CD11b antibody (Azide Free)
- Known as:
- CD11b (anti-) (Azide Free)
- Catalog number:
- 10r-cd11bgmsle
- Product Quantity:
- USD
- Category:
- -
- Supplier:
- Fitzgerald industries international
- Gene target:
- CD11b antibody (Azide Free)
Related genes to: CD11b antibody (Azide Free)
- Gene:
- ITGAM NIH gene
- Name:
- integrin subunit alpha M
- Previous symbol:
- CR3A, CD11B
- Synonyms:
- MAC-1, CD11b
- Chromosome:
- 16p11.2
- Locus Type:
- gene with protein product
- Date approved:
- 1988-08-05
- Date modifiied:
- 2019-04-23
Related products to: CD11b antibody (Azide Free)
Related articles to: CD11b antibody (Azide Free)
- Chronic inflammation is a well-established driver of colorectal cancer (CRC), with the resulting inflammatory microenvironment facilitating tumor initiation and progression. The integrin CD11b/CD18, a leukocyte-specific heterodimeric adhesion receptor, mediates critical immunoregulatory functions during inflammatory responses. However, the roles and mechanisms of CD11b/CD18 in colitis-associated colorectal cancer (CAC) remain unclear. - Source: PubMed
Publication date: 2026/06/18
Lin YingLiu Yuan-KunMi PengZhao Xian-LingLin Jun-HuiCheng Xiao-ShenLiang Lai-YingHuang Yong-DongHuo Ya-NiXie Gui-JingYe Zhen-YuGuleng Bayasi - Leukemia progression is strongly influenced by the immune microenvironment, yet the regulatory gene networks that coordinate immune activity in leukemia remain incompletely defined. Identifying immune-associated hub genes with diagnostic, prognostic, and functional relevance may improve biological understanding and guide biomarker development. - Source: PubMed
Publication date: 2026/05/20
Chen HaiyanLiao JunyaoLiu JunxiaLi LingLiao JingWang Wenru - Transcriptome-wide association studies (TWAS) identify genetically regulated expression (GReX) components and can pinpoint causal genes in genome-wide association studies but are often limited by a single-cell context. - Source: PubMed
Publication date: 2026/05/26
Zhang HaihanPatrick Matthew TSarkar Mrinal KBogle RachaelLi QinmenggeUppala RanjithaPerez White Bethany EPetukhova LynnDand NickStuart Philip EChristiano Angela MSimpson Michael ABarker Jonathan NWeidinger StephanModlin Robert LBetz Regina CKhanna DineshVarga JohnKahlenberg J MichelleHe KevinElder James TZhou XiangGudjonsson Johann ETsoi Lam C - Influenza virus A (H1N1) can lead to acute respiratory infection, while Guillain-Barré Syndrome (GBS) is an autoimmune peripheral neuropathy, which can work as a post-infectious disease. There are clinical observations showing the existence of a possible relationship between the presence of H1N1 infection and GBS disease, and therefore there could be common immunopathological pathways associated with H1N1 infection and GBS. The possible similarity between H1N1 infection and GBS was further investigated using integrated bioinformatics and systems biology approaches. Differentially expressed genes (DEGs) were identified from GEO datasets related to H1N1 infection and GBS. Enrichment analysis was conducted to understand the functional roles of identified DEGs by performing GO and KEGG pathway analysis. For the interacting proteins of the common DEGs, protein-protein interaction (PPI) network analysis helped to find out TLR4, TNF, and ITGAM as key hub genes. These hub genes might have common molecular pathways in H1N1 infection and GBS. To predict possible drug targets for treatment, analysis of interactions between hub genes and miRNAs, TFs, and related diseases was performed. - Source: PubMed
Publication date: 2026/05/20
Deng YeTang ZhongwenChen QiongHu XujuanZhang YingjieChen MengWang QiongyaRen Fuli - Hyaluronic acid, as a natural extracellular matrix component, possesses excellent biocompatibility and moisturizing properties, and demonstrates unique advantages in wound repair. - Source: PubMed
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