Mouse polyclonal to CNTNAP4, Host Mouse
- Known as:
- Mouse pab CNTNAP4, Host Mouse
- Catalog number:
- YF-PA26766
- Product Quantity:
- 50 uL
- Category:
- -
- Supplier:
- Abfron
- Gene target:
- Mouse polyclonal CNTNAP4 Host
Ask about this productRelated genes to: Mouse polyclonal to CNTNAP4, Host Mouse
- Gene:
- CNTNAP4 NIH gene
- Name:
- contactin associated protein like 4
- Previous symbol:
- -
- Synonyms:
- CASPR4, KIAA1763
- Chromosome:
- 16q23.1
- Locus Type:
- gene with protein product
- Date approved:
- 2004-05-10
- Date modifiied:
- 2016-04-25
Related products to: Mouse polyclonal to CNTNAP4, Host Mouse
Related articles to: Mouse polyclonal to CNTNAP4, Host Mouse
- Presently, we investigated hypothesized roles and mechanisms of cell type-specific, selective activation of different vascular NOX (NADPH oxidase) isoforms in obesity and metabolic syndrome. - Source: PubMed
Publication date: 2026/04/23
Huang KaiHuang YuanliZhang YuhanZhang YixuanHatch Nicholas WFreed Julie KCai Hua - Lung cancer pathogenesis involves complex interactions between immune system components and metabolic pathways. However, the causal relationships between these factors remain unclear. This study aimed to employ Mendelian randomization (MR) analysis to establish causal links between immunological markers, metabolic factors, and lung cancer development, while integrating multi-omics data for comprehensive molecular characterization. - Source: PubMed
Publication date: 2025/07/28
Yang DunpengZhang WentianWang Qibin - Autism spectrum disorder (ASD) involves challenges in social interaction and communication and repetitive behaviours. CNTNAP4 is implicated in neuronal signalling, and its deficiency plays a role in ASD. Transcriptomic analyses revealed similar gene expression between the brain and in humans as well as in mice. However, the relationships between the brain and testicular gene expression profiles and metabolism in ASD remain unclear. In this study, the effects of Cntnap4 deletion on gene expression and metabolic profiles in the cerebral cortex and testes were investigated to better understand ASD pathogenesis. - Source: PubMed
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