CARD6 (A2) pAb host: Rabbit

Price:

411.95 €

Known as:: CARD6 (A2) pAb production species: Rabbit
Catalog number:: bs1038
Product Quantity:: USD
Category:: -
Supplier:: Bioworld
Gene target:: CARD6 (A2) pAb host: Rabbit

Related genes to: CARD6 (A2) pAb host: Rabbit

Gene:: A2MP1 ^{NIH gene}
Name:: alpha-2-macroglobulin pseudogene 1
Previous symbol:: A2MP
Synonyms:: -
Chromosome:: 12p13.31
Locus Type:: pseudogene
Date approved:: 1991-09-12
Date modifiied:: 2016-08-22

Gene:: AAR2 ^{NIH gene}
Name:: AAR2 splicing factor
Previous symbol:: C20orf4
Synonyms:: bA234K24.2
Chromosome:: 20q11.23
Locus Type:: gene with protein product
Date approved:: 2001-06-21
Date modifiied:: 2019-02-18

Gene:: ABHD17AP1 ^{NIH gene}
Name:: abhydrolase domain containing 17A pseudogene 1
Previous symbol:: FAM108A2, ABHD17AP2, FAM108A3, FAM108A11P, FAM108A3P
Synonyms:: C1orf47
Chromosome:: 1q21.2
Locus Type:: pseudogene
Date approved:: 2006-01-17
Date modifiied:: 2015-01-29

Gene:: ACBD5 ^{NIH gene}
Name:: acyl-CoA binding domain containing 5
Previous symbol:: -
Synonyms:: DKFZp434A2417, KIAA1996
Chromosome:: 10p12.1
Locus Type:: gene with protein product
Date approved:: 2003-11-11
Date modifiied:: 2014-11-19

Gene:: ACRV1 ^{NIH gene}
Name:: acrosomal vesicle protein 1
Previous symbol:: -
Synonyms:: SPACA2, SP-10, D11S4365
Chromosome:: 11q24.2
Locus Type:: gene with protein product
Date approved:: 1991-06-07
Date modifiied:: 2014-11-19

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guanine nucleotide binding protein alpha inhibiting activity polypeptide 1 (GNAI1) polyclonal antibodykinase suppressor of ras (KSR) polyclonal antibody'F 4_80 Antigen (mouse) Host Rat'F 4_80 Antigen (mouse) Host Rat(Alpha)_ 1 _ antitrypsin (A1AT) POLYCLONAL Rabbit anti_human(Alpha)_ Feto Protein (AFP) POLYCLONAL Rabbit anti_human(Alpha)_ Feto Protein (AFP) POLYCLONAL Rabbit anti_human(Alpha)_1_ antitrypsin (A1AT) POLYCLONAL Rabbit anti_human(Arg6,b_cyclohexyl_Ala8,D_Tic16,Arg17,Cys18)_Atrial Natriuretic Factor (6_18) amide (mouse, rabbit, rat) Salt _ Binding (Disulfide_bond) Synonym A71915 SumFormula C69H116N26O15S2(Arg6,b_cyclohexyl_Ala8,D_Tic16,Arg17,Cys18)_Atrial Natriuretic Factor (6_18) amide (mouse, rabbit, rat) Salt _ Binding (Disulfide_bond) Synonym A71915 SumFormula C69H116N26O15S2(Arg6,β-cyclohexyl-Ala8,D-Tic16,Arg17,Cys18)-Atrial Natriuretic Factor (6-18) amide (mouse, rabbit, rat) A71915 98% C69H116N26O15S2 CAS:(Arg8)-Vasopressin - Diluted Antiserum for RIA, Host Guinea Pig(Arg8)-Vasopressin - Diluted Antiserum for RIA, Host Rabbit(Arg8)-Vasopressin - Diluted Antiserum for RIA, Host: Guinea Pig(Arg8)-Vasopressin - Diluted Antiserum for RIA, Host: Guinea Pig

