GABRB2 antibody - N-terminal region (ARP35338_T100)
- Known as:
- GABRB2 (anti-) - N-terminal region (ARP35338_T100)
- Catalog number:
- arp35338_t100
- Product Quantity:
- USD
- Category:
- -
- Supplier:
- Aviva Systems Biology
- Gene target:
- GABRB2 antibody - N-terminal region (ARP35338_T100)
Related genes to: GABRB2 antibody - N-terminal region (ARP35338_T100)
- Gene:
- GABRB2 NIH gene
- Name:
- gamma-aminobutyric acid type A receptor beta2 subunit
- Previous symbol:
- -
- Synonyms:
- -
- Chromosome:
- 5q34
- Locus Type:
- gene with protein product
- Date approved:
- 1994-04-29
- Date modifiied:
- 2016-02-04
Related products to: GABRB2 antibody - N-terminal region (ARP35338_T100)
Related articles to: GABRB2 antibody - N-terminal region (ARP35338_T100)
- Histone deacetylases (HDACs) regulate neuroprotection; however, Trichostatin A (TSA), an HDAC inhibitor, lacks clear molecular mechanisms and core targets in Alzheimer's disease (AD), limiting clinical translation. This study aimed to decipher TSA's AD-regulating network, screen core genes, and support AD early diagnosis and multi-target therapies. - Source: PubMed
Publication date: 2026/04/20
Ou ChangzeChen BinbinDeng JunLong Huajun - Accumulating evidence highlights the critical role of palmitoylation in tumorigenesis, including the regulation of oncogenic signaling pathways, metabolic reprogramming, and immune evasion mechanisms. This study was designed to systematically investigate the prognostic significance of palmitoylation-related genes in glioblastoma multiforme (GBM). - Source: PubMed
Publication date: 2026/01/27
Ji YujieDai XiangyuJiang LeiZeng HaixinCai ZhengLi Bing - Dravet syndrome (DS) is a developmental and epileptic encephalopathy caused mainly by SCN1A variants, several other genes have been implicated in DS-like phenotype. - Source: PubMed
Publication date: 2026/01/30
Tian XiaojuanCheng MiaomiaoYang YingZeng QiChen YiLiu AijieYang XiaolingZhang JingTan QuanzhenLiu WenweiWang TingOuyang ShijiaLiu ChanghaoWu YeJiang YuwuZhang Yuehua - - Source: PubMed
Chen Chih-Ping - Pathogenic variants in γ-aminobutyric acid type A (GABA) receptor genes have been associated with a wide spectrum of neurological disorders. We aimed to delineate the clinical trajectories associated with gain-of-function (GoF) and loss-of-function (LoF) variants in GABRB2 and GABRB3, and to develop a risk-prediction model for gross motor dysfunction based on age at seizure onset. - Source: PubMed
Publication date: 2026/01/28
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