Tyrosine kinase receptor A, Human
- Known as:
- Tyrosine phosphorylation catalyst receptor A, Human
- Catalog number:
- Z03106-10
- Product Quantity:
- 10ug
- Category:
- -
- Supplier:
- Genscript
- Gene target:
- Tyrosine kinase receptor Human
Related genes to: Tyrosine kinase receptor A, Human
- Gene:
- ANKK1 NIH gene
- Name:
- ankyrin repeat and kinase domain containing 1
- Previous symbol:
- -
- Synonyms:
- X-kinase
- Chromosome:
- 11q23.2
- Locus Type:
- gene with protein product
- Date approved:
- 2003-05-13
- Date modifiied:
- 2013-01-10
Related products to: Tyrosine kinase receptor A, Human
Related articles to: Tyrosine kinase receptor A, Human
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Zheng JunZou QiqiWang ChuyanYang QianqianRen JiuyuCao XiangyueWang HuijuanGao Hui - Obesity, characterized by abnormal fat accumulation with comorbidities, continues to increase dramatically, particularly in the pediatric population. Identifying the environmental and genetic causes underlying the development of obesity during early childhood is crucial for establishing preventive and protective treatments for this complex disease. We aimed to investigate genetic variants related to non-syndromic early-onset childhood obesity. - Source: PubMed
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Olgun Celebioglu Hazal BanuOzturk Ayse PinarPoyrazoglu SukranTuncer Feyza Nur - Obesity represents a significant health challenge worldwide, with an increasing trend and high prevalence in Mexico. This metabolic disease, characterized by an increase in body mass index (BMI), leads to abnormal fat accumulation that contributes to several pathologies, and is determined by a complex interaction between diet, lifestyle and genetic factors, such as single nucleotide variants (SNVs) that may influence appetite regulation, adipose tissue function, energy metabolism, reward mechanisms, motivation, food intake behavior control, energy expenditure, fatty acid transport, lipid accumulation, lipolysis, insulin sensitivity, glucose metabolism, among other biochemical processes. However, the frequency of obesity-associated genetic variants remains poorly characterized in Mexican populations-which are highly admixed-as demonstrated by population genetic studies which have established the influence of this admixture in the prevalence and distribution of obesity-related SNVs. - Source: PubMed
Publication date: 2026/04/10
Perezcano CarlosSánchez AndreaCendejas GerardoBorrayo FranciscoPérez-Coria MarianaPérez-Hernández Gerardo - Obesity has been increasing at an uncontrolled rate. Genetic variations have been shown to contribute to a higher risk of developing the condition, due to impaired satiety regulation, predisposition to addictive eating behaviors, or even specific personality traits. This study aimed to evaluate the influence of gene polymorphisms related to the dopaminergic system on developing severe obesity. - Source: PubMed
Publication date: 2026/04/16
Teixeira Myrela RibeiroFelício Rafaela de Freitas MartinsAssis Izadora Sthephanie da SilvaGouvea Laura WendlingAbreu Gabriella MedeirosSouza Ritiele Bastos dePalhinha LohannaJunior Mario Camposde Mello Neto Cícero BrasileiroMaya-Monteiro Clarissa MenezesBozza Patrícia TorresCarneiro João Regis IvarZembrzuski Verônica MarquesSalum Kaio Cezar Rodriguesda Fonseca Ana Carolina ProençaKohlrausch Fabiana Barzotto - The present study aimed to: (i) compare a patient group with solely alcohol use disorder (AUD) to a group with poly-substance use disorder (poly-SUD) regarding sociodemographic background, morbidity, mortality, and the prevalence of the A1 allele of the Taq1A polymorphism. (ii) Investigate whether gender, age, poly-SUD, and the prevalence of the A1 allele or interactions among these factors, are associated with mortality risk over an 18-year follow-up period. - Source: PubMed
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