Mouse IgM (µ) (min X Hu, Bov, Hrs Sr Prot)

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403 EUR
483 USD
334 GBP
known as: Mouse IgM (µ) (min X Hu, Bov, Hrs Sr Prot)
Catalog number: genta-JGM005075
Product Quantity: 1 mg.
Category:
Supplier: Accu

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Gene target: min x hu bov hrs sr prot

Related genes to: Mouse IgM (µ) (min X Hu, Bov, Hrs Sr Prot)

Symbol : Hrs NIH gene
LocusTag : Dmel_CG2903
Synonyms : CG2903|DmHRS|DmelCG2903|HRS|HRS-2|Vps27|dHrs|hrs|l(2)23AB5|l(2)23Ad|l(2)23Ad(Hrs)|vps27
dbXrefs : FLYBASE:FBgn0031450
chromosome : 2L
map location : 23A3-23A3
description : Hepatocyte growth factor regulated tyrosine kinase substrate
type of gene : protein-coding
Symbol from nomenclature authority : Hrs
Full name from nomenclature authority : Hepatocyte growth factor regulated tyrosine kinase substrate
Nomenclature status : O
Other designations : CG2903-PA|CG2903-PB|CG2903-PC|F[[1]]F[[0]] ATPase|HGF-regulated tyrosine kinase substrate|Hrs-PA|Hrs-PB|Hrs-PC|hepatocyte growth factor regulated tyrosine kinase|hepatocyte growth factor regulated tyrosine kinase substrate|hepatocyte growth factor-re
Modification date : 2016-06-11
Symbol : IGM NIH gene
description : IgM constant region
type of gene : other
Modification date : 2016-04-02
Symbol : min NIH gene
Synonyms : mi
dbXrefs : FLYBASE:FBgn0002747
chromosome : 2
map location : 56E-56E|2-90 cM
description : mini
type of gene : unknown
Modification date : 2016-05-09
Symbol : Prot NIH gene
dbXrefs : MGI:MGI:107578
chromosome : 18
description : proline transporter
type of gene : protein-coding
Symbol from nomenclature authority : Prot
Full name from nomenclature authority : proline transporter
Nomenclature status : O
Modification date : 2016-07-02

Related Pathways to: Mouse IgM (µ) (min X Hu, Bov, Hrs Sr Prot)

Gene about :IgM
Pathway :Rn Inflammatory Response Pathway
IgM

Related product to: Mouse IgM (µ) (min X Hu, Bov, Hrs Sr Prot)

Related Articles about: Mouse IgM (µ) (min X Hu, Bov, Hrs Sr Prot)

Specific IgM and Regulation of Antibody Responses.

Specific IgM, administered together with the antigen it recognizes, enhances primary antibody responses, formation of germinal centers, and priming for secondary antibody responses. The response to all epitopes on the antigen to which IgM binds is usually enhanced. IgM preferentially enhances responses to large antigens such as erythrocytes, malaria parasites, and keyhole limpet hemocyanine. In order for an effect to be seen, antigens must be administered in suboptimal concentrations and in close temporal relationship to the IgM. Enhancement is dependent on the ability of IgM to activate complement, but the lytic pathway is not required. Enhancement does not take place in mice lacking complement receptors 1 and 2 (CR1/2) suggesting that the role of IgM is to generate C3 split products, i.e., the ligands for CR1/2. In mice, these receptors are expressed on follicular dendritic cells (FDCs) and B cells. Optimal IgM-mediated enhancement requires that both cell types express CR1/2, but intermediate enhancement is seen when only FDCs express the receptors and low enhancement when only B cells express them. These observations imply that IgM-mediated enhancement works through several, non-mutually exclusive, pathways. Marginal zone B cells can transport IgM-antigen-complement complexes, bound to CR1/2, from the marginal zone and deposit them onto FDCs. In addition, co-crosslinking of the BCR and the CR2/CD19/CD81 co-receptor complex may enhance signaling to specific B cells, a mechanism likely to be involved in induction of early extrafollicular antibody responses. - Source :PubMed

Reliability, validity, and norms of the 2-min walk test in children with and without neuromuscular disorders aged 6-12.

