NADH Oxidase Landscape (Large Format PEP)
- Known as:
- NADH Oxidase Landscape (Large Format PEP)
- Catalog number:
- ab000502
- Product Quantity:
- USD
- Category:
- -
- Supplier:
- Arraybridge
- Gene target:
- NADH Oxidase Landscape (Large Format PEP)
Related genes to: NADH Oxidase Landscape (Large Format PEP)
- Gene:
- ANPEP NIH gene
- Name:
- alanyl aminopeptidase, membrane
- Previous symbol:
- CD13, PEPN
- Synonyms:
- LAP1, gp150, p150
- Chromosome:
- 15q26.1
- Locus Type:
- gene with protein product
- Date approved:
- 1989-02-28
- Date modifiied:
- 2016-10-05
- Gene:
- APLN NIH gene
- Name:
- apelin
- Previous symbol:
- -
- Synonyms:
- apelin, XNPEP2
- Chromosome:
- Xq26.1
- Locus Type:
- gene with protein product
- Date approved:
- 2004-06-01
- Date modifiied:
- 2016-10-05
- Gene:
- C17orf80 NIH gene
- Name:
- chromosome 17 open reading frame 80
- Previous symbol:
- -
- Synonyms:
- HLC-8, MIG3, FLJ20721, SPEP1
- Chromosome:
- 17q25.1
- Locus Type:
- gene with protein product
- Date approved:
- 2006-02-13
- Date modifiied:
- 2016-09-30
- Gene:
- CNDP2 NIH gene
- Name:
- carnosine dipeptidase 2
- Previous symbol:
- PEPA
- Synonyms:
- FLJ10830, CN2, HsT2298, CPGL
- Chromosome:
- 18q22.3
- Locus Type:
- gene with protein product
- Date approved:
- 2004-04-14
- Date modifiied:
- 2017-03-23
- Gene:
- DNPEP NIH gene
- Name:
- aspartyl aminopeptidase
- Previous symbol:
- -
- Synonyms:
- DAP, ASPEP
- Chromosome:
- 2q35
- Locus Type:
- gene with protein product
- Date approved:
- 1998-09-22
- Date modifiied:
- 2016-10-05
Related products to: NADH Oxidase Landscape (Large Format PEP)
(E)_2_Hexen_1_ol formate trans_2_Hexenyl format1 x 5ml Adapter, Large Bore For Rotor F-35-6-301-step Polymer HISTO STAT Alkaline Phosphatase Fast Red kit for IHC staining of Mouse & Rabbit & Rat primary antibodies, 900 slides plus (large size1-step Polymer HISTO-STAT Peroxidase (HRP) Multivalent AEC kit for IHC staining of Mouse, Rabbit & Rat primary antibodies, 900 plus slides (large s1-step Polymer HISTO-STAT Peroxidase (HRP) Multivalent DAB kit for IHC staining of Mouse, Rabbit & Rat primary antibodies, 900 plus slides (large size1.7ml Microtubes, Clear Large Cap, Polypropylene1.7ml Microtubes, Clear Large Cap, Polypropylene1.7ml Microtubes, Clear Large Cap, Polypropylene10x ELISA WASH BUFFER, Product Type Accessory Reagent, Specificity 10x ELISA WASH BUFFER, Target Species , Host N_A, Format Concentrate, Isotypes , Applications E, Clone10x ELISA WASH BUFFER, Product Type Accessory Reagent, Specificity 10x ELISA WASH BUFFER, Target Species , Host N_A, Format Concentrate, Isotypes , Applications E, Clone10x ELISA WASH BUFFER, Product Type Accessory Reagent, Specificity 10x ELISA WASH BUFFER, Target Species , Host N_A, Format Concentrate, Isotypes , Applications E, Clone10x ELISA WASH BUFFER, Product Type Accessory Reagent, Specificity 10x ELISA WASH BUFFER, Target Species , Host N_A, Format Concentrate, Isotypes , Applications E, Clone10x ELISA WASH BUFFER, Product Type Accessory Reagent, Specificity 10x ELISA WASH BUFFER, Target Species , Host N_A, Format Concentrate, Isotypes , Applications E, Clone10x ELISA WASH BUFFER, Product Type Accessory Reagent, Specificity 10x ELISA WASH BUFFER, Target Species , Host N_A, Format Concentrate, Isotypes , Applications E, Clone10X PHOSPHATE BUFFERED SALINE, Product Type Accessory Reagent, Specificity 10X PHOSPHATE BUFFERED SALINE, Target Species , Host N_A, Format Concentrate, Isotypes , Applications IY, Clone Related articles to: NADH Oxidase Landscape (Large Format PEP)
- The etiology of drug-resistant epilepsy (DRE) is multifactorial. A small proportion of affected patients are diagnosed with genetics. Nowadays, specific gene panels and whole-exome sequencing (WES) have increased the opportunities for specific diagnosis and treatments with developments in genetics. In this cohort study, we determined the specific diagnostic value of gene panels and WES analysis in our cases with the diagnosis of DRE. - Source: PubMed
Publication date: 2026/01/09
