IP-10 Antibody, mAb, Mouse
- Known as:
- IP-10 Antibody, mAb, Mouse
- Catalog number:
- a00163-500
- Product Quantity:
- USD
- Category:
- -
- Supplier:
- Genscript
- Gene target:
- IP-10 Antibody mAb Mouse
Related genes to: IP-10 Antibody, mAb, Mouse
- Gene:
- BBIP1 NIH gene
- Name:
- BBSome interacting protein 1
- Previous symbol:
- NCRNA00081
- Synonyms:
- bA348N5.3, BBIP10, BBS18
- Chromosome:
- 10q25.2
- Locus Type:
- gene with protein product
- Date approved:
- 2008-09-02
- Date modifiied:
- 2016-10-05
- Gene:
- CXCR3 NIH gene
- Name:
- C-X-C motif chemokine receptor 3
- Previous symbol:
- GPR9
- Synonyms:
- CKR-L2, CMKAR3, IP10-R, MigR, CD183
- Chromosome:
- Xq13.1
- Locus Type:
- gene with protein product
- Date approved:
- 1994-11-01
- Date modifiied:
- 2016-10-05
- Gene:
- MED24 NIH gene
- Name:
- mediator complex subunit 24
- Previous symbol:
- THRAP4, CRSP4
- Synonyms:
- TRAP100, KIAA0130, DRIP100, CRSP100, MED5
- Chromosome:
- 17q21.1
- Locus Type:
- gene with protein product
- Date approved:
- 2004-01-30
- Date modifiied:
- 2016-10-05
- Gene:
- PPIAP10 NIH gene
- Name:
- peptidylprolyl isomerase A pseudogene 10
- Previous symbol:
- PPIAL2, PPIP10
- Synonyms:
- CRP
- Chromosome:
- 20q13.2
- Locus Type:
- pseudogene
- Date approved:
- 1998-10-12
- Date modifiied:
- 2017-08-21
- Gene:
- PRPF40A NIH gene
- Name:
- pre-mRNA processing factor 40 homolog A
- Previous symbol:
- FNBP3
- Synonyms:
- FLJ20585, FBP11, HYPA, NY-REN-6, HIP10, FBP-11, FLAF1, Prp40
- Chromosome:
- 2q23.3
- Locus Type:
- gene with protein product
- Date approved:
- 2001-08-28
- Date modifiied:
- 2018-11-19
Related products to: IP-10 Antibody, mAb, Mouse
Related articles to: IP-10 Antibody, mAb, Mouse
- Palmoplantar inflammatory dermatoses, including hyperkeratotic palmoplantar eczema (HPE), palmoplantar psoriasis (PP) and palmoplantar pustulosis (PPP), present overlapping clinical features that complicate diagnosis and limit targeted therapy. This prospective observational study evaluated whether minimally invasive tape strip sampling, combined with targeted proteomics, can distinguish these conditions at a molecular level. Adults with HPE (n = 14), PP (n = 10), and PPP (n = 12) were enrolled, and tape strips from esional and non-lesional skin were analysed using the Olink Reveal panel (1034 proteins) with validation by MSD multiplex immunoassays. Differentially expressed proteins, pathway enrichment analysis, and protein-protein interaction analyses were conducted across the three diseases, and longitudinal analyses were conducted in a subset of patients with PP. Protein expression patterns showed partial separation of PPP from PP and HPE, whereas PP and HPE overlapped. Lesional samples across all three diagnoses exhibited increased expression of inflammatory proteins compared with non-lesional skin. PPP showed a distinct molecular profile from PP and HPE, with enriched pathways related to neutrophil degranulation, innate immune activation, and Th17-associated signalling networks centred on IL-17A and CXCL8. No proteins were significantly differentially expressed between PP and HPE lesions. PP and HPE showed overlapping interferon-associated chemokines CXCL10-11, consistent with a shared inflammatory module. Inflammatory protein levels decline with clinical improvement in longitudinal PP samples. Overall, tape strip-based targeted proteomics is a feasible approach for palmoplantar diseases, revealing a distinct inflammatory signature that differentiates PPP from PP and HPE, although discrimination between PP and HPE remains limited. - Source: PubMed
Johansen Mila BAltintas SuleBronze MarianaWoetmann AndersZachariae ClausNäslund-Koch CharlotteLøvendorf Marianne BSkov Lone - Cancer progression involves not only uncontrolled proliferation but also the strategic entry of tumour cells into reversible (quiescent) or irreversible (senescent) states of cell cycle arrest (G0). These states can give rise to rare persister-like cancer cells that survive hostile tumour microenvironment conditions, facilitating drug resistance, metastasis and disease relapse. Despite their importance, identifying and understanding the mechanisms regulating these cell populations remains challenging. - Source: PubMed
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