human dihydropyrimidinase-like 2,DPYSL2 ELISA Kit
- Known as:
- H. sapiens dihydropyrimidinase-like 2,DPYSL2 Enzyme-linked immunosorbent assay test Kit
- Catalog number:
- SL0618Hu
- Product Quantity:
- 48T
- Category:
- -
- Supplier:
- Sunlong
- Gene target:
- human dihydropyrimidinase-like 2 DPYSL2 ELISA Kit
Related genes to: human dihydropyrimidinase-like 2,DPYSL2 ELISA Kit
- Gene:
- DPYSL2 NIH gene
- Name:
- dihydropyrimidinase like 2
- Previous symbol:
- -
- Synonyms:
- DRP-2, DHPRP2, CRMP2, DRP2
- Chromosome:
- 8p21.2
- Locus Type:
- gene with protein product
- Date approved:
- 1997-02-27
- Date modifiied:
- 2016-10-05
Related products to: human dihydropyrimidinase-like 2,DPYSL2 ELISA Kit
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Arndt NicoMair ThomasRiedner MariaBiabani AliVoß HannahSchlüter HartmutFörster LukasKnochenhauer TimSachse MarcoBeyer MartinLeonhardt Mayavon Kodolitsch YskertSchlein ChristianSauter GuidoNauth TheresaNaito ShihoGirdauskas EvaldasReichenspurner HermannDetter ChristianRosenberger GeorgDemal Till Joscha - Chronic venous disease (CVD) is a prevalent vascular disorder with a poorly characterized genetic basis. In this study, we employed an integrative omics strategy combining genome-wide association studies (GWAS), expression quantitative trait loci (eQTL) mapping, endothelial cell functional assays, and transcriptomic correlation analysis to elucidate the molecular architecture of CVD. A GWAS conducted in a Taiwanese population identified two CVD-associated single nucleotide polymorphisms: VSTM2L rs1998049 and DPYSL2 rs1442887. Through eQTL analysis and endothelial functional assays, four QTLs (VSTM2L, RPRD1B, SAMHD1, and PNMA2) were found to significantly affect VEGF consumption, vWF expression, and endothelial tube formation. Co-expression and correlation analyses further linked these QTLs to key vascular effector genes, including VEGF, vWF, MMP9, and CCM2. A logistic regression model based on QTL expression profiles demonstrated high diagnostic performance (area under the curve, AUC = 0.898), highlighting their translational potential. These findings offer novel insights into the functional genomics of CVD, particularly in relation to vascular remodeling, endothelial dysfunction, and inflammation. They also demonstrate the utility of multi-omics integration for biomarker discovery in complex vascular disorders. - Source: PubMed
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