Related articles to: CARD6 (A2) pAb host: Rabbit

Caspase Recruitment Domain 6 (CARD6) is the therapeutic target of dihydroartemisinin for lupus nephritis based on Proteolysis Targeting Chimera (PROTAC) technology.
Lupus nephritis (LN) is a common complication of systemic lupus erythematosus (SLE) that symbolizes a primary contributor of morbidity and mortality. Dihydroartemisinin (DHA; artenimol) shows good therapeutic effects on LN. Its pharmacological target remains unclear. Proteolysis targeting chimeras (PROTAC) technology was used to identify the targets of DHA in LN treatment. - Source: PubMed
Publication date: 2026/05/19
Li HonglianNi ZhihaoRao YuzhenZhang JunzheGuo HuiyiYuan HaoxingYang HaoChen JiayunYang ChanWang JigangRao YuLiu Shuwen
Microarray Analysis of Differentially Expressed Genes in Peripheral Blood of Postpartum Women with Gestational Diabetes Mellitus and Type 2 Diabetes.
The etiology of women with gestational diabetes mellitus (GDM) and a greater risk of developing type 2 diabetes (T2D) after delivery remains unknown. This study aimed to investigate the global gene expression in four postpartum women with previous GDM (pGDM), three with T2D, and three with a history of normoglycemic pregnancy (controls). Total RNA was extracted from whole blood between March and May 2020. Global mRNA expression was determined using an Affymetrix Human Gene 2.0 ST Array. The expression of the selected focused genes was validated by RT-PCR. The microarray revealed 140 transcripts ( < 0.05, fold change cut-off ≥ 2) in patients with pGDM compared to controls. We identified 583 gene-altered transcripts between patients with T2D and controls. Interestingly, 60 transcripts had genes shared by pGDM or T2D versus the controls. The selected upregulated genes involved in inflammatory response, glycosylation, and death-like domains, according to the functional network analysis of pGDM (, ) and T2D ( and ), showed similar trends to those obtained via microarray. Thus, these differentially expressed genes and their corresponding network and pathway analyses in women with pGDM and T2D offer valuable insights into the possible biological mechanisms of the progression of GDM to T2D. - Source: PubMed
Publication date: 2025/08/11
Sultan Samar
Corrigendum to "Circ-CARD6 inhibits oxidative stress-induced apoptosis and autophagy in ARPE-19 cells via the miR-29b-3p/PRDX6/PI3K/Akt axis" [(2024) 238 109690 doi: 10.1016/j.exer.2023.109690. Epub 2023 6].
- Source: PubMed
Publication date: 2025/08/18
Fan XinyuYang YanniWu GuojiuKong YanboZhang YuanpingZha Xu
Investigation and Confirmation of PYCARD as a Potential Biomarker for the Management of Psoriasis Disease.
Psoriasis is not yet completely curable, and its etiology and pathogenesis are unclear. Necroptosis, also known as programmed necrosis, is a regulated mode of necrotic cell death. The interaction between inflammatory diseases and necrotic apoptosis has recently attracted significant attention. We explored the molecular mechanisms of necrotic apoptosis-related genes in psoriasis using bioinformatics methods to identify potential biomarkers for psoriasis. - Source: PubMed
Publication date: 2024/09/16
Xu BingyangYu BiaoXu ZiningYe ShuhongQing YuxinSun HongZhao BinWu NaWu Jiawen
A Bruton tyrosine kinase inhibitor-resistance gene signature predicts prognosis and identifies TRIP13 as a potential therapeutic target in diffuse large B-cell lymphoma.
Bruton tyrosine kinase inhibitor (BTKi) combined with rituximab-based chemotherapy benefits diffuse large B-cell lymphoma (DLBCL) patients. However, drug resistance is the major cause of relapse and death of DLBCL. In this study, we conducted a comprehensive analysis BTKi-resistance related genes (BRRGs) and established a 10-gene (CARD16, TRIP13, PSRC1, CASP1, PLBD1, CARD6, CAPG, CACNA1A, CDH15, and NDUFA4) signature for early identifying high-risk DLBCL patients. The resistance scores based on the BRRGs signature were associated with prognosis. Furthermore, we developed a nomogram incorporating the BRRGs signature, which demonstrated excellent performance in predicting the prognosis of DLBCL patients. Notably, tumor immune microenvironment, biological pathways, and chemotherapy sensitivity were different between high- and low-resistance score groups. Additionally, we identified TRIP13 as a key gene in our model. TRIP13 was found to be overexpressed in DLBCL and BTKi-resistant DLBCL cell lines, knocking down TRIP13 suppresses cell proliferation, promotes cell apoptosis, and enhances the apoptosis effect of BTKi on DLBCL cells by regulating the Wnt/β-catenin pathway. In conclusion, our study presents a novel BRRGs signature that could serve as a promising prognostic marker in DLBCL, and TRIP13 might be a potential therapeutic target for resistant DLBCL. - Source: PubMed
Publication date: 2024/09/11
Ding YangyangHuang KekeSun ChengLiu ZelinZhu JinliJiao XunyiLiao YaFeng XiangjiangGuo JingjingZhu ChunhuaZhai ZhiminXiong Shudao

CARD6 (A2) pAb host: Rabbit

Related genes to: CARD6 (A2) pAb host: Rabbit

Related products to: CARD6 (A2) pAb host: Rabbit

Related articles to: CARD6 (A2) pAb host: Rabbit

Caspase Recruitment Domain 6 (CARD6) is the therapeutic target of dihydroartemisinin for lupus nephritis based on Proteolysis Targeting Chimera (PROTAC) technology.

Microarray Analysis of Differentially Expressed Genes in Peripheral Blood of Postpartum Women with Gestational Diabetes Mellitus and Type 2 Diabetes.

Corrigendum to "Circ-CARD6 inhibits oxidative stress-induced apoptosis and autophagy in ARPE-19 cells via the miR-29b-3p/PRDX6/PI3K/Akt axis" [(2024) 238 109690 doi: 10.1016/j.exer.2023.109690. Epub 2023 6].

Investigation and Confirmation of PYCARD as a Potential Biomarker for the Management of Psoriasis Disease.

A Bruton tyrosine kinase inhibitor-resistance gene signature predicts prognosis and identifies TRIP13 as a potential therapeutic target in diffuse large B-cell lymphoma.