CHROMOGENIC ECD W_MUG (Store below 8ºC) (for detection of Escherichia coli by chromogenic and fluorogenic substrates.)
- Known as:
- CHROMOGENIC ECD W_MUG (Store 8ºC) (to measure quantification Escherichia coli chromogenic fluorogenic substrates.)
- Catalog number:
- TM 1343
- Product Quantity:
- *1 Ltr.
- Category:
- -
- Supplier:
- TitanBiotech
- Gene target:
- CHROMOGENIC ECD W_MUG (Store below 8º) (for detection Escherichia coli chromogenic and fluorogenic substrates.)
Ask about this productRelated genes to: CHROMOGENIC ECD W_MUG (Store below 8ºC) (for detection of Escherichia coli by chromogenic and fluorogenic substrates.)
- Gene:
- A1BG-AS1 NIH gene
- Name:
- A1BG antisense RNA 1
- Previous symbol:
- NCRNA00181, A1BGAS, A1BG-AS
- Synonyms:
- FLJ23569
- Chromosome:
- 19q13.43
- Locus Type:
- RNA, long non-coding
- Date approved:
- 2009-07-20
- Date modifiied:
- 2013-06-27
- Gene:
- A2M NIH gene
- Name:
- alpha-2-macroglobulin
- Previous symbol:
- -
- Synonyms:
- FWP007, S863-7, CPAMD5
- Chromosome:
- 12p13.31
- Locus Type:
- gene with protein product
- Date approved:
- 1986-01-01
- Date modifiied:
- 2018-05-03
- Gene:
- AACS NIH gene
- Name:
- acetoacetyl-CoA synthetase
- Previous symbol:
- -
- Synonyms:
- FLJ12389, SUR-5, ACSF1
- Chromosome:
- 12q24.31
- Locus Type:
- gene with protein product
- Date approved:
- 2003-06-02
- Date modifiied:
- 2015-08-24
- Gene:
- AADACL3 NIH gene
- Name:
- arylacetamide deacetylase like 3
- Previous symbol:
- -
- Synonyms:
- OTTHUMG00000001887
- Chromosome:
- 1p36.21
- Locus Type:
- gene with protein product
- Date approved:
- 2006-06-30
- Date modifiied:
- 2018-05-03
- Gene:
- AADACL4 NIH gene
- Name:
- arylacetamide deacetylase like 4
- Previous symbol:
- -
- Synonyms:
- OTTHUMG00000001889
- Chromosome:
- 1p36.21
- Locus Type:
- gene with protein product
- Date approved:
- 2006-06-30
- Date modifiied:
- 2018-04-17
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Huynh Anna LGaynor Brady JCole John WMcArdle Patrick FXu HuichunJern ChristinaPare GuillaumeKittner Steven JChong MichaelStanne Tara MMitchell Braxton D - Myocardial injury activates the complement lectin pathway (LP) in acute coronary syndrome (ACS) and LP inhibition improved cardiac function in experimental studies, suggesting a direct pathogenic role. The clinical consequences of LP activation are insufficiently defined. We investigated how plasma levels of the LP activators mannose-binding lectin (MBL) and ficolin-2 (FCN2) relate to cardiac recovery and prognosis in ACS patients. - Source: PubMed
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