Chimpanzee IL-6 ELISPOT kit, enzymatic staining
- Known as:
- Chimpanzee Interleukin-6 ELISPOT reagent, enzymatic staining
- Catalog number:
- ct174-pr5
- Product Quantity:
- EUR
- Category:
- -
- Supplier:
- U-CyTech biosciences
- Gene target:
- Chimpanzee IL-6 ELISPOT kit enzymatic staining
Related genes to: Chimpanzee IL-6 ELISPOT kit, enzymatic staining
- Gene:
- CEBPB NIH gene
- Name:
- CCAAT enhancer binding protein beta
- Previous symbol:
- TCF5
- Synonyms:
- LAP, CRP2, NFIL6, IL6DBP, C/EBP-beta
- Chromosome:
- 20q13.13
- Locus Type:
- gene with protein product
- Date approved:
- 1991-02-27
- Date modifiied:
- 2018-02-23
- Gene:
- CEBPD NIH gene
- Name:
- CCAAT enhancer binding protein delta
- Previous symbol:
- -
- Synonyms:
- CRP3, CELF, C/EBP-delta, NF-IL6-beta
- Chromosome:
- 8q11.21
- Locus Type:
- gene with protein product
- Date approved:
- 1992-06-24
- Date modifiied:
- 2018-02-23
- Gene:
- ENTPD6 NIH gene
- Name:
- ectonucleoside triphosphate diphosphohydrolase 6
- Previous symbol:
- CD39L2, IL6ST2
- Synonyms:
- NTPDase-6, dJ738P15.3
- Chromosome:
- 20p11.21
- Locus Type:
- gene with protein product
- Date approved:
- 1998-03-20
- Date modifiied:
- 2019-02-28
- Gene:
- IL6 NIH gene
- Name:
- interleukin 6
- Previous symbol:
- IFNB2
- Synonyms:
- IL-6, BSF2, HGF, HSF
- Chromosome:
- 7p15.3
- Locus Type:
- gene with protein product
- Date approved:
- 1986-01-01
- Date modifiied:
- 2017-07-12
- Gene:
- IL6RP1 NIH gene
- Name:
- interleukin 6 receptor pseudogene 1
- Previous symbol:
- IL6RL1
- Synonyms:
- -
- Chromosome:
- 9q22.2
- Locus Type:
- pseudogene
- Date approved:
- 1991-08-18
- Date modifiied:
- 2014-11-19
Related products to: Chimpanzee IL-6 ELISPOT kit, enzymatic staining
Human ELC ELISA KIT 96 TEST
OxiSelect In Vitro ROS/RNS Assay Kit (Green Fluorescence), Trial Size
OxiSelect Methylglyoxal (MG) Competitive ELISA Kit
OxiSelect Methylglyoxal (MG) Competitive ELISA Kit
OxiSelect TBARS Assay Kit (MDA Quantitation), Trial Size
OxiSelect Total Antioxidant Capacity (TAC) Assay Kit, Trial Size
OxiSelect™ In Vitro ROS RNS Assay Kit (Green Fluorescence), Trial Size(1-Kit )11,12-EET DHET Immunoassay Kit(1-Kit )11,12-EET_DHET Immunoassay Kit(1-Kit) 11,12-DHET Immunoassay Kit(1-Kit) 14,15-DHET Human Urine ELISA Kit(1-Kit) 14,15-DHET Hypertension ELISA Kit(1-Kit) 14,15-DHET sEH activity ELISA Kit(1-Kit) 14,15-EET DHET Hypertension ELISA Kit(1-Kit) 14,15-EET_DHET Hypertension ELISA Kit Related articles to: Chimpanzee IL-6 ELISPOT kit, enzymatic staining
- Approximately 30% of epilepsy patients develop pharmacoresistant epilepsy (PRE), characterized by persistent seizures refractory to antiseizure medications (ASMs). While neuroinflammation is implicated in both epileptogenesis and treatment failure, its precise temporal contribution remains unclear. This study aimed to delineate the distinct roles of acute versus chronic neuroinflammation in seizure susceptibility and the maintenance of drug resistance. - Source: PubMed
Wang XianmeiDeng JieShi DaiLiu ZhiyanWang LikunWu Guofeng - Orchitis is a testicular inflammatory disorder that may impair spermatogenesis and compromise male fertility. Although fluoroquinolones are widely used in infectious orchitis and possess ancillary anti-inflammatory properties, concerns remain regarding their potential reproductive toxicity. Therapeutic ultrasound has emerged as a noninvasive modality with anti-inflammatory and tissue-modulating effects, but its role in testicular inflammation remains poorly characterized. - Source: PubMed
Publication date: 2026/08/01
Elfar NourhanAbdelkader Ahmed MamdouhMostafa Doaa SayedMostafa NihalAbdelaziz Sarah SamiGressy Nadia Saad Sayed Ahmed ElAtwa Ahmed MRadwan Abdullah F - Acute mesenteric ischemia (AMI) is a life-threatening vascular emergency associated with high morbidity and mortality, largely because diagnosis is frequently delayed by non-specific clinical manifestations. Although computed tomography angiography (CTA) remains the diagnostic gold standard, considerable research has focused on identifying laboratory biomarkers that may facilitate earlier recognition and improve clinical decision-making. This narrative review summarizes the current evidence regarding routinely