Anti-Mouse CD101 PE 100 ug
- Known as:
- Antibody toMouse CD101 PE 100 ug
- Catalog number:
- 12-1011-82
- Category:
- -
- Supplier:
- eBioscience
- Gene target:
- Anti-Mouse CD101 100
Related genes to: Anti-Mouse CD101 PE 100 ug
- Gene:
- CD101 NIH gene
- Name:
- CD101 molecule
- Previous symbol:
- IGSF2
- Synonyms:
- V7
- Chromosome:
- 1p13.1
- Locus Type:
- gene with protein product
- Date approved:
- 1998-12-03
- Date modifiied:
- 2016-10-05
Related products to: Anti-Mouse CD101 PE 100 ug
Related articles to: Anti-Mouse CD101 PE 100 ug
- Bone and joint infections secondary to species are associated with high morbidity and mortality, requiring extended courses of antifungal therapy, with parenteral medications being first-line therapy. The reported use of antifungals in outpatient parenteral antimicrobial therapy (OPAT) for deep-seated bone and joint infections is limited. We report and discuss the use of rezafungin, a novel long-acting echinocandin, in an OPAT setting for spondylodiscitis secondary to . - Source: PubMed
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Richards Alexander JAsif AmmaraIvan MonicaLillie Patrick JBarlow GavinWalsh Chloe - Candidemia remains associated with substantial morbidity and mortality despite advances in antifungal therapy, yet multiple aspects of management remain controversial. This review synthesizes emerging evidence to address unresolved clinical dilemmas in candidemia management. - Source: PubMed
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Cluck David BWhite Bryan PHilzendager Sarah ARyder Jonathan HTu Patrick JMcPherson Amanda ZRogers Daniel JChastain Daniel B - Human papillomavirus (HPV) prevalence is high among men who have sex with men (MSM), increasing the risk of anal cancer. Key risk factors include HIV co-infection and persistent HPV infection, although the underlying mechanisms remain unclear. - Source: PubMed
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Ober VeronicaGruener EvaWang DanniBaranov OlgaStirner RenateReiling GabrieleConca RaffaeleDächert ChristopherIbarra GerardoSeybold UlrichBogner Johannes RGeldmacher ChristofHeld KathrinRoider Julia - Reproduction traits constitute the primary objective of porcine genetic improvement programs, with litter size being the principal determinant of herd reproduction output. Relative to the intensively selected Large White line, as a breed derived from Large White (LW) and Beijing Black (BJB) pigs, Jishen Black (JSB) pigs exhibit markedly lower litter size, indicating substantial potential for genetic improvement. - Source: PubMed
Publication date: 2026/06/29
Chen ChangyiZhang XiaoranLi JingKe JuanSun HaoBai ChunyanSun Boxing - Tuberculosis (TB), caused by Mycobacterium tuberculosis (Mtb), is associated with clinical diversity and outcomes ranging from latent TB to active TB with distinct pathophysiologies. However, our understanding of the innate immune mechanisms related to the protection or progression of TB is limited. Among innate immune cells, the role of neutrophils is not fully elucidated, as they have been shown to exhibit both protective and harmful capacities in TB. We hypothesized that Mtb infection induces changes in neutrophil phenotype and function, influencing the infection outcomes. Based on clinical, bacteriological, and positron emission tomography with x-ray computed tomography (PET/CT) scan parameters, cynomolgus macaques infected by Mtb were stratified into two categories: animals that rapidly progressed to an active form of TB, designated as "fast progressors," and "slow progressors," which include low symptomatic or asymptomatic animals. In this study, we identified transcriptomic signatures of type I interferons and neutrophil degranulation in macaques with fast progression to active TB, which were not observed in animals with slow TB progression. Unsupervised mass cytometry analysis showed the emergence of blood immature neutrophils (CD101+ CD10-) in fast-progressing animals. In addition, bulk blood neutrophils from infected animals displayed capacities to modulate TNF-α production and cytotoxic function of CD8 T cells in a contact-dependent mechanism. In lung granulomas, neutrophils showed a tissue-specific phenotype (CD101- CD10+), with greater infiltration in animals with active TB. These data suggest that neutrophil subpopulations are associated with disease progression, with capacities to modulate CD8 T cells' functions, which in turn may contribute to disease progression. - Source: PubMed
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