Monkey apelin 36(AP36) ELISA kit
- Known as:
- Monkey apelin 36(AP36) Enzyme-linked immunosorbent assay test reagent
- Catalog number:
- e09a1567
- Product Quantity:
- USD
- Category:
- -
- Supplier:
- Blue gene shanghai
- Gene target:
- Monkey apelin 36(AP36) ELISA kit
Related genes to: Monkey apelin 36(AP36) ELISA kit
- Gene:
- APLN NIH gene
- Name:
- apelin
- Previous symbol:
- -
- Synonyms:
- apelin, XNPEP2
- Chromosome:
- Xq26.1
- Locus Type:
- gene with protein product
- Date approved:
- 2004-06-01
- Date modifiied:
- 2016-10-05
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- Degenerative diseases of the musculoskeletal system, including sarcopenia, osteoarthritis, and osteoporosis, represent a cluster of age-associated conditions pathologically linked to chronic inflammation, oxidative stress imbalance, and endocrine dysregulation. These musculoskeletal diseases have emerged as a critical public health concern in aging populations worldwide, significantly compromising functional mobility and overall quality of life. Apelin, an endogenous ligand for the G protein-coupled receptor apelin receptor (APJ), constitutes a crucial component of the apelin/APJ signaling system that exhibits extensive distribution throughout human physiological systems. The multifaceted regulatory roles of apelin in cellular homeostasis, tissue remodeling, and inflammatory responses position it as a promising therapeutic target for these degenerative conditions. This review mainly discusses the effects of apelin on the occurrence and development of degenerative diseases in different musculoskeletal systems, aiming to establish a scientific foundation for future investigations and to identify novel therapeutic strategies for these conditions. - Source: PubMed
Publication date: 2025/08/02
Xu JinkunLi HengzhenSun JianfengLiu GaomingYao YumingYuan DongliangXiao WenfengLi Yusheng - - Source: PubMed
Publication date: 2026/07/22
Guo JiwuWu WenzhangQi YujingGuo LingyunMao JieLi Yumin - Apelin and Elabela are endogenous ligands of the apelin receptor (apelin receptor/APJ), a GPCR involved in cardiovascular regulation and body fluid homeostasis. The human receptor contains a conserved disulfide bridge linking the N-terminal domain to extracellular loop 3 (ECL3) via Cys19 and Cys281, forming a structural constraint analogous to a fourth extracellular loop ('ECL4'). In the chemokine receptor family, this motif plays a critical role in ligand engagement and receptor activation. - Source: PubMed
Publication date: 2026/07/03
Bonef ChloéBouhsine HelaHuynh CarineChaigneau MarionDubois StevenGarcia-Marcos MikelLlorens-Cortes CatherineServent DenisRobin PhilippeNozach HervéAlves IsabelIturrioz Xavier - Apelin is a secreted peptide hormone involved in vasodilation, fluid homeostasis, and angiogenesis, and is therefore considered an important regulator of cardiovascular physiology. The APLN T-1860C (rs56204867) polymorphism is a genetic variation in the promoter region of the apelin gene (APLN) that is associated with increased susceptibility to cardiovascular risk factors. This study aimed to elucidate the relationship between the apelin gene -1860T>C single-nucleotide polymorphism, plasma apelin levels, and the risk of coronary artery disease (CAD) in a Syrian population. - Source: PubMed
Publication date: 2026/06/26
Abd-Alkareem Maisaa HassanShibli Hussam Eddin MohammedAlquobaili Faizeh Ali - Exerkines are bioactive molecules released in response to physical exercise and are considered important mediators of systemic adaptations. While previous research has largely focused on the effects of exercise modalities, the role of exercise intensity in regulating exerkine responses remains unclear. This narrative review summarizes findings on the effects of different exercise intensities on nine circulating exerkines with sufficient available data in healthy populations, including irisin, follistatin-like 1, myostatin, fibroblast growth factor 21, follistatin, leptin, adiponectin, apelin and brain-derived neurotrophic factor, without systematically covering all known exercise-responsive molecules. Given the narrative design of this review, the findings should be interpreted as descriptive and hypothesis-generating rather than as definitive evidence of intensity-dependent effects. The included studies show that acute exercise is associated with changes in several exerkines, with some direct within-study comparisons reporting larger responses under higher-intensity exercise conditions, whereas others exhibit increases, decreases, or no measurable changes across intensities. In contrast, studies examining chronic exercise interventions report changes in some studies and no measurable differences in others. Overall, the current evidence in this review suggests that exercise intensity may influence exerkine responses under some conditions, particularly during acute exercise, although the available findings remain limited and inconsistent across studies. - Source: PubMed
Publication date: 2026/06/10
Zhao YanqiWang TutuZhang XinuanWang WangeFu YuLaher IsmailLi Shunchang