NTF4, 81-210aa, Human, E.coli
- Known as:
- NTF4, 81-210aa, Human, E.coli
- Catalog number:
- ATGP1175
- Product Quantity:
- 0.5mg
- Category:
- -
- Supplier:
- ATGen
- Gene target:
- NTF4 81-210aa Human .coli
Related genes to: NTF4, 81-210aa, Human, E.coli
- Gene:
- FCN2 NIH gene
- Name:
- ficolin 2
- Previous symbol:
- -
- Synonyms:
- P35, FCNL, EBP-37, ficolin-2
- Chromosome:
- 9q34.3
- Locus Type:
- gene with protein product
- Date approved:
- 1996-07-11
- Date modifiied:
- 2016-10-05
- Gene:
- NTF4 NIH gene
- Name:
- neurotrophin 4
- Previous symbol:
- NTF5
- Synonyms:
- NT-4/5, GLC1O
- Chromosome:
- 19q13.3
- Locus Type:
- gene with protein product
- Date approved:
- 1992-10-15
- Date modifiied:
- 2014-11-19
Related products to: NTF4, 81-210aa, Human, E.coli
Related articles to: NTF4, 81-210aa, Human, E.coli
- Major depressive disorder (MDD) is associated with multiple pathophysiological mechanisms, including dysregulation of monoaminergic systems, excessive glutamatergic activity, reduced neurotrophic support, inflammation, and oxidative stress. The combination of dextromethorphan, an N-methyl-D-aspartate (NMDA) receptor antagonist and sigma-1 receptor agonist, with bupropion, a dopamine/norepinephrine reuptake inhibitor, has been reported to exert rapid antidepressant effects. In the present study, we evaluated the therapeutic effects of the dextromethorphan-bupropion combination on behavioral outcomes and neurotransmitter, inflammatory, and apoptotic pathways in a mouse model of MDD. - Source: PubMed
Publication date: 2026/08/19
Buran Kavuran İlayÖnalan EbruTürk Zeliha İremEröksüz Hatice - Stroke is a major complication of atrial fibrillation (AF), and risk prediction using the congestive heart failure, hypertension, age, diabetes, stroke, vascular disease, and sex category score (CHA₂DS₂-VASc) remains limited by residual heterogeneity. We aimed to identify plasma proteins associated with post-AF stroke and evaluate whether a protein score provides incremental predictive information beyond CHA₂DS₂-VASc. - Source: PubMed
Publication date: 2026/07/23
Huang ShengkangFeng XinWu HuilinLiang RongxuanGao YuanWang ZhaojunTang BotaoWu JiatongFang JunkaiYang ZiangTiemuerniyazi XierailiLin JianchengDuan YijingxuanXu WenruMao HuaZhao WeiHu ZhanDuan ChuanzhiFeng Wei - This study aims to investigate the role of neurotrophin-4 (NTF4) in gestational diabetes mellitus (GDM) and to elucidate the underlying mechanism by which NTF4 regulates high glucose-induced apoptosis and inflammation in HTR-8/SVneo cells via the PI3K/AKT signaling pathway. - Source: PubMed
Zhang LiYang Juan - Following the publication of the above article, the authors subsequently realized that, during the process of collating the raw data, Fig. 1 [the immunohistochemical (IHC) results for stage IV colorectal cancer (CRC)], Fig. 2A (the control β‑actin blots) and Fig. 5C and D (both the images selected for the clone formation assays, and the histograms showing the quantification of the data) were inadvertently assembled incorrectly. These errors arose as a consequence of the affected files having been named similarly to those of the correct panels. The revised versions of Figs. 1, 2 and 5, now featuring the correct IHC data for stage IV CRC in Fig. 1, the correct control western blots in Fig. 2 and the correct colony formation assay data (and quantification thereof) in Fig. 5, are shown on the next three pages. Note that the correction of these figures does not affect the key findings of the study (either the existing published results or the conclusions reached from the results). The authors thank the Editor of for granting them the opportunity to publish this corrigendum. All the authors agree with the publication of this corrigendum; furthermore, they apologize to the readership of the journal for any inconvenience caused. [International Journal of Oncology 56: 1442‑1454, 2020; DOI: 10.3892/ijo.2020.5027]. - Source: PubMed
Publication date: 2025/02/14
Yang ZhouChen YushengWei XiyiWu DejunMin ZhijunQuan Yingjun - Long non-coding RNAs (lncRNAs) are importantly involved in the initiation and progression of non-small cell lung cancer (NSCLC). However, the classification and mechanisms of lncRNAs remain largely elusive. - Source: PubMed
Publication date: 2024/10/28
Liu YinqiangYang HongjvLv GuoliDuan JinZhao WeiShi YunfeiLei Youming