Human Interleukin 26,IL-26 ELISA Kit
- Known as:
- Human Interleukin 26,Interleukin-26 Enzyme-linked immunosorbent assay test Kit
- Catalog number:
- 201-12-0054
- Product Quantity:
- USD
- Category:
- -
- Supplier:
- Sunredbio SunBT Sun red bio
- Gene target:
- Human Interleukin 26 IL-26 ELISA Kit
Related genes to: Human Interleukin 26,IL-26 ELISA Kit
- Gene:
- IL26 NIH gene
- Name:
- interleukin 26
- Previous symbol:
- -
- Synonyms:
- AK155, IL-26
- Chromosome:
- 12q15
- Locus Type:
- gene with protein product
- Date approved:
- 2001-11-23
- Date modifiied:
- 2008-08-04
Related products to: Human Interleukin 26,IL-26 ELISA Kit
Related articles to: Human Interleukin 26,IL-26 ELISA Kit
- The monoterpene 1,8-Cineol is a natural plant-based anti-inflammatory agent that is used to treat different respiratory diseases with regard to its mucolytic and anti-microbial properties. 1,8-Cineol has recently been shown to support the natural microbial colonization of the middle ear, the intestinal bacterial community, as well as the clinical inflammatory situation of patients with chronic otitis media (COM). The aim of this study was to understand better the influence of 1,8-Cineol treatment on the systemic balances of inflammatory mediators of the interleukin-10 (IL-10) family of cytokines. - Source: PubMed
Leichtle AnkeFleckner JonasPlötze-Martin KirstinPenxová ZuzanaBruchhage Karl-LudwigPries Ralph - The present study aimed to characterize the immune response, differential gene expression, and functional alterations observed following an in vivo challenge of Classical Swine Fever Virus (CSFV) in crossbred pigs. RNA-sequencing was performed on whole blood samples collected from three pigs before and at 7 days post-infection. A total of 3428 differentially expressed genes (DEGs) were identified, including 970 significantly upregulated and 261 downregulated genes (|log fold change| ≥ 1.5, adjusted p < 0.05). The most prominent DEG was LAMB4, a gene associated with cellular attachment receptors facilitating CSFV binding to porcine cells. Several cytokine-cytokine receptor interaction genes, such as CXCL12, CCL2, CCR1, and IL10RA, were upregulated, indicating a strong activation of innate immune responses. Simultaneously, multiple adaptive immune genes, including CD28, CD83, SLA-DQB1, and IL1A, IL12A, IL26 were downregulated, suggesting viral-mediated suppression of antigen presentation and T-cell signaling. Pathway analysis highlighted the involvement of platelet activation and coagulation cascades during viral evasion. Protein-protein interaction (PPI) analysis revealed a core antiviral module comprising MX1, MX2, ISG15, IFIH1, OASL, IFIT1, and UBE2L6, which are central to interferon signaling and viral restriction. Transcription factors such as ETV7, TOX3, and MSC were upregulated, pointing to immune modulation and possible T-cell exhaustion. Conversely, downregulation of HES1, PRDM6, and MYOG indicated impaired lymphocyte differentiation and tissue repair. Overall, the findings suggest a dual host response to CSFV, with strong innate activation alongside adaptive immune suppression, providing valuable insights for vaccine and therapeutic development. - Source: PubMed
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