Kılıç BetülTopçu YaseminAyaz AkifÖzpınar EsraSeyhan SerhatDemir Aslı G ÖPalaz MehmetTuranlı GüzideAydın Kürşad - Rheumatoid arthritis (RA) is a common chronic systemic inflammatory disease that causes musculoskeletal impairments and fatigue. Physical activity is recommended for individuals with RA, and health-enhancing physical activity (HEPA) has been shown to improve health perception and physical fitness in this group. However, the molecular adaptations of skeletal muscle in response to an exercise intervention are still unexplored in individuals with RA. This study aimed to assess the skeletal muscle response to a 2-year HEPA intervention in individuals with RA. - Source: PubMed
Publication date: 2025/10/20
Shorter EmilyOssipova ElenaAlves Estela SantosIdborg HelenaVanluyten JoneKosek EvaZhengye LiuNordgren BirgittaFridén Ceciliavon Walden FerdinandMalm ChristerJakobsson Per-JohanOpava Christina HKorotkova MarinaLundberg Ingrid ELanner Johanna T - The Detumescence Analgesic Plaster (DAP) has been widely used in clinical practice for knee osteoarthritis (KOA) treatment, yet its active ingredients and molecular mechanisms remain incompletely understood. - Source: PubMed
Publication date: 2025/10/20
Li ChunxiaLi WeijieYin YueXiang XiaomeiFu LuWang PingZhang YanqiongXu Haiyu - Lower back pain caused by intervertebral disk degeneration (IDD) is a common problem among middle-aged and older adults. We aimed to identify novel diagnostic biomarkers of IDD and analyze the potential association between key genes and immune cell infiltration. We screened differentially expressed genes (DEGs) related to IDD and gene sets associated with mitochondrial energy metabolism using the Gene Expression Omnibus and GeneCards databases, respectively. Subsequently, we used multiple enrichment analysis methods to determine the biological functionalities of mitochondrial energy metabolism-related differentially expressed genes (MEMRDEGs). Key genes were selected using logistic regression analysis, a support vector machine algorithm, and least absolute shrinkage and selection operator regression analysis to construct an IDD diagnostic model. To obtain further insights, we examined the relationship between key genes and the presence of infiltrating immune cells. We screened 1304 DEGs that exhibited substantial differences in 20 pathways, including the Wnt signaling pathway, between the IDD and control groups. We identified 33 MEMRDEGs and selected 7 key genes (NDUFA6, YWHAZ, DLAT, BDNF, ECI2, ACO1, and ALDH7A1) to construct an IDD diagnostic model. Receiver operating characteristic curve analysis revealed that these genes exhibited high accuracy in assessing IDD risk, with BDNF and DLAT particularly distinguishing between the low- and high-risk IDD groups. Finally, using single-sample gene set enrichment analysis, we identified a relationship between IDD and immune infiltration, with most immune cells showing strong correlations. A significant positive relationship was found between ACO1 and the immune cells, known as immature dendritic cells. These results offer remarkable insights into the mechanisms underlying the occurrence and development of IDD, potentially identifying new opportunities for diagnosis and therapeutic intervention. - Source: PubMed
Lv JianlanWang Zhenwei - Pathological growth of cardiomyocytes known as cardiac hypertrophy (CH). Differential expressions of miRNAs have an immense therapeutic potential against cardiac hypertrophy. The current study aim is to evaluate the therapeutic potential of miRNA-137-3p/383-5p in cardiac hypertrophy by regulation of PGC-1α signaling nexus. Silencing of pro-hypertrophic miRNAs e.g. miR-137-3p and miR-383-5p leads to the restoration of their common target gene PGC-1α in hypertrophic cells. Interestingly, the results of this invivo study showed the cardioprotective effects of these antagomirs. Moreover, PGC-1α associated signaling events e.g. fatty acid oxidation (Cpt1a, Cpt1b), mitochondria membrane potential (MMP), mitochondrial reactive oxygen species (mtROS), oxidative phosphorylation (Ndufa6, Atp5me), apoptosis (Bcl-2, BAX), antioxidants (SOD, GSH, CAT), mitochondrial dynamic (Mfn-2, Drp-1) were significantly restored in the treated groups of miRNA antagomirs. Conclusively, this study uncovers that the pharmacological inhibition of miR-137-3p and miR-383-5p have a potential to rescue from the cardiac hypertrophy by regulation of PGC-1α signaling nexus. - Source: PubMed
Publication date: 2025/06/23
Hussain KhadamKhurram SomiaYousaf MuhammadIshtiaq AyeshaMushtaq IramAli TahirMurtaza Iram