GATA3 Pre-design Chimera RNAi
- Known as:
- GATA3 Pre-design Chimera RNAi
- Catalog number:
- H00002625-R01
- Product Quantity:
- 10 nmol
- Category:
- -
- Supplier:
- Abno
- Gene target:
- GATA3 Pre-design Chimera RNAi
Related genes to: GATA3 Pre-design Chimera RNAi
- Gene:
- GATA3 NIH gene
- Name:
- GATA binding protein 3
- Previous symbol:
- -
- Synonyms:
- HDR
- Chromosome:
- 10p14
- Locus Type:
- gene with protein product
- Date approved:
- 1992-11-03
- Date modifiied:
- 2016-10-05
Related products to: GATA3 Pre-design Chimera RNAi
Related articles to: GATA3 Pre-design Chimera RNAi
- Female infertility affects approximately one in six couples worldwide and disproportionately impacts women aged 30-42 years, yet evidence on which interventions improve clinically meaningful outcomes (live birth and safety) is heterogeneous. - Source: PubMed
Publication date: 2026/06/09
Barroso Alverde Maria JimenaYu SionPascal Pontón DanielBenardete Harari Denise NizaTesone Lasman José Elias - Cervical metastases from breast carcinoma should be considered in the differential diagnosis of uterine lesions in patients with prior breast cancer. Re-biopsy is essential to detect receptor discordance (ER/PR up to 29%) and guide therapy. GCDFP-15, mammaglobin, and GATA3 confirm mammary origin. - Source: PubMed
Publication date: 2026/06/22
Assenza MarioZappalà AdeleNallbani ArmandoWabersich Jacopo - Papillary renal neoplasm with reversed polarity is a rare entity, with its latest definition established only in 2019. Given its rarity, with less than 100 reported cases, further characterization is needed to enhance diagnostic accuracy and inform management strategies. - Source: PubMed
Publication date: 2026/06/17
Wang YuZhang MengsiSu GuangzongZhou Qiao - Higher prevalence of hepatocellular carcinoma (HCC) in men is largely attributed to the sex hormones and behavioral risk factors. This study investigated the effects of male (testosterone) and female (estradiol) hormones on Aurora-A/B kinases, GATA-3, and microRNAs (miR-3941, miR-4500, miR-4742) in HepG2 and Huh-7 cell lines. - Source: PubMed
Publication date: 2026/06/22
Rezaei TaranehAbidi MobinaMohammadi YaserFarzam FarnooshBahreini Elham - Hydrogen sulfide (HS) inhalation injures the respiratory, immune, and nervous systems, altering homeostasis and health, but the molecular mechanisms have been still unclear. In this study, we tested the effects of different concentrations of HS on immune index and enzyme activity of mouse tissues and measured differentially expressed genes (DEGs) in mouse lung tissues by transcriptome sequencing. The results showed that HS significantly increased tumor necrosis factor-alpha (TNF-α) (p < 0.05), catalase (CAT) (p < 0.01) and glutathione peroxidase (GSH-Px) (p < 0.05) activity levels; 80 and 100 ppm HS treatment significantly increased interleukin-1β(IL-1β), interleukin-10 (IL-10) (p < 0.01) and glutathione reductase activation coefficient (GRAC) (p < 0.01) but decreased malonaldehyde (MDA) and superoxide dismutase (SOD) levels (p < 0.05); 100 ppm HS treatment significantly increased glutathione S-transferase (GST) and reactive oxygen species (ROS) levels (p < 0.01). In addition, 963 DEGs were identified in different group. These genes were notably involved in immune responses, DNA modification, enzymatic activity, and cell cycle processes. FGFBP1, GATA3, KLRG1 and TBX21 may be crucial roles in the response to HS exposure. This study provides a reference and many differential genes for the study of hydrogen sulfide damage mechanism. - Source: PubMed
Publication date: 2026/06/22
Gao Jun LeiLi Bai HaoBan Zhi BinLi Li JiaLiang HaoYan Xiao GangXiao ChengZhang Fang Yu