E. coli Host Cell Protein ELISA
- Known as:
- E. coli Host Cell Protein Enzyme-linked immunosorbent assay test
- Catalog number:
- ab000105
- Product Quantity:
- USD
- Category:
- -
- Supplier:
- Arraybridge
- Gene target:
- . coli Host Cell Protein ELISA
Related genes to: E. coli Host Cell Protein ELISA
- Gene:
- A1BG-AS1 NIH gene
- Name:
- A1BG antisense RNA 1
- Previous symbol:
- NCRNA00181, A1BGAS, A1BG-AS
- Synonyms:
- FLJ23569
- Chromosome:
- 19q13.43
- Locus Type:
- RNA, long non-coding
- Date approved:
- 2009-07-20
- Date modifiied:
- 2013-06-27
- Gene:
- A1CF NIH gene
- Name:
- APOBEC1 complementation factor
- Previous symbol:
- -
- Synonyms:
- ACF, ASP, ACF64, ACF65, APOBEC1CF
- Chromosome:
- 10q11.23
- Locus Type:
- gene with protein product
- Date approved:
- 2007-11-23
- Date modifiied:
- 2016-10-05
- Gene:
- A2M NIH gene
- Name:
- alpha-2-macroglobulin
- Previous symbol:
- -
- Synonyms:
- FWP007, S863-7, CPAMD5
- Chromosome:
- 12p13.31
- Locus Type:
- gene with protein product
- Date approved:
- 1986-01-01
- Date modifiied:
- 2018-05-03
- Gene:
- A2ML1 NIH gene
- Name:
- alpha-2-macroglobulin like 1
- Previous symbol:
- CPAMD9
- Synonyms:
- FLJ25179, p170
- Chromosome:
- 12p13.31
- Locus Type:
- gene with protein product
- Date approved:
- 2005-07-20
- Date modifiied:
- 2018-04-17
- Gene:
- A2MP1 NIH gene
- Name:
- alpha-2-macroglobulin pseudogene 1
- Previous symbol:
- A2MP
- Synonyms:
- -
- Chromosome:
- 12p13.31
- Locus Type:
- pseudogene
- Date approved:
- 1991-09-12
- Date modifiied:
- 2016-08-22
Related products to: E. coli Host Cell Protein ELISA
Related articles to: E. coli Host Cell Protein ELISA
- Stroke incidence is declining in older adults but rising in younger populations. Circulating plasma proteins are promising biomarkers of vascular disease, yet their causal role in early-onset stroke (EOS) remains unclear. We used proteome-wide Mendelian randomization (MR) to identify proteins causally associated with EOS and its subtypes. - Source: PubMed
Publication date: 2026/09/15
Huynh Anna LGaynor Brady JCole John WMcArdle Patrick FXu HuichunJern ChristinaPare GuillaumeKittner Steven JChong MichaelStanne Tara MMitchell Braxton D - Myocardial injury activates the complement lectin pathway (LP) in acute coronary syndrome (ACS) and LP inhibition improved cardiac function in experimental studies, suggesting a direct pathogenic role. The clinical consequences of LP activation are insufficiently defined. We investigated how plasma levels of the LP activators mannose-binding lectin (MBL) and ficolin-2 (FCN2) relate to cardiac recovery and prognosis in ACS patients. - Source: PubMed
Publication date: 2026/07/24
Zhong BaojunKing BenMares Razvan GheorghitaArbanasi Emil-MarianYndigegn TroelsEngelbertsen DanielBjörkbacka HarryNilsson JanGoncalves IsabelBlom AnnaSchiopu Alexandru - The first wave of Coronavirus disease 2019 (COVID-19), driven by the global emergence of Severe Acute Respiratory Syndrome Coronavirus 2 (SARS-CoV-2), severely affected Spain with high infection and mortality rates across the country. Although numerous common and rare genetic variants affecting immune-related pathways have been associated with susceptibility to infection and severe disease, the contribution of complement system remains comparatively understudied. - Source: PubMed
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De La Morena-Barrio María EugeniaVan Den Rym AnaEscorial Sanz OlgaCorvillo FernandoGarcía-Sánchez RosarioGonzález-Sánchez LauraMuñoz-Barrera AdriánGonzález-Montelongo RafaelaLorenzo-Salazar José MiguelFlores Carlosde Andrés-Martín AnaRodríguez-Gallego CarlosAllende LuisAlsina LaiaSánchez-Ramón SilviaLópez-Collazo EduardoLópez-Trascasa MargaritaSánchez-Corral PilarPérez de Diego RebecaCorral de la Calle JavierLópez-Lera Alberto - The complement system has an important role in physiological bone healing, as studied mainly in animal models and local bone tissues. However, the role of the complement system in human fractures, particularly at the systemic level, remains insufficiently characterized. This study aimed to investigate the activation levels of the serum complement system during three healing phases (inflammation, repair and remodeling) of normal healing of long bone fractures and in patients diagnosed with fracture non-union. - Source: PubMed
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El-Sherbiny Yasser MAli Youssif MJones ElenaGiannoudis Peter VEl-Jawhari Jehan J - Down syndrome (DS), or trisomy 21 (T21), resulting from an extra copy of chromosome 21, occurs in 1 in 700-1,000 live births. Neuroinflammation is increasingly recognized as a critical contributor to DS neuropathology, although its underlying drivers remain unclear. - Source: PubMed
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