ENO2, His
- Known as:
- ENO2, histidine
- Catalog number:
- ENZ-298
- Product Quantity:
- 2µg
- Category:
- -
- Supplier:
- Prospecbio
- Gene target:
- ENO2 His
Related genes to: ENO2, His
- Gene:
- ENO2 NIH gene
- Name:
- enolase 2
- Previous symbol:
- -
- Synonyms:
- -
- Chromosome:
- 12p13.31
- Locus Type:
- gene with protein product
- Date approved:
- 2001-06-22
- Date modifiied:
- 2016-04-28
Related products to: ENO2, His
(Des-Gly10,D-His(Bzl)6,Pro-NHEt9)-LHRH (Histrelin) - EIA Kit,(Des-Gly10,D-His(Bzl)6,Pro-NHEt9)-LHRH (Histrelin) - EIA Kit, Host Rabbit, High Sensitivity, CE-marked(Des-Gly10,D-His(Bzl)6,Pro-NHEt9)-LHRH (Histrelin) - EIA Kit, Host RabbitHigh SensitivityCE-marked(Des-Gly10,D-His(Bzl)6,Pro-NHEt9)-LHRH (Histrelin) - EIA Kit, Host: Rabbit, High Sensitivity, CE-marked(Des-Gly10,D-His(Bzl)6,Pro-NHEt9)-LHRH (Histrelin) - EIA Kit, Host: Rabbit, High Sensitivity, CE-marked(Des-Gly10,D-His(Bzl)6,Pro-NHEt9)-LHRH (Histrelin) - Purified Antiserum - IgG(Des-Gly10,D-His(Bzl)6,Pro-NHEt9)-LHRH (Histrelin) - Purified Antiserum - IgG, Host Rabbit(Des-Gly10,D-His(Bzl)6,Pro-NHEt9)-LHRH (Histrelin) - Purified Antiserum - IgG, Host Rabbit(Des-Gly10,D-His(Bzl)6,Pro-NHEt9)-LHRH (Histrelin) - Purified Antiserum - IgG, Host: Rabbit(Des-Gly10,D-His(Bzl)6,Pro-NHEt9)-LHRH (Histrelin) - Purified Antiserum - IgG, Host: Rabbit(Des-Gly10,D-His(Bzl)6,Pro-NHEt9)-LHRH (Histrelin), High Sensitivity ELISA(Des_Gly10,His(Bzl)6,Pro_NHEt9)_LHRH Salt _ Binding _ Synonym (His(Bzl)6)_Histrelin SumFormula C66H86N18O12(Des_Gly10,His(Bzl)6,Pro_NHEt9)_LHRH Salt _ Binding _ Synonym (His(Bzl)6)_Histrelin SumFormula C66H86N18O12(His(1_Me)2)_LHRH Salt Trifluoroacetate Binding _ Synonym (His(t_Me)2)_LHRH SumFormula C56H77N17O13(His(1_Me)2)_LHRH Salt Trifluoroacetate Binding _ Synonym (His(t_Me)2)_LHRH SumFormula C56H77N17O13 Related articles to: ENO2, His
- Histone lactylation is a lactate-dependent epigenetic modification driven by glycolysis. Although elevated histone lactylation occurs in various cancers, the biological relevance of histone H4 lactylation in hepatocellular carcinoma (HCC) remains unclear. The aim of this study was to investigate the clinicopathological relevance of histone H4 lysine 5 lactylation (H4K5la) and its role in regulating lactate metabolism to promote HCC progression. - Source: PubMed
Publication date: 2026/02/17
Yamane MasahiroAkiyama YoshimitsuShimada ShuWatanabe ShuichiHatano MegumiTsukihara ShuMiyazawa SuguruTamura HanakoNara AtsushiKodera KeitaOkazaki KoheiTanji YoshiakiIgarashi YosukeKamachi AtsushiUmemura KentaroYasukawa KoyaAkahoshi KeiichiOhashi KenichiBan DaisukeTanaka Shinji - Alzheimer's disease (AD) lacks effective early diagnostic tools. N-glycosylation dysregulation involving the enzyme MGAT3 is implicated in AD pathogenesis, with untapped clinical potential for biomarker development. - Source: PubMed
Publication date: 2026/01/08
Shi GuoXunJu FengDong QiGangFang Ye - Molecular subtypes are potential prognostic and predictive tools in muscle-invasive bladder cancer (MIBC). However, subtype concordance between primary tumors and metastases, as well as subtype-specific differences in metastatic patterns, remain poorly characterized. The present study aimed to evaluate the concordance of molecular subtypes between primary tumors and matched lymph node (LN) metastases and to explore their association with metastatic patterns. Gene expression-based molecular subtypes were determined according to the five-tiered Lund Taxonomy in 182 primary tumor samples and 34 matched LN metastases from patients with MIBC who underwent upfront radical cystectomy. Subtypes identified