SEMA3A (P737) pAb host: Rabbit
- Known as:
- SEMA3A (P737) pAb production species: Rabbit
- Catalog number:
- bs3691
- Product Quantity:
- USD
- Category:
- -
- Supplier:
- Bioworld
- Gene target:
- SEMA3A (P737) pAb host: Rabbit
Related genes to: SEMA3A (P737) pAb host: Rabbit
- Gene:
- PPP1R18 NIH gene
- Name:
- protein phosphatase 1 regulatory subunit 18
- Previous symbol:
- KIAA1949
- Synonyms:
- phostensin
- Chromosome:
- 6p21.33
- Locus Type:
- gene with protein product
- Date approved:
- 2004-03-02
- Date modifiied:
- 2016-10-05
- Gene:
- SEMA3A NIH gene
- Name:
- semaphorin 3A
- Previous symbol:
- SEMAD
- Synonyms:
- SEMA1, SemD, coll-1, Hsema-I
- Chromosome:
- 7q21.11
- Locus Type:
- gene with protein product
- Date approved:
- 1999-06-25
- Date modifiied:
- 2016-10-05
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guanine nucleotide binding protein alpha inhibiting activity polypeptide 1 (GNAI1) polyclonal antibody kinase suppressor of ras (KSR) polyclonal antibody'F 4_80 Antigen (mouse) Host Rat'F 4_80 Antigen (mouse) Host Rat(Alpha)_ 1 _ antitrypsin (A1AT) POLYCLONAL Rabbit anti_human(Alpha)_ Feto Protein (AFP) POLYCLONAL Rabbit anti_human(Alpha)_ Feto Protein (AFP) POLYCLONAL Rabbit anti_human(Alpha)_1_ antitrypsin (A1AT) POLYCLONAL Rabbit anti_human(Arg6,b_cyclohexyl_Ala8,D_Tic16,Arg17,Cys18)_Atrial Natriuretic Factor (6_18) amide (mouse, rabbit, rat) Salt _ Binding (Disulfide_bond) Synonym A71915 SumFormula C69H116N26O15S2(Arg6,b_cyclohexyl_Ala8,D_Tic16,Arg17,Cys18)_Atrial Natriuretic Factor (6_18) amide (mouse, rabbit, rat) Salt _ Binding (Disulfide_bond) Synonym A71915 SumFormula C69H116N26O15S2(Arg6,β-cyclohexyl-Ala8,D-Tic16,Arg17,Cys18)-Atrial Natriuretic Factor (6-18) amide (mouse, rabbit, rat)
A71915 98% C69H116N26O15S2 CAS:(Arg8)-Vasopressin - Diluted Antiserum for RIA, Host Guinea Pig(Arg8)-Vasopressin - Diluted Antiserum for RIA, Host Rabbit(Arg8)-Vasopressin - Diluted Antiserum for RIA, Host: Guinea Pig(Arg8)-Vasopressin - Diluted Antiserum for RIA, Host: Guinea Pig Related articles to: SEMA3A (P737) pAb host: Rabbit
- Bone metabolism is regulated by immune cells that maintain skeletal homeostasis through cytokine secretion and cell interactions. Disruption of this regulation-by inflammation, autoimmunity, or aging-drives pathological bone diseases including osteoporosis and rheumatoid arthritis. Single-cell technologies and exosome biology have revealed previously unrecognized immune subsets and regulatory layers within the bone microenvironment. - Source: PubMed
Publication date: 2026/08/03
Jin ZhuoruLiu LishengWang HeranWei YufeiZhou ZimoLuo XiaohuiLiu Da - Macrophage-associated responses at sites of nerve injury are involved in the initiation and persistence of neuropathic pain (NP). Immune semaphorins (SEMAs), including SEMA3A, SEMA3E, SEMA4A, SEMA4D, and SEMA7A, regulate macrophage migration and activation, but their roles in NP remain unclear. This study aimed to identify immune SEMAs associated with NP by analyzing their serum levels and expression in sensory nerve tissues of patients with NP, and to evaluate the potential effects of SEMA-targeted intervention in a mouse model. - Source: PubMed
Publication date: 2026/08/02
Yoshidomi SatoFujii TakayukiHonda HiroyukiKashu Kaoru YoshidaMiyachi YukinoInoue YukaOgata HidenoriYamasaki RyoIwaki ToruIsobe Noriko - The pathogenesis of multiple sclerosis (MS) remains incompletely elucidated. Semaphorins play an active role in the immune and nervous systems by regulating receptor-mediated adhesion mechanisms. Immunosemaphorins have recently emerged as diagnostic biomarkers or therapeutic targets in MS. - Source: PubMed
Publication date: 2026/07/14
Sarıdaş FurkanAydın BirnurÖzpar RifatKoç Emine RabiaHakyemez BahattinAlkan TülinTuran Ömer Faruk - To determine whether serum semaphorin 3 A (Sema3A) and interleukin 6 (IL-6) play a role in the common etiopathogenesis of nAMD and nAMD-associated systemic diseases by evaluating these mediators levels. - Source: PubMed
Publication date: 2026/07/14
Okutucu MuratArpa Medeni - The nervous system plays a crucial role in regulating bone remodeling, with both sensory and autonomic nerves distributed within bone tissue. In the mandible, the inferior alveolar nerve (IAN) and sympathetic nerves provide the primary innervation. Numerous studies have demonstrated their essential functions in mandibular development and bone metabolism. Focusing on mandibular innervation, we synthesize current evidence to demonstrate the modulatory effects of sensory and sympathetic nerves on bone growth and regeneration, and further elucidate the underlying mechanisms. Notably, we discuss multiple bioactive factors (CGRP, SP, VIP, NE, NPY, Sema3A, NGF) for their roles in mediating neural regulation of bone. Additionally, neuro-bone crosstalk in the mandible is examined in an integrated way. This review offers a deeper understanding of mandibular homeostasis modulated by nerves and thus seeks to facilitate future clinical translation. - Source: PubMed
Publication date: 2026/07/12
Zhao ZhiyiFu HanqiTian HuifeiChen QianWang PengChen PengLiao ChiWu ZupingChen Xiaoyan