PVR _ CD155 _ NECL5 Protein
- Known as:
- PVR _ CD155 _ NECL5 Protein
- Catalog number:
- 10109-H02H
- Product Quantity:
- 100
- Category:
- -
- Supplier:
- Smart Serology
- Gene target:
- PVR _ CD155 NECL5 Protein
Related genes to: PVR _ CD155 _ NECL5 Protein
- Gene:
- PVR NIH gene
- Name:
- PVR cell adhesion molecule
- Previous symbol:
- PVS
- Synonyms:
- CD155, HVED, Necl-5, NECL5, Tage4
- Chromosome:
- 19q13.31
- Locus Type:
- gene with protein product
- Date approved:
- 2001-06-22
- Date modifiied:
- 2019-03-14
Related products to: PVR _ CD155 _ NECL5 Protein
Related articles to: PVR _ CD155 _ NECL5 Protein
- To evaluate storage safety parameters and patient-reported bladder/bowel outcomes in adults with incomplete SCI using urodynamics and validated PROMs. - Source: PubMed
Publication date: 2026/08/20
Yadav SanjaySingh YashasviTrivedi SameerAgrawal AmritYadav Santosh KumarKumar UjwalAgarwal Lalit KumarShubham NarayanBhardwaj Madan Gopal - Acute bacterial prostatitis (ABP) is a common complication in men with lower urinary tract symptoms (LUTS). While antibiotics are the mainstay of treatment, the choice between transurethral (TC) and suprapubic (SC) catheterization remains debated. This study aimed to identify factors influencing catheterization choice and its impact on clinical outcomes. - Source: PubMed
Publication date: 2026/08/20
Handke AnalenaGassen CharlotteRausch PatriciaOrf ChristopherBahlburg HenningBach PeterRoghmann FlorianTully Karl - Superficial brachytherapy enables localized dose delivery for superficial cutaneous lesions, but conventional applicators may be limited in treating extended, irregular, or curved surfaces. Geometric constraints, gaps or overlaps between adjacent applicators, and placement-related uncertainties can reduce dose uniformity, underscoring the need to optimize applicator geometry and deployment strategy. - Source: PubMed
Jegal JinPark HyeoungWooKang SeongheeKim Jung-InChoi Chang HeonKim Siyong - Proliferative vitreoretinopathy (PVR) remains the principal biological cause of failed retinal detachment repair. Classical pathogenic models centred on retinal pigment epithelium (RPE) dispersion, retinal injury responses or compartment-restricted mechanisms do not fully explain the heterogeneity, biomechanical behaviour and clinical variability of PVR. We propose an integrative framework in which PVR is conceptualised as a biological process arising from the interaction of three interdependent pathways: vitreous scaffolding (residual vitreous cortex and embedded hyalocyte populations); inflammatory conditioning (RPE dispersion with blood-retinal barrier disruption and a profibrotic intraocular milieu); and retinal remodelling (retinal injury responses with glial remodelling and altered biomechanical properties). Within this framework, inflammatory conditioning establishes a permissive profibrotic milieu, retinal remodelling alters tissue compliance and force propagation and vitreous remnant scaffolds at the vitreoretinal interface facilitate spatial organisation of fibrocontractile activity. Although mechanistic evidence supports the contribution of each of the three pathways to PVR pathogenesis, direct prospective validation of their threshold-dependent convergence as a unified construct requires further research. The proposed model, therefore, distinguishes biological plausibility from proven causality and aims to generate testable hypotheses rather than prescriptive conclusions. By integrating inflammatory, retinal and vitreoretinal interface perspectives within a unified conceptual structure, this framework provides a biologically coherent explanation for the spatial distribution, contractility, recurrence patterns and clinical heterogeneity of PVR. - Source: PubMed
Publication date: 2026/08/19
van Overdam Koen ASebag J - To describe a user-friendly voiding monitoring method based on a bedwetting alarm, and to evaluate its clinical significance as a complementary assessment of lower urinary tract function in neurogenic and non-toilet-trained children for whom standard urodynamic studies are unfeasible, technically challenging, or yield suboptimal results. - Source: PubMed
Publication date: 2026/07/25
Ben-Meir DavidTal YuliaBuyevich EvgeniaBabaoff AnnaFrumer Michael