TNFSF13B 3&_39;UTR Lenti-reporter-Luc Virus
- Known as:
- TNFSF13B 3&_39;UTR Lenti-reporter-Luc Virus
- Catalog number:
- MV-h26039
- Product Quantity:
- 3.0ml
- Category:
- -
- Supplier:
- ABM
- Gene target:
- TNFSF13B 3&_39;UTR Lenti-reporter-Luc Virus
Related genes to: TNFSF13B 3&_39;UTR Lenti-reporter-Luc Virus
- Gene:
- TNFSF13B NIH gene
- Name:
- TNF superfamily member 13b
- Previous symbol:
- TNFSF20
- Synonyms:
- BAFF, THANK, BLYS, TALL-1, TALL1, CD257
- Chromosome:
- 13q33.3
- Locus Type:
- gene with protein product
- Date approved:
- 1999-07-19
- Date modifiied:
- 2018-11-22
Related products to: TNFSF13B 3&_39;UTR Lenti-reporter-Luc Virus
Mouse Nanog Stem Cell Differentiation Reporter (pGreenZeo, plasmid)140 kDa nucleolar phosphoprotein,HCV NS5A-transactivated protein 13,Hepatitis C virus NS5A-transactivated protein 13,Homo sapiens,Human,KIAA0035,NOLC1,Nopp140,NS5ATP13,Nucleolar 130 kDa protein,Nucleo14_3_3_sigma (SFN) Gene Promoter Reporter Vector25% Kaolin for Rubella Virus HI Test25% Kaolin for Rubella Virus HI Test for Cobas Hitachi Bayer Behring Beckman Coulter Olympus Idexx293 5-HT7 CRE-Luc Stable Cell Line293 ADRB3 CRE-Luc Stable Cell Line293 CRE-Luc293 Luc Cell Line293 MC3 CRE-Luc Stable Cell Line293 PTH2 CRE-Luc Stable Cell Line293/5-HT7/CRE-Luc Stable Cell Line 293/ADRB3/CRE-Luc Stable Cell Line 293/MC3/CRE-Luc Stable Cell Line 293/PTH2/CRE-Luc Stable Cell Line Related articles to: TNFSF13B 3&_39;UTR Lenti-reporter-Luc Virus
- Acute inflammatory relapses in primary CNS demyelinating diseases, including relapsing multiple sclerosis (RMS), MOG-antibody-associated disease (MOGAD), and neuromyelitis optica spectrum disorder (NMOSD), drive hospitalization, disability, and socioeconomic burden. Blood-based biomarkers are increasingly used for relapse detection and treatment monitoring. - Source: PubMed
Publication date: 2026/09/07
Chumakova Anastasia PDemetriou MichaelSy Michael - Persistent minimal residual disease (MRD) significantly contributes to chemotherapy resistance and relapse in pediatric B-cell acute lymphoblastic leukemia (B-ALL), with conventional clinical risk factors insufficiently explaining the variability in patient prognoses. This study sought to explore potential mechanisms linked to sustained MRD by integrating analyses of clinical cohorts with single-cell transcriptomics, aiming to identify reliable biomarkers for risk stratification and targeted therapy. - Source: PubMed
Publication date: 2026/08/18
Zhang LinlinLiu XiaoyangChen FangXu GangFan Yang - Postoperative delirium (POD) and Alzheimer's disease (AD) are increasingly recognized as related neurocognitive conditions, but the aging-associated cell states that may connect them remain poorly defined. Here, we integrated two brain transcriptomic discovery datasets analyzed at single-cell/single-nucleus resolution, including a POD-related cohort (GSE291019) and an AD cohort (GSE129308), together with two independent peripheral-blood bulk transcriptomic datasets (GSE163943 and GSE63060), to identify aging-associated cellular programs and prioritize convergent molecular candidates. Across 184,168 high-quality cells, inhibitory neurons showed the most consistent aging-associated perturbation across the POD- and AD-related datasets. Re-clustering further identified three inhibitory-neuron subtypes, Inh_Neurons2, Inh_Neurons3, and Inh_Neurons5, with relatively high aging-related gene activity and preferential localization to later pseudotime states. Cross-platform integration of aging-associated inhibitory-neuron genes with a shared bulk DEG set identified four convergent candidates: RGL2, AKT1, SYK, and TNFSF13B. Among them, RGL2 emerged as the leading candidate, with the strongest downstream support concentrated in AD-related analyses. In two-sample Mendelian randomization, genetically predicted higher RGL2 expression was associated with increased AD risk, whereas the estimate for the delirium genome-wide association study proxy used for POD-related analyses was not significant. Pathway analyses further linked higher RGL2 expression to complement/coagulation and innate immune-inflammatory programs in AD-related analyses. These findings suggest a model in which POD and AD may partially intersect through aging-vulnerable inhibitory-neuron states and identify RGL2 as a prioritized candidate for downstream mechanistic investigation. - Source: PubMed
Publication date: 2026/08/29
Fan WeiHan ShuaiWang CunjingGao Ju - Epstein-Barr virus-associated gastric cancer (EBVaGC) is a distinct molecular subtype of gastric cancer, but reliable biomarkers linking EBV-related biology, prognosis, and microenvironmental remodeling remain limited. - Source: PubMed
Jin LufeiJiang ManPan YubinWang YanZhang LeiChen Wujie - Renal fibrosis represents the final common pathway of chronic kidney disease (CKD); however, both its definitive diagnostic biomarkers and the principal cellular mediators driving its progression remain incompletely characterized. - Source: PubMed
Publication date: 2026/08/24
Fazeli Seyed AmirhosseinJavanmard Amir-Reza