ACTN3 NIH gene
- Name:
- actinin alpha 3 (gene/pseudogene)
- Previous symbol:
- -
- Synonyms:
- -
- Chromosome:
- 11q13.2
- Locus Type:
- gene with protein product
- Date approved:
- 1991-07-16
- Date modifiied:
- 2019-01-10
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- Sports injuries are a significant concern for both professional and recreational athletes, influencing performance, longevity, and rehabilitation outcomes. While external factors such as biomechanics and workload management have been extensively studied, emerging research highlights the role of genetic predispositions in injury susceptibility. This systematic review and meta-analysis consolidated findings from 24 studies examining the association between genetic polymorphisms and sports-related injuries, with a focus on musculoskeletal tissue integrity, muscle function, and inflammatory response. The analysis identified key genetic markers, including COL1A1, COL5A1, and ACTN3, associated with ligament and tendon injuries, as well as the impact of cytokine gene variants (IL-6, TNF-α) on recovery processes. The pooled odds ratio suggested a significantly increased risk of injury among individuals carrying specific genetic variants. Subgroup analyses further revealed gene-specific effects on the injury type and athlete classification. Despite these insights, gene-environment interactions and methodological variability remain challenges in fully elucidating genetic contributions to injury risk. The findings underscore the potential for personalized injury prevention strategies based on genetic screening, enhancing both sports performance and rehabilitation efficiency. - Source: PubMed
Publication date: 2026/04/02
Łosińska KingaCięszczyk PawełZajac AdamMarkowski JarosławPilch JanSmółka WojciechMaszczyk Adam - The common ACTN3 R577X polymorphism leads to α-actinin-3 deficiency in ~20% of the human population and may be detrimental to their muscle power. However, the impact of ACTN3R577X on metabolic health and exercise capacity in the general population remains unclear. The objective of the current study was to compare metabolic health markers, musculoskeletal traits, and cardiorespiratory capacity between untrained overweight α-actinin-3-deficient (XX; 20 men and 19 women) and α-actinin-3 expressing (RR; 20 men and 21 women) individuals. The participants were aged 43 ± 7 years and had a BMI of 28.6 ± 3.2 kg·m. Various metabolic health and exercise capacity aspects encompassing segmental body composition, bone density, systemic low-grade inflammation, blood lipid profile, whole-body glucose tolerance and insulin sensitivity, resting and exercise metabolism, and exercise capacity were evaluated. While XX groups had lower fat-free mass than RR groups, other anthropometrical and body composition features, including bone mineral content, did not differ between the genotype groups in either women or men. We found no significant differences between XX and RR individuals for blood lipid profile, markers of systemic inflammation, glucose tolerance, resting metabolism, and leg strength. Moreover, no clear genotype-related differences were observed in markers of insulin resistance and sensitivity, although XX women exhibited a slightly smaller increase in insulin concentration than RR women during an oral glucose tolerance test. An incremental cardiopulmonary cycling test revealed no differences in metabolic and heart rate responses, maximal fat oxidation, or exercise capacity. In conclusion, we observed no associations between α-actinin-3 deficiency and metabolic health, body composition, muscle function, or cardiorespiratory capacity in untrained overweight men and women. - Source: PubMed
Venckunas TomasChaillou ThomasDegens HansSubocius AndrejusMinderis PetrasStasiulis ArvydasMaconyte ViktorijaMickeviciene DaliaMickevicius MantasSnieckus AudriusVanckaviciene AurikaCesanelli LeonardoWesterblad HakanKamandulis Sigitas - To investigate whether polymorphisms in collagen Type V alpha 1 chain (COL5A1), actinin alpha 3 (ACTN3) and angiotensin-converting enzyme (ACE) are associated with susceptibility to anterior cruciate ligament (ACL) and medial collateral ligament (MCL) injuries in professional football players. - Source: PubMed
Publication date: 2026/04/20
Saita YoshitomoYamamoto NanakoMiyamoto-Mikami EriOhtaki TakayaIzawa HidenoriFukushima YoshifumiIshijima MuneakiFuku Noriyuki - - Source: PubMed
Publication date: 2026/04/10
Veena K MHasil V MohammedHussain Mohammad AmjadLaha SuparnaDas RanajitShenoy PrashanthChatra LaxmikanthPrabhu RachanaShetty Prathima - Vitamin D has been implicated in cellular aging through its effects on telomere maintenance, but the extent of its influence on leukocyte telomere length (LTL), telomerase activity (TA), and interactions with aging-related genetic polymorphisms remains underexplored in Asian Indians with prediabetes. This study investigated the relationship between serum vitamin D levels, LTL, TA, and variants in aging-associated genes- - Source: PubMed
Publication date: 2026/03/24
Bhatt Surya PrakashKhurana AmishaPandey ShivamMisra Anoop