ACHE Antibody
- Known as:
- ACHE Antibody
- Catalog number:
- AF1017a
- Product Quantity:
- 0.1mg
- Category:
- -
- Supplier:
- Abgen
- Gene target:
- ACHE Antibody
Related genes to: ACHE Antibody
- Gene:
- ACHE NIH gene
- Name:
- acetylcholinesterase (Cartwright blood group)
- Previous symbol:
- YT
- Synonyms:
- -
- Chromosome:
- 7q22.1
- Locus Type:
- gene with protein product
- Date approved:
- 1989-06-02
- Date modifiied:
- 2019-04-23
Related products to: ACHE Antibody
Related articles to: ACHE Antibody
- Neuropathic pain is frequently accompanied by anxiety and depressive symptoms, resulting in a comorbid state of pain and negative emotions, the underlying mechanisms of which remain incompletely understood. This study aims to investigate the regulatory mechanism of the descending anterior cingulate cortex (ACC)-spinal dorsal horn (SDH) pathway in negative emotion-facilitated neuropathic pain and to identify and validate key molecules acting at the spinal level. - Source: PubMed
Wang JianDing ZhuofengXu WeiZou WangyuanGuo QulianSong Zongbin - Chronic Knee pain is one of the most reported types of pain in the United States and causes a significant issue in rural and underserved communities. Radiofrequency ablation (RFA) is a treatment that has shown significant improvement for up to 24 months for people suffering from chronic knee pain, but consensus guidelines for RFA in the management of chronic knee pain with emphasis on the unique aspects of rural health care have not been established. - Source: PubMed
Publication date: 2026/09/19
Gonzalez Eyeneys ValdiviaFabio ShaniceMartin CherylRawson EleanorMaye John - To evaluate the association between interaction with a certified therapy dog prior to awake, in-office laryngologic procedures and patient-reported anxiety, depression, and pain compared with standard care. - Source: PubMed
Publication date: 2026/09/19
Saleem NawfalWeiss NatalieFranklin AndrewGreen KaterinaRutt Amy - While recent studies have highlighted sexual dysfunctions as a common comorbidity of several psychiatric disorders very few have focused on social anxiety disorder. However, its shared impact with sexual dysfunctions on interpersonal relationships and self-esteem suggested a potential bidirectional relationship worth investigating. - Source: PubMed
Publication date: 2026/09/19
Pailloux LucAchour VincentFaugere MélanieVerney PierreLançon ChristopheKorchia Théo - Endometriosis is a chronic inflammatory disease characterized by substantial heterogeneity in pain, lesion behavior, and treatment response. Although hormonal and surgical therapies can reduce disease activity and symptoms, their effects are variable, and the biological mechanisms underlying persistent pain and lesion recurrence remain incompletely understood. Emerging evidence supports reciprocal interactions among sensory neurons, immune cells, stromal populations, the vasculature, and the extracellular matrix in endometriotic lesions. Rather than proposing these interactions as a new observation, we use them as the biological basis for a testable Neuroimmune Fitness Landscape, in which lesion persistence is hypothesized to reflect the capacity of the local microenvironment to maintain coordinated neural, immune, stromal, vascular, and metabolic activity despite defined perturbations. We further propose that this framework may help organize established mechanisms underlying pain heterogeneity, tissue remodeling, lesion persistence, and therapeutic response, while generating experimentally testable predictions regarding network persistence, resilience, and reversibility. Importantly, the framework is conceptual and does not establish neuroimmune fitness or neuroimmune memory as validated biological entities. We discuss how this model could guide future studies of neuroimmune modulation, metabolic intervention, targeted delivery, and precision phenotyping. We also identify neuroimmune memory as a distinct, unresolved hypothesis that would require longitudinal demonstration of persistence, enhanced responses to subsequent challenge, and reversibility after targeted intervention. Thus, the proposed framework should be viewed as a hypothesis-generating model for integrating existing evidence and directing experimental validation, rather than as an established explanation of endometriosis persistence or therapeutic resistance. - Source: PubMed
Publication date: 2026/09/19
Zahiri ZibaGhayur ShivaAlborzi SaeedGhasemi MaryamMiri Mahboobe SadatTaftian AzadehKhonsarian Zahra SadatBahrampour AidaGoodarzi VahidEtezadi Atoosa