STOM antibody
- Known as:
- STOM (anti-)
- Catalog number:
- 38857
- Product Quantity:
- USD
- Category:
- -
- Supplier:
- Signalway
- Gene target:
- STOM antibody
Related genes to: STOM antibody
- Gene:
- STOM NIH gene
- Name:
- stomatin
- Previous symbol:
- EPB7, EPB72
- Synonyms:
- BND7
- Chromosome:
- 9q33.2
- Locus Type:
- gene with protein product
- Date approved:
- 1992-09-14
- Date modifiied:
- 2016-10-05
Related products to: STOM antibody
Related articles to: STOM antibody
- Anaerobic membrane bioreactors (AnMBRs) are considered a promising technology for sustainable wastewater treatment. However, membrane fouling remains a major obstacle to their practical application. Meanwhile, efficient phosphorus recovery through vivianite crystallization is essential for improving the resource recovery potential of AnMBRs. This study investigated the use of magnetic biochar (MBC) as a dual-functional mediator to simultaneously address these two challenges. The results showed that MBC efficiently mitigated membrane fouling, reducing the average fouling rate from 1.11-1.74 kPa/d to 1.07-1.28 kPa/d. This improvement was mainly attributed to the regulation of sludge extracellular polymeric substances (EPS). Specifically, the tightly bound EPS content in the MBC-AnMBR was 22 % lower than that in the control, resulting in the formation of a loose and less adhesive fouling layer. More importantly, MBC acted as a magnetic mediator that promoted vivianite crystallization. During the initial 5 days of operation, the ion activity product and saturation index in the MBC-AnMBR were significantly higher than those in the control system, accelerating vivianite nucleation. Solid-phase elemental mass balance analysis demonstrated a high theoretical magnetic separation potential for phosphorus and iron, with phosphorus recovery reaching 98 % under a low magnetic field intensity, compared with only 55 % in the control system. A synergistic effect between membrane fouling mitigation and vivianite recovery was observed, highlighting MBC as a novel and practical strategy for the simultaneously optimizing AnMBR performance and phosphorus resource recovery, thereby advancing the sustainable operation of AnMBRs. - Source: PubMed
Publication date: 2026/08/09
Yang Feng-AiDong YingChang JifeiChen RongWang XinLiu JunchengLobo Fernanda LeiteStom Devard ILi Nan - Sepsis is a life-threatening organ dysfunction caused by a dysregulated immune response to infection. Ammonia-induced cell death (AICD) is a novel form of cell death, and its association with sepsis remains unclear. This study aims to identify key genes involved in AICD in pediatric sepsis. - Source: PubMed
Publication date: 2026/07/16
Huang Yong FengLi Xing ChuanJiang Ran - ObjectiveThis study investigates how task priority, task difficulty, and their interaction influence task-switching decisions in a complex multitasking environment, where operators manage multiple supervisory tasks distributed across displays.BackgroundThe Strategic Task Overload Management (STOM) model posits that task priority and difficulty influence task-switching behavior, but empirical research has yielded inconsistent results.MethodParticipants performed four concurrent supervisory tasks, each shown on a separate display, using a simulated unmanned aerial vehicle monitoring platform. Task priority was manipulated through performance-based rewards, and the difficulty of the target detection task varied across four levels.ResultsParticipants were more likely to switch to the target detection task and dwelled longer on it when it was high-priority. Increased task difficulty led to lower overall switching frequency and longer dwell time on the more difficult task. Manipulations of priority and difficulty mutually reinforced perceptions of each other and amplified each other's impact on task-switching behaviors.ConclusionTask priority has a significant impact on task-switching behaviors in complex multitasking scenarios. The impact of task difficulty on switching behaviors is more pronounced at the global level than at the local level, where a threshold effect is observed. The two attributes mutually reinforce each other's impact.ApplicationThis study provides implications for refining the STOM model and offers insights into designing interventions that support effective task switching. - Source: PubMed
Publication date: 2026/07/05
Liu YangGao QinLi XinyunYang Xinyue - Diabetic retinopathy (DR) is a leading cause of blindness, but its underlying molecular mechanisms remain poorly understood. This study aimed to investigate the role of STOM in DR-related retinal angiogenesis and to elucidate its potential signaling mechanism. - Source: PubMed
Publication date: 2026/06/11
Chi JingyiLiu XinyuSong WeichenChen WenXue XiaoxiaoZhang YongqiZhu Wenwen - Inflammation-induced pulmonary fibrosis is an irreversible and severe complication that leads to persistent decline in lung function and increased mortality; however, its early pathogenesis is still unclear. This study aimed to systematically elucidate the initiation mechanism of pulmonary fibrosis in the early stages of inflammation. By integrating multi-omics data and animal models, we found that lung exhibits stronger immune amplification and more severe mitochondrial dysfunction in comparison with other organs during inflammation, consequently fibrotic signaling is initiated in the acute phase. Mitochondria-related gene analysis identified six key genes (Bcl2l1, Gsr, Msrb3, AA467197, Stom, and Sod2) involved in the regulation of reactive oxygen species (ROS) metabolism, which were closely associated with clinical outcomes in sepsis. Temporal data and TNF-α/IL-1β intervention experiments revealed that these cytokines are persistently overexpressed in septic lungs, serving as critical drivers of ROS activation. In vitro assays further confirmed that ROS overload directly induces cellular damage and functional reprogramming of fibroblasts. Through bulk and single-cell transcriptomic analyses, we elucidated the alteration of intercellular communication between immune and parenchymal cells, and identified Col13a1 fibroblasts as a key subpopulation with the capability to drive fibrotic remodeling. In conclusion, sustained TNF-α/IL-1β signaling in septic lungs exacerbates ROS accumulation, thereby driving aberrant fibroblast repair and initiating pulmonary fibrosis, in which Col13a1 fibroblasts represent the major profibrotic subpopulation. Thus, the early inhibition of TNF-α/IL-1β expression, suppression of ROS accumulation, and regulation of Col13a1 fibroblast activation may provide an effective therapeutic strategy for sepsis. - Source: PubMed
Publication date: 2026/05/16
Zhong ZhaoqianWu KanWang JunhaoChen GuimingLuo HaihuaChen GuangqinSun ChangGuo DanyanLi LeyiLi LeiJiang Yong