Rat obestatin ELISA KIT
- Known as:
- Rat obestatin Enzyme-linked immunosorbent assay test KIT
- Catalog number:
- 201-11-0230
- Product Quantity:
- 96T
- Category:
- -
- Supplier:
- SunBT
- Gene target:
- Rat obestatin ELISA KIT
Related genes to: Rat obestatin ELISA KIT
- Gene:
- GHRL NIH gene
- Name:
- ghrelin and obestatin prepropeptide
- Previous symbol:
- -
- Synonyms:
- MTLRP, ghrelin, obestatin
- Chromosome:
- 3p25.3
- Locus Type:
- gene with protein product
- Date approved:
- 2006-01-05
- Date modifiied:
- 2016-08-10
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- Taste perception, taste preferences, and food preferences are three interrelated constructs underlying food-related behavior and contribute to individual dietary choices. Both genetic and environmental factors contribute to shaping these traits. Taste perception refers to interindividual differences in biological sensitivity to basic taste modalities. Taste preferences represent the hedonic evaluation of taste modalities, including salty, sweet, bitter, sour, or umami, and fatty tastes, and are closely related to, but distinct from, taste perception. Recent evidence in sensory genetics has identified numerous single-nucleotide variants (SNVs) associated with interindividual differences in both taste perception and taste-modality-related responses. Essential genes include and for sweet taste, and for umami, for bitter taste, for fat perception, and epithelial sodium channel (ENaC) subunits for salt sensitivity. Regarding broader food preferences, variants in genes involved in metabolic regulation and reward processing-such as , , , , and -also contribute to food-related behaviors and choice patterns. Health status and genetic variants may also influence taste perception and food preferences. This review summarizes current evidence on the genetic determinants of taste perception, taste modality preferences, and food liking, emphasizing the multifactorial nature of food-related behaviors and the role of genetic variability in shaping individual differences in dietary choices. - Source: PubMed
Publication date: 2026/08/04
Miller-Kasprzak EwaBogdański Paweł - This study aims to investigate the influence of age and gender on the efficacy of metabolic surgery in patients with obesity with type 2 diabetes mellitus (T2DM). - Source: PubMed
Publication date: 2026/07/27
Ma BoDong YangfanLi TaoWang JunMa HuaShang Shaohua - This study evaluated the effects of live and heat-killed HP7 on intestinal motility, host physiological responses, and gut microbiota in a mouse model of delayed intestinal transit. BALB/c mice were treated with live HP7 (HP7L), heat-killed HP7 (HP7K), or bisacodyl following loperamide administration. HP7L and HP7K improved intestinal motility-related parameters, including fecal output, stool consistency, fecal moisture content, and charcoal meal transit. HP7 treatment also modulated circulating gastrointestinal hormones and inflammatory cytokines, with changes in serotonin, ghrelin, peptide YY, IL-6, and IL-17A. In intestinal tissues, HP7 regulated the expression of genes related to neuroendocrine signaling, water transport, mucus production, and tight junction integrity, including and . Histological analysis further showed reduced colonic tissue alterations and improved structural parameters. Microbiota analysis revealed that HP7L exerted broader effects on microbial community structure, whereas HP7K induced more selective taxonomic changes. Predicted functional profiling suggested that both HP7L and HP7K were associated with shifts in carbohydrate metabolism-related pathways, with HP7K showing additional predicted enrichment of pathways related to redox-associated metabolism, lipid metabolism, and microbial substrate utilization. These findings suggest that heat-killed HP7 retains biological activity and has potential as a postbiotic candidate for regulating intestinal motility. - Source: PubMed
Publication date: 2026/07/01
Gwon HyeonjunKim HyeonjiKim Joo-YunShim Jae-JungLee Jae-Hwan - Colorectal cancer (CRC) and non-Hodgkin lymphoma (NHL) are among the most prevalent cancer types globally by incidence and mortality. Both types are influenced differentially by chronic inflammation. Central to this inflammation are inflammatory genes that are meticulously regulated by nuclear factor kappa B (NF-κB) and tumor necrosis factor-α (TNF-α). NF-κB is negatively regulated by IκBα (encoded by ), while TNF-α's actions can be modulated by ghrelin (encoded by ). We investigated four single nucleotide polymorphisms (SNPs) in (rs4648068), (rs2233406), (rs1800629), and (rs1629816) as biomarkers for CRC and NHL risk in a cohort of Kuwaiti individuals. DNA samples from patients and controls were collected and genotyped for all SNPs, and their association with CRC or NHL risk was assessed. While rs4648068 showed a modest association with increased CRC risk, it had no significant impact on NHL risk. Conversely, rs2233406 increased NHL risk without affecting CRC risk. Interestingly, while rs1800629 showed a protective effect against NHL, it showed an increased risk for CRC. Finally, rs1629816 was associated with greater NHL but not CRC risk. Our findings suggests that variations of these inflammatory genes may be useful indicators for predicting cancer risk but might have unpredictable effects on cancer susceptibility, depending on the cancer type. - Source: PubMed
Publication date: 2026/05/23
AlSrayea SaraAlrashid Maryam HBastaki Nasmah KAl-Barrak Jasem - With the ongoing integration of offshore wind power and marine aquaculture, increasing attention has been paid to the potential biological effects of continuous low-frequency noise generated during wind farm operations on surrounding fish species. In this study, juvenile black scrapers (Thamnaconus modestus), a demersal species with low auditory sensitivity, were exposed to 500 Hz noise at low, medium, and high intensities (root-mean-square (RMS) sound pressure levels (SPLrms): 95 ± 5, 115 ± 5, and 135 ± 5 dB re 1 μPa) for 14 days to investigate physiological responses and molecular regulatory mechanisms. Low-intensity noise primarily suppressed the expression of feeding and digestion genes (Ghrl, Prss1) and activated oxidative stress and neuroprotective pathways. Medium-intensity noise caused dysregulation of lipid metabolism genes (Gba1, Degs2), significantly elevated malondialdehyde (MDA) content, and downregulated the expression of key genes in the neutrophil extracellular trap formation pathway (Ncf1, Ncf2, Ncf4). High-intensity noise disrupted the expression of circadian clock genes (Bmal1, Per2) and upregulated cholesterol synthesis pathway genes (Sqle, Cyp51a1, Dhcr24). Collectively, long-term low-frequency noise exposure during operation induced dose-dependent physiological stress on juvenile T. modestus, affecting digestive function, redox balance, lipid metabolism, immune responses, and circadian rhythm regulation. These findings contribute to understanding the potential impacts of low-frequency noise from offshore wind farms on marine fish and suggest that low-frequency noise should be incorporated as a key assessment criterion in the siting of marine ranching and fishery resource conservation. - Source: PubMed
Publication date: 2026/06/09
Song ShiqiShan Xiujuan