Rat Anti-Mouse CD90 Thy-1
- Known as:
- Rat Antibody toMouse CD90 Thy-1
- Catalog number:
- 128-10059-1
- Product Quantity:
- 500
- Category:
- -
- Supplier:
- Ray Biotech
- Gene target:
- Rat Anti-Mouse CD90 Thy-1
Ask about this productRelated genes to: Rat Anti-Mouse CD90 Thy-1
- Gene:
- THY1 NIH gene
- Name:
- Thy-1 cell surface antigen
- Previous symbol:
- -
- Synonyms:
- CD90
- Chromosome:
- 11q23.3
- Locus Type:
- gene with protein product
- Date approved:
- 2001-06-22
- Date modifiied:
- 2015-07-22
Related products to: Rat Anti-Mouse CD90 Thy-1
Related articles to: Rat Anti-Mouse CD90 Thy-1
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Publication date: 2026/10/02
Albertson AsherBowen Ryan MAyoub KyrillosLeon-Alvarado RoseWang BrendonPati RomanBauer Adam QLee Jin-Moo - Traumatic brain injury (TBI) is a leading cause of mortality and morbidity and a major contributor to loss of working capacity in young adults. Current therapeutic options for TBI remain limited. This study aimed to assess the potential role and therapeutic value of ubiquitin-specific protease 18 (USP18) in TBI. - Source: PubMed
Publication date: 2026/09/29
Zhao YangyangLei PanLi YulongLiu ChengxiZhang YichenZeng ChuanSun YangyangChen XinhuiYuan RaoraoFan HengyiCheng ShiqiZhang Yan - Adoptive T cell transfer remains a fundamental technique for dissecting the molecular mechanisms governing T cell differentiation. A successful adoptive-transfer experiment typically relies on labor-intensive construction of specific congenic mouse strains to distinguish donor from host cells. This limitation may be addressed by a readily detectable surface reporter. Thy1 is a candidate, but it remains unclear whether it is a reliable reporter for monitoring adoptively transferred CD8 T cell differentiation because Thy1 is a functional protein involved in thymic development and cell activation. To address this question, we systematically evaluated the reliability, stability, and functional neutrality of Thy1.1 and Thy1.2, two Thy1 isoforms, as adoptive-transfer reporters during CD8 T cell differentiation . Our results show that endogenous Thy1 expression remains stable across the differentiation stages examined. In the experimental settings tested, exogenous Thy1 reporter expression did not measurably alter the assessed differentiation phenotypes or donor-cell recovery. We further constructed and validated a Thy1 reporter-based platform that captured phenotypes associated with both loss-of-function (shRNA) and gain-of-function (cDNA) modulation of the transcription factor T-bet. This system provides a practical option for congenic background-independent adoptive transfer and genetic studies of CD8 T cell differentiation. - Source: PubMed
Publication date: 2026/09/16
Liu ChunlongLiu ChunlingZhong JingYang XiaodiWang Dapeng - Autism spectrum disorder (ASD) is a group of neurodevelopmental disorders characterized by deficits in social communication and interaction, and restricted interests or repetitive behaviors. ASD is approximately four times more prevalent in males than in females. In this study, we investigated whether sex hormones or sex chromosomes underlie the male bias in ASD susceptibility. - Source: PubMed
Publication date: 2026/09/07
Niu ChangranAn Juan JiMasterson Hannah VXu Baoji - Equine follicular aspirates obtained during ovum pick-up contain a heterogeneous mixture of mesenchymal stromal cells (MSC) and granulosa cells (GC). This study evaluates how selective isolation and expansion of MSC decrease GC populations. Follicular aspirates-derived cells were cultured up to passage four (P4) following a previously validated protocol. While freshly isolated follicular aspirates cells (FAC) displayed heterogeneous morphologies, early passages (P3-P4) showed a uniform, spindle-shaped phenotype compatible with MSC. Gene expression analysis by RT-qPCR revealed a marked upregulation of mesenchymal and stemness-associated genes (FN1, THY1, FGF2 and CD105) in P4-MSC compared with FAC, confirming enrichment of the MSC population. Conversely, GC-specific genes (FSHR, FOXL2 and CYP11A1) were significantly upregulated in FAC. These findings suggest that early passages efficiently promote selective MSC expansion while progressively eliminating GC. - Source: PubMed
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