Canine acylated ghrelin (AG) ELISA kit
- Known as:
- Canine acylated ghrelin (AG) Enzyme-linked immunosorbent assay test reagent
- Catalog number:
- e08a1308
- Product Quantity:
- USD
- Category:
- -
- Supplier:
- Blue gene shanghai
- Gene target:
- Canine acylated ghrelin () ELISA kit
Related genes to: Canine acylated ghrelin (AG) ELISA kit
- Gene:
- GHRL NIH gene
- Name:
- ghrelin and obestatin prepropeptide
- Previous symbol:
- -
- Synonyms:
- MTLRP, ghrelin, obestatin
- Chromosome:
- 3p25.3
- Locus Type:
- gene with protein product
- Date approved:
- 2006-01-05
- Date modifiied:
- 2016-08-10
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- This study examined the intestinal effects of polyethylene (PE; size 46.6 ± 11.3 μm) and polystyrene (PS; size 52.5 ± 11.5 μm) microparticles at three different concentrations (0.5%, 2%, and 5%) on juvenile rainbow trout () using an integrative approach combining transcriptomics, proteomics, and microbiome analysis. After a six-week experiment, molecular analyses identified concentration-dependent transcriptional responses, including downregulation of genes involved in ion exchange () and appetite regulation (), alongside upregulation of immune- and stress-related genes (, ), particularly at higher concentrations. Proteomic profiling showed a more pronounced effect of PS compared to PE, including suppression of digestive enzymes, disruption of lipid and energy metabolism, and activation of proteins associated with oxidative stress and immune responses. Microbiome analysis confirmed plastic-induced dysbiosis, characterised by reduced microbial diversity, depletion of beneficial taxa (e.g., , ), and shifts in short-chain fatty acid-producing bacteria. By integrating multiple levels of biological organisation, this study provides new insights into how microplastics interfere with intestinal physiology, beyond acute toxicity. The findings emphasise the relevance of polymer type and exposure concentration in shaping biological responses and highlight the vulnerability of freshwater species to environmental microplastics. - Source: PubMed
Publication date: 2026/08/21
Hodkovicova NikolaHollerova AnetaGebauer JanCrhanova MagdalenaStastny KamilSvobodova ZdenkaFaldyna Martin - Systemic interactions between the liver and pancreas are critical for regulating lipid and carbohydrate homeostasis. These organs are also deeply interdependent in pathological conditions such as insulin resistance, where the liver fails to respond appropriately to insulin stimulation, leading to dysfunction in both organs. To investigate the crosstalk between these tissues using human models, we developed an advanced organ-on-chip (OoC) device hosting human induced pluripotent stem cell-derived liver and pancreatic tissues. The proteomic response of pancreatic-like cell spheroids cocultured with hepatocyte-like cells was characterized. We observed that the presence of liver tissue enhanced the synthesis of proteins associated with extracellular matrix remodelling, inflammatory responses, and pro-apoptotic signalling in pancreatic cells. Conversely, monocultures of pancreatic cells exhibited a higher abundance of proteins involved in fatty acid metabolism, glycolysis, and the citrate cycle. Key pancreatic markers (GCG, TPH1, SCGN3, SCGN, CHGA, CHGB) were upregulated in monocultures, while no reduction was observed in the secretion of pancreatic hormones (IGF2, GHRL, and C-peptide). Overall, our results suggest a metabolic switch in cocultures, accompanied by cellular stress and moderate alterations in pancreatic secretion. These findings provide insights into liver-pancreas crosstalk and may guide the design of future multicellular OoC models. - Source: PubMed
Essaouiba AmalVicogne JérômeStella AlexandreDanoy MathieuGonzalez de Peredo AnneLegallais CécileSakai YasuyukiJellali RachidLeclerc Eric - Taste perception, taste preferences, and food preferences are three interrelated constructs underlying food-related behavior and contribute to individual dietary choices. Both genetic and environmental factors contribute to shaping these traits. Taste perception refers to interindividual differences in biological sensitivity to basic taste modalities. Taste preferences represent the hedonic evaluation of taste modalities, including salty, sweet, bitter, sour, or umami, and fatty tastes, and are closely related to, but distinct from, taste perception. Recent evidence in sensory genetics has identified numerous single-nucleotide variants (SNVs) associated with interindividual differences in both taste perception and taste-modality-related responses. Essential genes include and for sweet taste, and for umami, for bitter taste, for fat perception, and epithelial sodium channel (ENaC) subunits for salt sensitivity. Regarding broader food preferences, variants in genes involved in metabolic regulation and reward processing-such as , , , , and -also contribute to food-related behaviors and choice patterns. Health status and genetic variants may also influence taste perception and food preferences. This review summarizes current evidence on the genetic determinants of taste perception, taste modality preferences, and food liking, emphasizing the multifactorial nature of food-related behaviors and the role of genetic variability in shaping individual differences in dietary choices. - Source: PubMed
Publication date: 2026/08/04
Miller-Kasprzak EwaBogdański Paweł - This study aims to investigate the influence of age and gender on the efficacy of metabolic surgery in patients with obesity with type 2 diabetes mellitus (T2DM). - Source: PubMed
Publication date: 2026/07/27
Ma BoDong YangfanLi TaoWang JunMa HuaShang Shaohua - This study evaluated the effects of live and heat-killed HP7 on intestinal motility, host physiological responses, and gut microbiota in a mouse model of delayed intestinal transit. BALB/c mice were treated with live HP7 (HP7L), heat-killed HP7 (HP7K), or bisacodyl following loperamide administration. HP7L and HP7K improved intestinal motility-related parameters, including fecal output, stool consistency, fecal moisture content, and charcoal meal transit. HP7 treatment also modulated circulating gastrointestinal hormones and inflammatory cytokines, with changes in serotonin, ghrelin, peptide YY, IL-6, and IL-17A. In intestinal tissues, HP7 regulated the expression of genes related to neuroendocrine signaling, water transport, mucus production, and tight junction integrity, including and . Histological analysis further showed reduced colonic tissue alterations and improved structural parameters. Microbiota analysis revealed that HP7L exerted broader effects on microbial community structure, whereas HP7K induced more selective taxonomic changes. Predicted functional profiling suggested that both HP7L and HP7K were associated with shifts in carbohydrate metabolism-related pathways, with HP7K showing additional predicted enrichment of pathways related to redox-associated metabolism, lipid metabolism, and microbial substrate utilization. These findings suggest that heat-killed HP7 retains biological activity and has potential as a postbiotic candidate for regulating intestinal motility. - Source: PubMed
Publication date: 2026/07/01
Gwon HyeonjunKim HyeonjiKim Joo-YunShim Jae-JungLee Jae-Hwan