Ask about this productRelated genes to: CAMK1D Blocking Peptide
- Gene:
- CAMK1D NIH gene
- Name:
- calcium/calmodulin dependent protein kinase ID
- Previous symbol:
- -
- Synonyms:
- CKLiK
- Chromosome:
- 10p13
- Locus Type:
- gene with protein product
- Date approved:
- 2003-11-05
- Date modifiied:
- 2015-12-04
Related products to: CAMK1D Blocking Peptide
Related articles to: CAMK1D Blocking Peptide
- Chronic hepatitis B virus (HBV) rarely leads to spontaneous hepatitis B surface antigen (HBsAg) seroclearance. However, host genetics contributing to this outcome remain poorly understood. We investigate genetic variants associated with HBsAg seroclearance and related viral markers. - Source: PubMed
Publication date: 2026/05/11
Tao JunHuang Chih-JenHuang Yu-HanPan Mei-HungLee Mei-HsuanYu Kelly JWang KevinZhu BinHildesheim AllanChen Chien-JenYang Hwai-ILiu Zhiwei - Pigs are one of the most important livestock species for providing meat products in the world. Deciphering the genetic architecture of feed efficiency-related traits is beneficial to improve the genetic progress of these traits and save the total cost of pork production. However, the genetic architecture of feed efficiency-related traits remains unclear. - Source: PubMed
Publication date: 2026/02/27
Lin ChangguangChen QiuyongLiu YaxuanCai WeiHuang TaoZhou YiLin JinyuZhou LunjiangChen Xinzhu - The gut microbiome is closely associated with malignant tumors; however the specific mechanisms by which it contributes to the development of lung adenocarcinoma remain unclear. In this study, we performed a two-sample bidirectional Mendelian randomization (MR) analysis to assess the causal relationship between the gut microbiome and lung adenocarcinoma. By identifying single nucleotide polymorphism markers linked to gut microbiome species, we aimed to discover potential biomarkers for lung adenocarcinoma. These findings may offer new insights into the role of the gut microbiome in the prevention and treatment of lung adenocarcinoma. - Source: PubMed
Publication date: 2026/03/18
Yan NuoZhang YangWang SilinHu ShengRuan LianchengWang YunzheFeng WeiqiangXiong WenxunZhang WenxiongWei YipingYao Chuan - Diabetic kidney disease (DKD) is a leading cause of end-stage renal disease (ESRD), accelerates age-related decline in estimated glomerular filtration rate (eGFR), leading to a markedly increased prevalence of DKD among elderly diabetic patients. Recent studies suggest that immune dysregulation plays a pivotal role in DKD progression; however, the cellular and molecular mechanisms linking aging, immune infiltration, and DKD remain unclear. - Source: PubMed
Publication date: 2026/02/13
Zhu PeiqiTang RuijieZhou YapingHe YiningZhao JingLiu ZhengxinZhao YingHe Weiming - Metabolic syndromes (MeS), marked by central obesity, high blood pressure, abnormal cholesterol and blood sugar, are key cardiovascular disease (especially coronary artery disease, CAD) risk factors. Genetic studies show MeS-CAD genetic overlap, indicating shared biological pathways. We used Summary-data-based Mendelian Randomization (SMR), Bayesian colocalization (with large GWAS summary stats for MeS/CAD and cis-eQTL data from 3 tissues) and Transcriptome-Wide Association Study (TWAS). We also investigated the effects of gene knockout on mouse phenotypes. SMR found 886/737/192 shared genes in blood/brain cortex/liver; colocalization identified 11/13/5 shared causal genes in these tissues and 46 shared loci (e.g., CAMK1D, OR=1.11; AGPAT1, OR=1.13; FDR<0.05). Moreover, knocking out these genes in mice affected metabolism, adipose tissue, cardiovascular function, glucose homeostasis, and the fat/muscle balance. This study identified common regulatory genes between MeS and CAD, suggesting that targeted therapies or interventions could potentially address both conditions simultaneously, offering prospects for more integrated treatment strategies. - Source: PubMed
Publication date: 2025/12/30
Yi PengchengYin QuantingZhang HuanhuanYang ChunhuaZhu YanpingXia ZhenhongXu FuyiMi Jia