Monkey IL-6 ELISPOT kit, silver staining
- Known as:
- Monkey Interleukin-6 ELISPOT reagent, silver staining
- Catalog number:
- ct130-pb5
- Product Quantity:
- EUR
- Category:
- -
- Supplier:
- U-CyTech biosciences
- Gene target:
- Monkey IL-6 ELISPOT kit silver staining
Related genes to: Monkey IL-6 ELISPOT kit, silver staining
- Gene:
- CEBPB NIH gene
- Name:
- CCAAT enhancer binding protein beta
- Previous symbol:
- TCF5
- Synonyms:
- LAP, CRP2, NFIL6, IL6DBP, C/EBP-beta
- Chromosome:
- 20q13.13
- Locus Type:
- gene with protein product
- Date approved:
- 1991-02-27
- Date modifiied:
- 2018-02-23
- Gene:
- CEBPD NIH gene
- Name:
- CCAAT enhancer binding protein delta
- Previous symbol:
- -
- Synonyms:
- CRP3, CELF, C/EBP-delta, NF-IL6-beta
- Chromosome:
- 8q11.21
- Locus Type:
- gene with protein product
- Date approved:
- 1992-06-24
- Date modifiied:
- 2018-02-23
- Gene:
- ENTPD6 NIH gene
- Name:
- ectonucleoside triphosphate diphosphohydrolase 6
- Previous symbol:
- CD39L2, IL6ST2
- Synonyms:
- NTPDase-6, dJ738P15.3
- Chromosome:
- 20p11.21
- Locus Type:
- gene with protein product
- Date approved:
- 1998-03-20
- Date modifiied:
- 2019-02-28
- Gene:
- IL6 NIH gene
- Name:
- interleukin 6
- Previous symbol:
- IFNB2
- Synonyms:
- IL-6, BSF2, HGF, HSF
- Chromosome:
- 7p15.3
- Locus Type:
- gene with protein product
- Date approved:
- 1986-01-01
- Date modifiied:
- 2017-07-12
- Gene:
- IL6RP1 NIH gene
- Name:
- interleukin 6 receptor pseudogene 1
- Previous symbol:
- IL6RL1
- Synonyms:
- -
- Chromosome:
- 9q22.2
- Locus Type:
- pseudogene
- Date approved:
- 1991-08-18
- Date modifiied:
- 2014-11-19
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- Chronic gingivostomatitis in cats is a debilitating inflammatory disease marked by severe oral pain and systemic immune dysregulation. Although localized oral pathology is well-documented, the systemic peptidomic profile remains largely unclear. - Source: PubMed
Ploypetch SekkarinPornthummawat ApisitAruvornlop PruetthaRoytrakul SittirukJaresitthikunchai JanthimaPhaonakrop NarumonBuamas SupakitSukho Panithi - With advancing age, individuals experience a decline in liver function, an increase in oxidative stress, and diminished mitochondrial efficiency. This research examined the protective effects of curcumin (CUR), in both free and nanoliposomal (CUR-LNP) forms, against D-galactose (D-gal)-induced liver aging in male Wistar rats. - Source: PubMed
Elmorsy Ekramy MAl-Ghafari Ayat BAl Doghaither Huda AShah Syed Sajid HussainSyed AsmaraJan MuhammadEmbaby Eman MElwakeel Eman E - Taraxerol (Tar) is a natural triterpenoid which exhibited anti-inflammatory and neuroprotective properties. Whereas, the roles of Tar in cerebral ischemia-reperfusion injury (CIRI) remain incompletely understood. The research investigated the protective effects and regulatory mechanism of Tar in CIRI. The middle cerebral artery occlusion (MCAO) model was established in vivo. Effects of Tar on brain injury and MCAO rats motor function were detected by HE staining and TUNEL assay. PC12 cells were subjected to oxygen-glucose deprivation/reoxygenation (OGD/R) and irritated with 20, 40, 80, and 160 μM Tar; the intracellular Fe, MDA, and ROS levels were detected utilizing interrelated assay kits. The pro-inflammatory cytokines and ferroptosis-related factors were examined through qRT-PCR and western blot assay. Cell viability and apoptosis of PC12 cells were examined by CCK-8 and flow cytometry assays. The AKT agonist (SC79) was used to investigate the regulatory functions of the AKT/NF-κB pathway in OGD/R-treated PC12 cells. Tar alleviated brain injury and motor function in MCAO rats. Tar eased inflammation, oxidative stress, and ferroptosis in MCAO rats. In vitro, OGD/R exposure reduced cell viability, promoted apoptosis, elevated IFN-γ, TNF-α, IL-6, Fe, MDA, and ROS levels but declined GPX4 and SLC7A11 in PC12 cells. But, the above-mentioned tendency of OGD/R-triggered PC12 cells injury was overturned by Tar. Mechanistically, Tar treatment inactivated the AKT/NF-κB pathway and decreased nuclear translocation of NF-κB in OGD/R-induced PC12 cells. The AKT agonist SC79 reversed the protective effects of Tar in OGD/R-induced PC12 cells. Tar improved CIRI induced inflammation and ferroptosis through regulating the AKT/NF-κB pathway. - Source: PubMed
Yang ZhengfeiZhao Zhenwu - People living with HIV (PLWH) have elevated systemic inflammation, even while adherent to anti-retroviral therapy (ART), contributing to high burden of cardiometabolic diseases. Obstructive sleep apnea (OSA), which promotes inflammation, is more prevalent in this population, yet its contribution to increased morbidity is unclear. - Source: PubMed
Publication date: 2026/08/25
Perryman Alexia NMasso-Silva Jorge AOrr Jeremy ESun XiaoyingJain SoniaDeYoung Pamela NNorby BrynnAncoli-Israel SoniaMalhotra AtulGrant IgorKarris Maile YoungCrotty Alexander Laura EOwens Robert L - Bisphenol S (BPS) is an emerging environmental contaminant that can disrupt oxidative and immune homeostasis in aquatic organisms, whereas myo-inositol (MI) is a water-soluble nutrient involved in growth and cellular signaling. This study evaluated growth, hepatopancreatic biochemical indices, and stress-related gene transcription in crayfish fed 0 or 1000 mg/kg supplemental MI and chronically exposed to 0-10 µg/L BPS for 6 weeks. Compared with the control group, the BPS group had significantly lower weight gain rate (WGR), specific growth rate (SGR), molting frequency, antioxidant enzyme activities (SOD, CAT, and POD), and immune enzyme activities (ACP, AKP, PO, and LZM), together with higher MDA content (P < 0.05). Compared with the BPS group, the MI + BPS group had significantly higher WGR, SGR, molting frequency, SOD, POD, ACP, and PO activities (P < 0.05). The MI + BPS group also showed higher nrf2 and hmc mRNA levels and lower keap1, bip, ire1, casp3, cytc, jnk, il-6, tnf-α, and nf-κb mRNA levels than the BPS group (P < 0.05). WGR, SGR, molting frequency, and hmc mRNA abundance in the MI + BPS group did not differ significantly from the control group (P > 0.05), the remaining endpoints showed indicator-specific responses. These findings indicate that dietary MI supplementation was associated with improved growth and selected antioxidant and immune indices, as well as altered transcription of genes related to oxidative, endoplasmic-reticulum-stress, inflammatory, and apoptosis-associated responses under BPS exposure. Pathway activation, tissue injury, and apoptosis were not directly assessed. - Source: PubMed
Publication date: 2026/08/25
Pu ChangchangLiu YuanyiWang JunhuiWang BingkeWang AiminZhang Chunnuan