MMP_2, human recombinant
- Known as:
- MMP_2, H. sapiens Rec.
- Catalog number:
- 7782-1000
- Product Quantity:
- 1 mg
- Category:
- -
- Supplier:
- Biovis
- Gene target:
- MMP_2 human recombinant
Ask about this productRelated genes to: MMP_2, human recombinant
- Gene:
- IMMP2L NIH gene
- Name:
- inner mitochondrial membrane peptidase subunit 2
- Previous symbol:
- IMMP2L-IT1
- Synonyms:
- IMP2
- Chromosome:
- 7q31.1
- Locus Type:
- gene with protein product
- Date approved:
- 2003-07-11
- Date modifiied:
- 2018-01-23
- Gene:
- MMP2 NIH gene
- Name:
- matrix metallopeptidase 2
- Previous symbol:
- CLG4, CLG4A
- Synonyms:
- TBE-1
- Chromosome:
- 16q12.2
- Locus Type:
- gene with protein product
- Date approved:
- 1989-05-25
- Date modifiied:
- 2015-02-23
- Gene:
- MMP15 NIH gene
- Name:
- matrix metallopeptidase 15
- Previous symbol:
- -
- Synonyms:
- MT2-MMP, MTMMP2, SMCP-2
- Chromosome:
- 16q21
- Locus Type:
- gene with protein product
- Date approved:
- 1996-11-13
- Date modifiied:
- 2016-10-05
- Gene:
- MMP23A NIH gene
- Name:
- matrix metallopeptidase 23A (pseudogene)
- Previous symbol:
- MMP21
- Synonyms:
- MIFR
- Chromosome:
- 1p36.33
- Locus Type:
- pseudogene
- Date approved:
- 1999-08-26
- Date modifiied:
- 2016-10-05
- Gene:
- MMP23B NIH gene
- Name:
- matrix metallopeptidase 23B
- Previous symbol:
- MMP22
- Synonyms:
- MIFR, MIFR-1
- Chromosome:
- 1p36.33
- Locus Type:
- gene with protein product
- Date approved:
- 1999-08-26
- Date modifiied:
- 2016-10-05
Related products to: MMP_2, human recombinant
Related articles to: MMP_2, human recombinant
- Pterygium is a common ocular surface disease characterized by a high recurrence rate and unknown etiology. - Source: PubMed
Publication date: 2025/01/16
Han ShichaoZhu WeiGuo Qianqian - Head and Neck Squamous Cell Carcinoma is a malignant tumor with high morbidity and mortality. The MMP family plays an important role in tumor invasion and metastasis. However, the mechanistic value of the MMP family as a therapeutic target and prognostic biomarker in HNSC has not been fully elucidated. - Source: PubMed
Publication date: 2023/03/20
Liu MaohuaHuang LijuanLiu YunlingYang SenRao YongChen XiaoNie MinhaiLiu Xuqian - Previous study implicated that genes of matrix metalloproteinase (MMP) family play an important role in tumor invasion, neoangiogenesis, and metastasis. However, the diverse expression patterns and prognostic values of 24 MMPs in colorectal cancer are yet to be analyzed. - Source: PubMed
Publication date: 2021/11/05
Yu JingHe ZhenHe XiaowenLuo ZhanhaoLian LeiWu BaixingLan PingChen Haitao - To use RNA sequencing (RNAseq) to characterize renal transcriptional activities of genes associated with proinflammatory and profibrotic pathways in ischemia-induced chronic kidney disease (CKD) in cats. - Source: PubMed
Lourenço Bianca NSchmiedt Chad WAlabady Magdy SStanton James BColeman Amanda EBrown Cathy ARissi Daniel RBrown Scott ATarigo Jaime L - Matrix metalloproteinases (MMPs) and their tissue inhibitors (TIMPs) substantially contribute to the regulation of intercellular interactions and thereby play a role in maintaining the tissue structure and function. We examined methylation of a subset of 5'-cytosine-phosphate-guanine-3' (CpG) dinucleotides in promoter regions of the , , and genes by methylation-sensitive restriction enzyme digestion PCR. In our collection of 183 breast cancer samples, abnormal hypermethylation was observed for CpGs in , and promoter regions. The non-methylated status of the examined CpGs in promoter regions of , and in tumors was associated with low HER2 expression, while the group of samples with abnormal hypermethylation of at least two of these MMP genes was significantly enriched with HER2-positive tumors. Abnormal methylation of and was significantly associated with a CpG island hypermethylated breast cancer subtype discovered by genome-wide DNA bisulfite sequencing. Our results indicate that abnormal hypermethylation of at least several MMP genes promoters is a secondary event not directly functional in breast cancer (BC) pathogenesis. We suggest that it is elevated and/or ectopic expression, rather than methylation-driven silencing, that might link MMPs to tumorigenesis. - Source: PubMed
Publication date: 2020/05/10
Simonova Olga AKuznetsova Ekaterina BTanas Alexander SRudenko Viktoria VPoddubskaya Elena VKekeeva Tatiana VTrotsenko Ivan DLarin Sergey SKutsev Sergei IZaletaev Dmitry VNemtsova Marina VStrelnikov Vladimir V