Kctd12 Antibody - C-terminal region (ARP35402_P050)
- Known as:
- Kctd12 Antibody - C-terminal region (ARP35402_P050)
- Catalog number:
- arp35402_p050
- Product Quantity:
- USD
- Category:
- -
- Supplier:
- Aviva Systems Biology
- Gene target:
- Kctd12 Antibody - C-terminal region (ARP35402_P050)
Ask about this productRelated genes to: Kctd12 Antibody - C-terminal region (ARP35402_P050)
- Gene:
- KCTD12 NIH gene
- Name:
- potassium channel tetramerization domain containing 12
- Previous symbol:
- C13orf2
- Synonyms:
- KIAA1778, PFET1
- Chromosome:
- 13q22.3
- Locus Type:
- gene with protein product
- Date approved:
- 2001-03-14
- Date modifiied:
- 2016-10-05
Related products to: Kctd12 Antibody - C-terminal region (ARP35402_P050)
Related articles to: Kctd12 Antibody - C-terminal region (ARP35402_P050)
- R.-y. Ye, X.-y. Kuang, H.-j. Zeng, N. Shao, Y. Lin, and S.-m. Wang, "KCTD12 Promotes G1/S Transition of Breast Cancer Cell through Activating the AKT/FOXO1 Signaling," Journal of Clinical Laboratory Analysis 34, no. 8 (2020): e23315, https://doi.org/10.1002/jcla.23315. The above article, published online on 24 March 2020 in Wiley Online Library (wileyonlinelibrary.com), has been retracted by agreement between the journal Editor-in-Chief, Rong Fu; and Wiley Periodicals LLC. A third party raised concerns on PubPeer [1] that the sh#1 image in Figure 4E had later been duplicated in another article by different authors as well as its erratum [Sheng et al. 2021 (https://doi.org/10.21037/atm-21-1069) and (https://doi.org/10.21037/atm-2022-43)]. The third party also reported that the sh#2 image in Figure 4E had been duplicated in another article byanother author group [Peng et al. 2022 (https://doi.org/10.1155/2022/9631036)]. While both of these articles were published subsequently, the images include rotations and additional field of view which indicate the data were derived from the same images, though each article describes different experimental conditions. The authors responded to an inquiry by the publisher, but the explanation and original data provided did not adequately explain how data had been inadvertently shared between all three author groups. The retraction has been agreed to because the evidence of data shared between three articles and the lack of a sufficient explanation has compromised the editors' confidence that the results presented in this article are accurate. The authors disagree with the retraction. References: [1] René Aquarius, Comments on "KCTD12 promotes G1/S transition of breast cancer cell through activating the AKT/FOXO1 signaling," PubPeer, August 2024. https://pubpeer.com/publications/B624C6A5FC4650FE0F16C93DDBF4D9. - Source: PubMed
Publication date: 2026/04/07
- () infections are prevalent among school-age children, and an increasing number of patients are developing resistance to azithromycin (AZM). However, effective biomarkers for diagnosing AZM resistance are currently lacking. This study aimed to identify potential biomarkers for AZM resistance in infections by analyzing serum exosomes. - Source: PubMed
Publication date: 2025/12/10
Fan HuifengHuang FengChen ChenRen QiaoliZhou JingYang DiyuanLu Gen - Cancer-associated fibroblasts (CAFs) promote hepatocellular carcinoma (HCC) progression. Identifying their cellular origin and molecular determinants may help to inform molecular targeted therapy for HCC. Identifying the tissue distribution of CAFs in HCC to indirectly reflect their cellular origin may provide a histological basis for further exploration of their cellular origin and molecular determinants. - Source: PubMed
Publication date: 2025/11/30
Yin ZeliWang QiuXiangXu ZheGuo KunWang Liming - Non-small cell lung cancer (NSCLC) is the predominant histological subtype of lung cancer, whose diverse genomic landscape complicates prognosis and outcome prediction. In this context, immunogenic cell death (ICD), a distinct mechanism of cell death, may exert important antitumor effects. However, the specific role of ICD in NSCLC has not been clarified, and there is no suitable method for using ICD to achieve the treatment and prognosis assessment of NSCLC. The purpose of the current research is to develop a new approach to predict the survival prognosis and response to chemotherapy and targeted therapy in patients with NSCLC. - Source: PubMed
Publication date: 2025/08/26
Sun YichenChen HaoWang ZhaoyangJiao RuiZehentmayr FranzTabbò FabrizioWu ChengyangZhang TaoYan HanyuWang JianYan Xiaolong - Sexual selection is widely recognized as a key driver of evolutionary processes; However, research on this topic in bats remains limited despite their considerable ecological importance. The flat-headed bat , a small bamboo-roosting species, typically forms either polygynous groups (one male with multiple females) or male-only groups. Previous studies have shown that males roosting with females experience significantly higher reproductive success, despite the absence of documented morphological sexual dimorphism. In this study, we constructed a reference transcriptome for using RNA-seq, combining assembly and transcript-guided assembly based on a closely related species. We compared gene expression profiles of brain tissue between solitary males and polygynous males from the Chongzuo region in Guangxi, China. A total of 107 differentially expressed genes (DEGs) were identified, including , , and , etc., upregulated in polygynous males, while , COTL1 and were upregulated in solitary males. These results suggest that polygynous males may possess superior survival and reproductive potential compared to solitary males. KEGG enrichment analysis revealed that polygynous males were significantly enriched in pathways including "lysosome" and "endocrine and other factor-regulated calcium reabsorption". GO enrichment analysis also indicated various differences between the two male groups. Our results demonstrate distinct gene expression patterns between the two male groups, offering preliminary molecular evidence for adaptive evolution driven by sexual selection in . - Source: PubMed
Publication date: 2025/09/10
Lv ChuyuZhang LibiaoLi KeHua Panyu