in the primary tumors were compared with those in matched positive LNs and patterns of distant metastasis were analyzed. In addition, the association between molecular and histological subtypes was also investigated. We found an overall 62% subtype concordance between primary tumors and corresponding LN metastases, with complete concordance in the basal/squamous subtype, lower concordance in the luminal subtypes (genomically unstable: 67%; urothelial-like: 57%), and low concordance (33%) in the mesenchymal-like (Mes) subtype. Luminal subtypes were associated with LN-only metastases and less frequent distant metastases. In contrast, the Mes subtype was associated with a higher rate of distant metastases (43%), and more frequent multiorgan involvement (≥3 organs: 40%). Higher expression of the mesenchymal gene CDH2 and the neuronal-differentiation genes GNG4 and ENO2 was associated with a higher number of metastatic sites. Gene expression-based molecular subtypes may change between primary MIBCs and matched LN metastases, and these differences appear to be subtype-dependent. Mes subtype and the expression of CDH2 as well as GNG4 and ENO2 are associated with more frequent and extensive metastases, indicating highly aggressive forms of MIBC. - Source: PubMed
Olah CsillaJuhász DánielVáradi MelindaReis HenningAl-Nader MulhamMahmoud OsamaGrünwald Barbara TGrünwald ViktorHadaschik BorisNyirády PéterSzarvas Tibor - Jalili syndrome (JS) is an autosomal recessive disorder with cone-rod dystrophy and amelogenesis imperfecta caused by CNNM4 variants. This study describes salivary proteome patterns observed in a small female JS cohort to characterize the oral molecular environment. - Source: PubMed
Publication date: 2026/03/24
Rattanapornsompong KhantiSriwattanapong KanokwanGavila PatcharapornRinkrathok MawikaSriwangyang KanokratJung Han SungPorntaveetus Thantrira - Glyphosate, one of the most widely used herbicides worldwide, has raised significant concerns regarding its potential involvement in hepatotoxicity and molecular changes associated with liver cancer biology. These concerns highlight the need to better understand its underlying molecular mechanisms in hepatoma cells. Emerging evidence suggests that glyphosate exposure may increase the risk of liver cancer and chronic liver disease. However, the precise molecular alterations and promising biomarkers associated with glyphosate-induced hepatic toxicity and disease remain largely unexplored. In this study, an RNA-Seq-based in silico systems biology approach was employed to elucidate glyphosate-induced differential transcriptional profiling in hepatoma cells. This analysis revealed significant transcriptional profiling characterized by the upregulated hub genes and . These genes were primarily associated with glucose metabolism, TNF-α/NF-κB signaling, epithelial-mesenchymal transition (EMT) and cellular stress responses. Conversely, several key genes were significantly downregulated, including , , , , , , , , and , which were involved in lipid metabolism, immune regulation and non-alcoholic fatty liver disease (NAFLD) pathways. Notably, all hub genes demonstrated strong diagnostic performance, highlighting their potential as sensitive biomarkers of glyphosate exposure. Collectively, this study provides comprehensive insights into gene expression changes associated with glyphosate exposure in hepatoma cells, linking them to hepatic metabolic dysregulation and immune modulation and suggesting a panel of hub genes with potential diagnostic and therapeutic significance. - Source: PubMed
Publication date: 2026/03/19
Mishra Divya