The 2-min walk test may be more appropriate functional exercise test for young children. This study aimed to examine the 2-min walk test's reliability; validity; and minimal clinically important difference; and to establish norms for children aged 6-12. - Source :PubMed

The elicitor protein AsES induces a SAR response accompanied by systemic microbursts and micro-HRs in Fragaria ananassa.

The elicitor AsES (Acremonium strictum Elicitor Subtilisin) is a 34 KDa subtilisin-like protein secreted by the opportunistic fungus Acremonium strictum. AsES activates the innate immunity and confers resistance against anthracnose and grey mold diseases in strawberry plants (Fragaria x ananassa Duch.) and the last disease also in Arabidopsis. In the present work, we show that upon AsES recognition, a cascade of defense responses is activated, including: calcium influx, biphasic oxidative burst (O2(.-) and H2O2), HR response, accumulation of autofluorescent compounds, cell wall reinforcement with callose and lignin deposition, salicylic acid accumulation, and expression of defense-related genes such as FaPR1, FaPG1, FaMYB30, FaRBOH-D, FaRBOH-F, FaCHI23 and FaFLS. All these responses occurred following a spatial and temporal program: firstly were induced in infiltrated leaflets (local acquired resistance, LAR), spreading out to lateral leaflets and later to distal untreated leaves (systemic acquired resistance, SAR). After AsES treatment, macro-HR and macro oxidative bursts were localized in infiltrated leaflets while micro-HRs and microbursts occurred later in untreated leaves, being confined to a single cell or a cluster of few epidermal cells that differentiate from the surrounding ones. The differentiated cells initiate a time dependent series of physiological and anatomical changes, evolving to idioblasts accumulating H2O2 and autofluorescent compounds that blast delivering its content unto surrounding cells. This kind of systemic cell death process in plants is described for first time in response to a single elicitor. All data presented in this study suggest that AsES has the potential to activate a wide spectrum of biochemical and molecular defense responses in F. ananassa what may explain the induced protection towards pathogens of opposite lifestyle like hemibiotrophic and necrotrophic fungi. - Source :PubMed

Anti-Fas2 IgM antibodies in Fasciola hepatica infected patients with positive IgG serology.

Fascioliasis is an infectious disease caused by parasites Fasciola hepatica and F. gigantica. Humans are infected by the consumption of vegetables and water contaminated with the infective form of the parasite. - Source :PubMed

5000 Meter Run Performance is not Enhanced 24 Hrs After an Intense Exercise Bout and Cold Water Immersion.

Cold water immersion (CWI) is used by endurance athletes to speed recovery between exercise bouts, but little evidence is available on the effects of CWI on subsequent endurance performance. The purpose of this study was to investigate the effects of CWI following an acute bout of interval training on 5000 m run performance 24 hrs after interval training, perceived muscle soreness (PMS), range of motion (ROM), thigh circumference (TC), and perceived exertion (RPE). Nine endurance-trained males completed 2 trials, each consisting of an interval training session of 8 repetitions of 1200 m at a running pace equal to 75% of VO2peak, either a control or CWI treatment, and a timed 5000 m run 24 hrs post interval training session. CWI was performed for 12 min at 12 degrees Celsius on the legs. Recovery treatments were performed in a counterbalanced design. Run time for 5000 m was not different between the CWI and control trials (CWI = 1317.33 ± 128.33 sec, control = 1303.44 ± 105.53 sec; p = 0.48). PMS increased significantly from baseline to immediately post exercise (BL = 1.17 ± 0.22, POST = 2.81 ± 0.52; p = 0.02) and remained elevated from baseline to 24 hrs post exercise (POST24 = 2.19 ± 0.32; p = 0.02), but no difference was observed between the treatments. No differences were observed for the interaction between time and treatment for TC (λ = 0.73, p = 0.15) and ROM (λ = 0.49; p = 0.10). CWI performed immediately following an interval training exercise bout did not enhance subsequent 5000 m run performance or reduce PMS. CWI may not provide a recovery or performance advantage when athletes are accustomed to the demands of the prior exercise bout. - Source :PubMed

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