Ask about this productRelated genes to: NTF4 antibody
- Gene:
- NTF4 NIH gene
- Name:
- neurotrophin 4
- Previous symbol:
- NTF5
- Synonyms:
- NT-4/5, GLC1O
- Chromosome:
- 19q13.3
- Locus Type:
- gene with protein product
- Date approved:
- 1992-10-15
- Date modifiied:
- 2014-11-19
Related products to: NTF4 antibody
Related articles to: NTF4 antibody
- Stroke is a major complication of atrial fibrillation (AF), and risk prediction using the congestive heart failure, hypertension, age, diabetes, stroke, vascular disease, and sex category score (CHA₂DS₂-VASc) remains limited by residual heterogeneity. We aimed to identify plasma proteins associated with post-AF stroke and evaluate whether a protein score provides incremental predictive information beyond CHA₂DS₂-VASc. - Source: PubMed
Publication date: 2026/07/23
Huang ShengkangFeng XinWu HuilinLiang RongxuanGao YuanWang ZhaojunTang BotaoWu JiatongFang JunkaiYang ZiangTiemuerniyazi XierailiLin JianchengDuan YijingxuanXu WenruMao HuaZhao WeiHu ZhanDuan ChuanzhiFeng Wei - This study aims to investigate the role of neurotrophin-4 (NTF4) in gestational diabetes mellitus (GDM) and to elucidate the underlying mechanism by which NTF4 regulates high glucose-induced apoptosis and inflammation in HTR-8/SVneo cells via the PI3K/AKT signaling pathway. - Source: PubMed
Zhang LiYang Juan - Following the publication of the above article, the authors subsequently realized that, during the process of collating the raw data, Fig. 1 [the immunohistochemical (IHC) results for stage IV colorectal cancer (CRC)], Fig. 2A (the control β‑actin blots) and Fig. 5C and D (both the images selected for the clone formation assays, and the histograms showing the quantification of the data) were inadvertently assembled incorrectly. These errors arose as a consequence of the affected files having been named similarly to those of the correct panels. The revised versions of Figs. 1, 2 and 5, now featuring the correct IHC data for stage IV CRC in Fig. 1, the correct control western blots in Fig. 2 and the correct colony formation assay data (and quantification thereof) in Fig. 5, are shown on the next three pages. Note that the correction of these figures does not affect the key findings of the study (either the existing published results or the conclusions reached from the results). The authors thank the Editor of for granting them the opportunity to publish this corrigendum. All the authors agree with the publication of this corrigendum; furthermore, they apologize to the readership of the journal for any inconvenience caused. [International Journal of Oncology 56: 1442‑1454, 2020; DOI: 10.3892/ijo.2020.5027]. - Source: PubMed
Publication date: 2025/02/14
Yang ZhouChen YushengWei XiyiWu DejunMin ZhijunQuan Yingjun - Long non-coding RNAs (lncRNAs) are importantly involved in the initiation and progression of non-small cell lung cancer (NSCLC). However, the classification and mechanisms of lncRNAs remain largely elusive. - Source: PubMed
Publication date: 2024/10/28
Liu YinqiangYang HongjvLv GuoliDuan JinZhao WeiShi YunfeiLei Youming - Rosemary leaf extract, a well-known medicinal plant, can induce neurotrophin gene expression and proliferation in stem cells. Human adipose-derived stem cells (hASCs) with high proliferation and differentiation capacity are easily accessible and can be extracted with the least damage. This study evaluated the effect of rosemary extract (RE) on neurotrophin gene expression at 48 h postinduction in hASCs. hASCs were isolated from healthy female donors, aged 28-35 years, who had undergone abdominal liposuction. Passage-4 stem cells were cultured and treated with different doses of RE (from 30 to 70 µg/ml) containing 40% carnosic acid for 48 h. Reverse transcription-polymerase chain reaction was used to check the expression of neurotrophin genes. The expression of , , and nerve growth factor genes in cells treated with 40-60 µg/ml and the expression of in cells treated with 50-70 µg/ml of RE for 48 h showed a significant increase compared to cells cultured in serum-containing medium. However, different doses of RE showed no effect on brain-derived neurotrophic factor gene expression in the treated cells. RE (50, 60 µg/ml) leads to an increase of neurotrophin gene expression in hASCs as compared to routine cell culture. Hence, this protocol can be used to prepare ideal cell sources for cell therapy. - Source: PubMed
Publication date: 2024/07/22
Altememy DhiyaKashani Maryam Haji GhasemFateme AmirahmadiKhosravian Pegah