available and emerging laboratory biomarkers for the diagnosis of AMI. A comprehensive literature search was performed using PubMed/Medical Literature Analysis and Retrieval System Online (MEDLINE), Scopus, Web of Science, and the Cochrane Library, supplemented by manual review of reference lists. Routine laboratory markers, including leukocyte count, C-reactive protein (CRP), serum lactate, metabolic acidosis, D-dimer, amylase, phosphate, and procalcitonin (PCT), were evaluated alongside investigational biomarkers, such as intestinal fatty acid-binding protein (I-FABP), D-lactate, citrulline, ischemia-modified albumin (IMA), alpha-glutathione S-transferase (α-GST), interleukin-6 (IL-6), and cell-count-derived inflammatory indices. Current evidence demonstrates that no single laboratory biomarker possesses sufficient sensitivity and specificity to independently confirm or exclude AMI. Serum lactate and metabolic acidosis primarily reflect advanced intestinal injury and systemic hypoperfusion, rather than early ischemia, whereas D-dimer offers relatively high sensitivity but poor specificity. Enterocyte-specific biomarkers, particularly I-FABP, remain biologically attractive because they may detect early mucosal injury; however, inconsistent diagnostic performance, limited assay availability, and methodological heterogeneity have prevented routine clinical implementation. Emerging multimarker strategies integrating indicators of enterocyte injury, coagulation activation, inflammation, and tissue hypoperfusion may improve diagnostic accuracy but require prospective validation. At present, laboratory investigations should be regarded as complementary tools that increase clinical suspicion, assist with risk stratification, and reinforce the need for urgent CTA. They should never delay definitive imaging or revascularization when AMI is suspected. Further high-quality prospective studies are required to establish standardized biomarker panels capable of facilitating earlier diagnosis and improving patient outcomes. - Source: PubMed
Publication date: 2026/07/17
Velikoudi Maria DKourgiali Charis GGotti Kalliopi-Kleio EChatzelas Dimitrios A - This study aimed to evaluate the efficacy and safety of hepatic arterial infusion chemotherapy of oxaliplatin, fluorouracil, and leucovorin (FOLFOX-HAIC) combined with tislelizumab as a neoadjuvant regimen in patients with resectable hepatocellular carcinoma (HCC) beyond the Milan criteria, and to explore predictive biomarkers of treatment response. - Source: PubMed
Publication date: 2026/08/12
Dai QuanweiDeng HuitingWang JunShi JingxiangDing JianminGao YanyingSun WeiShen YunzhiWang XimoLou Cheng - Evolutionary-developmental theories of stress responsivity contend that the in utero environment shapes offspring biobehavioral regulation in a manner that promotes postnatal functioning. To elucidate how the prenatal environment influences stress regulation, this study tested associations of maternal psychological distress and inflammation during pregnancy, based on both levels and changes over time, with infant behavioral stress regulation (n = 118). In early, mid, and late pregnancy, mothers reported on depressive and anxiety symptoms and completed blood draws that were assayed for pro-inflammatory cytokines (IL-6, TNF-⍺). When infants were 1 month old, research staff administered a heel stick blood draw and coded infant behavioral responses. Greater increases in maternal depressive symptoms and IL-6 from early to late pregnancy were associated with greater behavioral reactivity to and recovery from the heel stick. Conversely, higher overall levels of IL-6 predicted lower infant behavioral arousal. Maternal anxiety symptoms during pregnancy did not directly predict infant behavioral regulation, but there was evidence that overall levels of prenatal anxiety predicted infant behavior indirectly via IL-6. Anxiety symptom levels were associated with TNF-⍺. Findings add to evidence on prenatal origins of offspring behavioral regulation and suggest that changes in psychobiological processes over the course of pregnancy may distinctly predict infant outcomes. - Source: PubMed
Rinne Gabrielle RRoss Kharah MDunkel Schetter Christine