EZRI_RABIT VIL2 ELISA tesk kit
- Known as:
- EZRI_RABIT VIL2 Enzyme-linked immunosorbent assay test tesk reagent
- Catalog number:
- gen16699
- Product Quantity:
- 1
- Category:
- Peptides
- Supplier:
- Other suppliers
- Gene target:
- EZRI_RABIT VIL2 ELISA tesk kit
Related genes to: EZRI_RABIT VIL2 ELISA tesk kit
- Gene:
- EZR NIH gene
- Name:
- ezrin
- Previous symbol:
- VIL2
- Synonyms:
- -
- Chromosome:
- 6q25.3
- Locus Type:
- gene with protein product
- Date approved:
- 1989-06-30
- Date modifiied:
- 2015-09-11
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- Combined immunodeficiencies (CIDs) are a severe class of inborn errors of immunity characterized by defective T cell development and function, often accompanied by impaired humoral and natural killer (NK) cell responses. Despite their shared clinical classification, the immunological heterogeneity within CIDs remains incompletely understood. This study aims to characterize the immune cell landscape in CID patients caused by disease-causing mutations in different genes to identify immunophenotypic patterns. - Source: PubMed
Publication date: 2026/06/12
Pérez-Caraballo Jareb JRoark Colleen MDobrose Megan MVan den Rym AnaStaines-Boone Aidé TamaraSalazar-Galvez YuridiaGomez-Hernandez NoemiCui JianBrown Lauren EZhen XinLugo Reyes Saul OBlancas-Galicia Lizbethde la Calle-Martín ÓscarMohammed ReemPérez de Diego RebecaMartínez-Barricarte Rubén - Enamel pits and enamel developmental defects have been reported in patients with tuberous sclerosis complex (TSC), but prevalence estimates and phenotypic descriptions vary widely. A standardized synthesis has been lacking. Recent updates in TSC clinical reviews and dermatologic-dental guidelines highlight the diagnostic relevance of subtle oral findings, underscoring the need for an updated synthesis. - Source: PubMed
Publication date: 2026/06/29
Nakagawa H - Ezrin, expressed by the gene, is a member of the ERM protein family that connects the plasma membrane to the actin cytoskeleton, participating in processes such as cell adhesion, migration, and signaling. However, its role in cardiac morphogenesis remains incompletely understood. In zebrafish (), two homologs, and , are present. CRISPR/Cas9 gene editing technology was used to generate knockout lines, and the simultaneous knockdown of and was induced via morpholino oligonucleotides (MOs). To investigate the molecular mechanisms, transcriptome sequencing and bioinformatic analysis were conducted on 48 h post-fertilization (hpf) -MO embryos, with subsequent validation using a real-time quantitative polymerase chain reaction (RT-qPCR) and whole-mount in situ hybridization (WISH) experiment. The results showed that exhibited a compensatory upregulation of without overt developmental defects, whereas -MO embryos presented with pericardial edema, reduced cardiac chamber size, and atrioventricular valve malformations at 48 hpf. RNA-seq revealed that myocardial contraction-related genes were significantly dysregulated and apoptotic signaling pathways were activated in -MO embryos. These findings demonstrate that and are functionally redundant in cardiac development and that the loss of function may lead to cardiac developmental defects and impaired myocardial contractility via the activation of apoptotic signaling pathways. - Source: PubMed
Publication date: 2026/06/07
Lv JinruiZeng TingLiao BeiyaLiu LingXiong LeiXie HaoZhu LinJiang XingziZhong ZhuchuyuXie Huaping - Esophageal squamous cell carcinoma (ESCC) is a prevalent gastrointestinal malignancy in China and is associated with a poor prognosis and limited therapeutic options. encodes Ezrin, a key scaffold protein that bridges the cell membrane and cytoskeleton. Its aberrant upregulation and carcinogenic role in driving malignant progression across numerous human malignancies has been established. However, the expression profile, specific biological functions, and underlying molecular mechanisms of in ESCC remain largely unclear, which has limited the exploration and development of novel targeted therapeutic strategies against ESCC. This study aims to explore the role of in the malignant progression of ESCC, identify its potential downstream molecules, and screen for its potential interacting proteins, so as to reveal the key mechanism by which regulates ESCC progression. - Source: PubMed
Publication date: 2026/05/20
Hu Zhen-YuDeng Wei-HaoHuang Jun-XiuLi ZheZheng Yu-ZhenLiao Hong-Ying - Breast cancer (BRCA) is the most common malignant tumor in women, with prognoses differing markedly by molecular subtype. Exercise is known to be related to tumor occurrence and progression, yet its role in BRCA remains understudied. This research seeks to explore the prognostic and immunomodulatory functions of exercise-related genes (ERGs) in BRCA, and their links to molecular subtypes, so as to inform targeted and precision treatment. - Source: PubMed
Publication date: 2026/05/29
Chen CaipingHan ChaoXue LiLu Xiang