Sterile Screw Tube with Caps
- Known as:
- Sterile Screw Tube Caps
- Catalog number:
- 2231-S0
- Product Quantity:
- 50 ea
- Category:
- -
- Supplier:
- Bioner
- Gene target:
- Sterile Screw Tube with Caps
Related genes to: Sterile Screw Tube with Caps
- Gene:
- CAPS NIH gene
- Name:
- calcyphosine
- Previous symbol:
- -
- Synonyms:
- CAPS1, MGC126562
- Chromosome:
- 19p13.3
- Locus Type:
- gene with protein product
- Date approved:
- 1990-05-31
- Date modifiied:
- 2016-07-18
Related products to: Sterile Screw Tube with Caps
Related articles to: Sterile Screw Tube with Caps
- When coordinating decisions in latency-sensitive 6G networks, distributed agents must act without exchanging messages at runtime. A well-known obstacle arises from the CHSH (Clauser-Horne-Shimony-Holt) inequality, which caps classical coordination accuracy at [Formula: see text] for the standard non-local game under a strict no-signaling protocol. We introduce eMARL (entanglement-assisted Multi-Agent Reinforcement Learning), a learning framework in which agents share pre-distributed Bell pairs and learn measurement policies to surpass this classical ceiling. Across [Formula: see text] independent training runs, eMARL reaches a win rate of [Formula: see text] and a CHSH parameter [Formula: see text], while transmitting zero bits during execution. Meanwhile, bandwidth-limited learned-communication baselines such as CommNet ([Formula: see text]) and TarMAC ([Formula: see text]) suffer from training instability and fall short of even the classical optimum. We characterize performance under both depolarizing and photon-loss channels. The depolarizing analysis fixes a fidelity threshold for automatically falling back to classical policies; the loss analysis distinguishes the two operating regimes of photonic networks (heralded post-selection and unheralded amplitude damping), the former preserving the conditional quantum advantage but paying a [Formula: see text] throughput penalty per round. We close the loop with a simplified Cell-Free MIMO (Multiple-Input Multiple-Output)-inspired beamforming task, where eMARL recovers [Formula: see text] of centralized throughput with zero runtime signaling. Taken together, these findings position entanglement as a promising coordination resource for CHSH-type distributed problems in principle. - Source: PubMed
Publication date: 2026/07/14
Miriyala SapthagiriChirra Venkata Ramireddy - The role of the vesicle priming factor CAPS (calcium-dependent activator protein for secretion) in somatic exocytosis of serotonin was studied in the large Retzius neuron of the leech. Increased electrical activity prompts Retzius neurons to release serotonin en masse solely from clusters of somatic dense-core vesicles (DCVs). This exocytosis is largely different from that at synapses, similar to that in endocrine cells. Therefore, the molecular complex driving DCV fusion may be composed by particular types of proteins. The search for such proteins began in the Retzius neuron-specific transcriptome. The high level of leech CAPS (Hve-CAPS1) mRNA in the transcriptome was confirmed by in situ hybridization in leech central ganglia. Dense staining developed in the somata of Retzius and other neuronal types. Immunostaining revealed accumulation of the protein in the soma shell and the nuclear periphery, where DCVs also accumulate. The role of Hve-CAPS1 in exocytosis was tested by knocking it down via injection of cocktails of synthetic interfering RNA (siRNA) into cultured neurons. Exocytosis was stimulated by 40 mM potassium depolarization and measured as fluorescent spots, each spot produced upon FM1-43 staining of a DCV cluster during exo/endocytosis cycles. Knocking down Hve-CAPS1 resulted in a 51.6% decrease in somatic FM1-43 spots compared to the control group injected with a scrambled sequence, indicating an equivalent reduction in vesicle clusters undergoing fusion. The contribution of Hve-CAPS1 to DCV priming introduces a highly regulated priming step into the conserved mechanism of somatic exocytosis in neurons. - Source: PubMed
Publication date: 2026/07/14
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