Fraction V BSA powder, protease free, 100gm
- Known as:
- Fraction V cow blood serum albumin powder, protease free, 100gm
- Catalog number:
- BAC65-0100
- Category:
- -
- Supplier:
- Equitech
- Gene target:
- Fraction BSA powder protease free 100gm
Related genes to: Fraction V BSA powder, protease free, 100gm
- Gene:
- HTRA1 NIH gene
- Name:
- HtrA serine peptidase 1
- Previous symbol:
- PRSS11
- Synonyms:
- HtrA, IGFBP5-protease, ARMD7
- Chromosome:
- 10q26.13
- Locus Type:
- gene with protein product
- Date approved:
- 1997-07-25
- Date modifiied:
- 2016-10-05
Related products to: Fraction V BSA powder, protease free, 100gm
(Arg8)-Vasopressin (free acid)
98% C46H64N14O13S2 CAS: 25255-33-8(Arg8)-Vasopressin - EIA Kit (H - sr, pl), Host Rabbit, Extraction-free, CE-marked(Arg8)-Vasopressin - EIA Kit (H - sr, pl), Host RabbitExtraction-freeCE-marked(Arg8)-Vasopressin - EIA Kit (H - sr, pl), Host: Rabbit, Extraction-free, CE-marked(Arg8)-Vasopressin - EIA Kit (H - sr, pl), Host: Rabbit, Extraction-free, CE-marked(Arg8)-Vasopressin - EIA Kit (R - sr, pl), Host Rabbit, Extraction-free, CE-marked(Arg8)-Vasopressin - EIA Kit (R - sr, pl), Host RabbitExtraction-freeCE-marked(Arg8)-Vasopressin - EIA Kit (R - sr, pl), Host: Rabbit, Extraction-free, CE-marked(Arg8)-Vasopressin - EIA Kit (R - sr, pl), Host: Rabbit, Extraction-free, CE-marked(Arg8)_Vasopressin (free acid) Salt Trifluoroacetate Binding (Disulfide_bond) Synonym Leiormone (free acid), Pitressin (free acid), AVP (free acid), Arginine Antidiuretic Hormone (free acid), Argip(Arg8)_Vasopressin (free acid) Salt Trifluoroacetate Binding (Disulfide_bond) Synonym Leiormone (free acid), Pitressin (free acid), AVP (free acid), Arginine Antidiuretic Hormone (free acid), Argip(Arg8)_Vasopressin (free acid) Salt Trifluoroacetate Binding (Disulfide_bond) Synonym Leiormone (free acid), Pitressin (free acid), AVP (free acid), Arginine Antidiuretic Hormone (free acid), Argip(Arg8)_Vasopressin (free acid) Salt Trifluoroacetate Binding (Disulfide_bond) Synonym Leiormone (free acid), Pitressin (free acid), AVP (free acid), Arginine Antidiuretic Hormone (free acid), Argip(Arg8)_Vasopressin _ EIA Kit (H _ sr, pl), Host Rabbit, Extraction Free(Arg8)_Vasopressin _ EIA Kit (R _ sr, pl), Host Rabbit, Extraction Free Related articles to: Fraction V BSA powder, protease free, 100gm
- Genome-wide association studies (GWASs) have identified common genetic risk loci for ischemic stroke (IS), primarily in European populations older than 55 years. We aimed to identify common and rare risk variants associated with early-onset IS (ages 18-54) in Taiwan. - Source: PubMed
Publication date: 2026/06/01
Wang Yin-ChengLiu Kai-MingGan Yu-LingChi Nai-FangLu Liang-SueiChou Che-YuWang Liang-YunChang Li-HsinHou Ming-ChihFu Yun-ChingChiou Jeng-FongWu Ming-ShiangYang Shun-FaPang See-TongWang Jaw-YuanTsai Yi-TingHuang Chih-YangChiu Kuan-MingChen MingChiang Fu-TienWang Chih-HungYao Wei-JenLee Sing-LianHuang Chi-HungLiu Yo-TsenWang Shuu-JiunKo Wen-YaChen Chien-HsiunLee I-Hui - - Source: PubMed
Publication date: 2026/05/27
Tsuchiya TakahiroMiyawaki SatoruHirano YudaiSakai YuOhara KentaInoue YoheiKomura DaisukeShinozaki-Ushiku AyaOgawa ShotaroSato DaisukeHongo HirokiHirata SoTeranishi YuOno HideakiOtani YoshihiroTakami HirokazuUshiku TetsuoIshikawa ShumpeiTanaka ShotaIchimura KoichiSaito Nobuhito - Ovarian cancer (OVCA) remains the most lethal gynecologic malignancy, with poor prognosis largely due to late-stage diagnosis and therapy resistance. Pyroptosis, a pro-inflammatory form of programmed cell death, has recently emerged as a regulator of tumor progression and immune regulation. This study aimed to systematically profile pyroptosis-related genes and identify robust biomarkers for OVCA. Microarray data from the GSE54388 dataset were analyzed to characterize pyroptosis-related gene expression. Immune cell infiltration was assessed using xCell, and pathway enrichment was performed via Gene Set Enrichment Analysis (GSEA). Weighted Gene Co-expression Network Analysis (WGCNA) identified hub genes, followed by Gene Ontology (GO) and Reactome enrichment. Machine learning algorithms (Support Vector Machine, XGBoost, and Generalized Linear Model) were employed for feature selection and biomarker identification. Validation was conducted across independent bulk and scRNA-seq datasets, with GEPIA2 used to compare OVCA and normal samples and KMplot for survival analysis. OVCA samples showed significantly reduced infiltration of CD4 and CD8 T cells, mast cells, monocytes, neutrophils, and immature dendritic cells compared to normal samples. GSEA revealed enrichment of cell cycle-related pathways, implicating pyroptosis-related genes as key regulators of mitotic progression. From 1097 differentially expressed genes, 22 pyroptosis-related DEGs (PYRDEGs) were identified, with nine hub genes (CASP1, CEP55, CHMP4C, HTRA1, IL18, MELK, PKM, PTX3, TNFSF13B) strongly associated with OVCA. Functional enrichment linked these genes to cytokinesis, inflammasome activity, and immune signaling. Machine learning consistently identified CEP55 as the core biomarker, demonstrating high diagnostic accuracy (AUC up to 0.972) and significant upregulation in OVCA samples. Correlation analysis linked CEP55 expression to altered immune cell populations, including positive associations with Th1 and class-switched memory B-cells and negative associations with iDCs, Tregs, and M2 macrophages. CEP55 was highly expressed across bulk and scRNA-seq datasets (cancer epithelial and CD8+ TEMRA cells) and negatively correlated with overall survival (OS) and progression-free survival (PFS). Pyroptosis-related genes play pivotal roles in OVCA pathogenesis. CEP55 emerges as a promising biomarker for early detection and a potential therapeutic target, bridging cell cycle regulation with immune modulation. - Source: PubMed
Publication date: 2026/05/21
Arya RakeshBiswas Viplov KumarShakya HemlataKim Jong-Joo - The rising incidence of CINIII, particularly in younger patients, has highlighted limitations of current surgical treatments, which can affect fertility and carry recurrence risks. This underscores an urgent clinical need for non-invasive therapies. Our study addresses this by investigating the cellular and molecular changes during CINIII progression to cervical squamous cell carcinoma (CSCC). We employed single-cell sequencing to meticulously analyze cell types and molecular mechanisms within cervical epithelial cells and their microenvironment throughout the CINIII-to-CSCC transition. Key findings include the identification of Sox2 and its signaling pathway as markers for specific cervical stem cells (CCSCs) during malignant transformation. In the microenvironment, upregulated VWF, MMP2, and HTRA1 were observed in vascular endothelial cells, while TXNIP+ and ARL4D+ fibroblasts underwent transformation into myofibroblasts. Immune cell proportions notably increased, particularly macrophages, T cells, B cells, NK cells, mast cells, and neutrophils, contrasting with a decrease in non-immune cells. Furthermore, interaction analysis revealed that communication between macrophages and cervical epithelial cells was the most prominent. This research comprehensively details the complex cellular and molecular remodeling inherent in CINIII progression. By pinpointing specific pathways and cell populations, our findings establish a crucial framework for developing innovative, non-invasive drug therapies to delay disease progression and ultimately improve long-term reproductive outcomes for patients. - Source: PubMed
Publication date: 2026/05/21
Ma YaomeiZhang SuLiu BeiLiu YiboHe YuchaoGong WenchenChen WenshuaiQi LishaWang KeGuo Hua - Age-related macular degeneration (AMD) is a leading cause of vision loss, with genetic factors playing a key role in disease susceptibility and progression. While extensive genetic research is being conducted, the genetic architecture of AMD in Middle Eastern populations remains understudied. This systematic review summarizes current evidence on genetic variants associated with AMD in Middle Eastern populations. - Source: PubMed
Publication date: 2026/05/08
Sannan Naif S