ADRB2 293T Cell Transient Overexpression Lysate(Denatured)
- Known as:
- ADRB2 293T Cell Transient Overexpression Lysate(Denatured)
- Catalog number:
- H00000154-T01
- Product Quantity:
- 100 uL
- Category:
- -
- Supplier:
- Abno
- Gene target:
- ADRB2 293T Cell Transient Overexpression Lysate(Denatured)
Related genes to: ADRB2 293T Cell Transient Overexpression Lysate(Denatured)
- Gene:
- ADRB2 NIH gene
- Name:
- adrenoceptor beta 2
- Previous symbol:
- ADRB2R
- Synonyms:
- ADRBR, BAR, B2AR
- Chromosome:
- 5q32
- Locus Type:
- gene with protein product
- Date approved:
- 1990-09-10
- Date modifiied:
- 2016-10-05
- Gene:
- HTR1A NIH gene
- Name:
- 5-hydroxytryptamine receptor 1A
- Previous symbol:
- ADRB2RL1, ADRBRL1
- Synonyms:
- 5-HT1A
- Chromosome:
- 5q12.3
- Locus Type:
- gene with protein product
- Date approved:
- 1986-01-01
- Date modifiied:
- 2016-02-04
Related products to: ADRB2 293T Cell Transient Overexpression Lysate(Denatured)
Related articles to: ADRB2 293T Cell Transient Overexpression Lysate(Denatured)
- Depression and anxiety are the most common mental health disorders. Though they affect people at any age and occur more often in females, the pathophysiological changes under these conditions are less investigated. In the present study, we examined the effects of age and stress on depression- and anxiety-related behaviors in female mice. Saccharin preference and the open field test were carried out before and after chronic unpredictable stress in 4-, 14- and 25-month-old female mice. After behavioral tests, mRNA levels of monoamine receptors in the hippocampus were measured by real-time RT-PCR. Chronic unpredictable stress decreased saccharin preference in 4-, 14- and 25-month-old mice and the time spent in the center in the open field test in 25-month-old mice. For monoamine receptors, analysis of variance revealed significant effects of age on mRNA levels of Htr1a, Htr2a, Htr6, Adra1a, Adrb2, and Adrb3, significant effects of stress on mRNA levels of Htr4, Adra2c, Adrb1, and Adrb2, and interactions of age × stress on mRNA levels of Htr1a, Htr5b, Adra1d, Adra2a, Adra2c, and Adrb1. Chronic unpredictable stress decreased mRNA levels of Htr4, Htr5b, Adra2c, and Adrb1 in 4-month-old female mice. Correlations were observed between saccharin preference and mRNA levels of Htr4, Htr5b, Htr6, Adra1d, Adra2a, and Adra2c in 4-month-old mice and between the time spent in the center in the open field test and mRNA levels of Htr1b in 4-month-old mice, Htr3a, Htr7, and Adrb2 in 14-month-old mice, and Drd2 in 4- and 14-month-old mice. Our findings support that stress induces depression- and anxiety-related behaviors and the expression of hippocampal monoamine receptors in an age-dependent manner in female mice. - Source: PubMed
Publication date: 2023/07/17
Zhou HanWang KaixinXu ZhichengLiu DunjiangWang YamengGuo Ming - Postherpetic neuralgia (PHN) is one of the most common complications of herpes zoster (HZ). Heritable factors have been found to play a role in various clinical pain symptoms. However, the effect of gene variability on the susceptibility of PHN remains poorly understood. - Source: PubMed
Xing XiufangBai YongyuSun KaiChen QunshanHuang HaoQiu WeidongYan Min - To investigate the mechanism underlying the effects of Shichangpu (Rhizoma Acori Tatarinowii) on attention deficit hyperactivity disorder (ADHD). - Source: PubMed
Feng KaiZhao YingRen XinxinChou YiqianYu QingyangLi LingWang Junhong - : To evaluate the association between polymorphisms in genes and comorbid presence of arthralgias and TMD.: This is a case-control study. The groups formed were individuals with chronic arthralgia and 1) myofascial pain (n = 42); 2) articular (n = 16); 3) multiple diagnoses (n = 69); 4) with TMD and without some other arthralgia (n = 16); 5) without TMD but with pain in other joints (n = 82); and 6) a control group (n = 72). SNPs in , and genes were investigated.: The CT genotype for the (rs9332377) gene was associated with the absence of myofascial pain ( = .05). In the (rs1042713) gene, the AA genotype was associated with the absence of myofascial pain ( = .03).: This study supports the hypothesis that alterations in the , and genes influence the presence of chronic pain and TMD. - Source: PubMed
Publication date: 2019/07/02
Bonato Letícia LadeiraQuinelato Valquiriade Felipe Cordeiro Patrícia CataldoVieira Alexandre RezendeGranjeiro José MauroTesch RicardoCasado Priscila Ladeira